"The Actual Cure For Male Pattern Baldness"

outcast1979

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does any1 have indepth guides how to cut out estrogen?

the real deal, not some bluepilled mens health ***t
If your target is mental issues and not sexual,then you must search for a way to wake up your gabba system,if you have sexual problems the most common could be cortizol prolactin estrogens out of balance.Its very difficult to eliminate all this together and have a balancing profile
 

Inaut

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I don't know why I often go back to Edgar Cayce's material but will post this as I think it falls in line with much of what is said on this forum regard baldness.



1. Physiological Considerations

Glandular insufficiency and spinal lesions (subluxations), according to the Cayce material, are by far the most common causes of hair loss (baldness) which may be accompanied by nail and even skin changes (abnormal pigmentation, vitiligo, etc.). Glandular dysfunction - usually the thyroid but the thymus and adrenals may also be involved may come about through the diet, i.e., insufficient amounts of necessary elements like calcium, or excesses of others like potassium. Other causes of glandular dysfunction include insufficient circulation, stress, infection, toxic chemicals (as found in cosmetics), general debilitation, etc.

The most commonly involved gland is the thyroid. It seems that when some elements necessary for proper thyroid functioning are missing, toxins which otherwise would have been eliminated are allowed to accumulate in the system. When this happens, inflammation, congestion, and circulatory disturbances occur, affecting the scalp and maybe the nails and skin. It is worth noting that the thyroid to some extent controls the circulation to the scalp, nails and skin, independent of the mechanism described above. When such disturbances occur, the outcome is hair loss with or without nail and skin changes, depending on the severity of the condition.

Impaired circulation from other causes may bring about the same effects without necessarily involving the thyroid or other glands, but is a much less common finding. The exception is spinal subluxation, being almost equally as frequent as glandular disturbance. It should be noted, though, that even when spinal subluxation is the primary condition, the glands often become involved as a result of impaired circulation through them (the thyroid especially). Hence in a large majority of cases the glands are either the primary or contributing cause of the condition.

In one or two instances, reference was made to prenatal tendencies as a contributory cause, but no definite information is available on this or on heredity.

II. Rationale of Therapy
The treatments recommended reflect the disease process involved and may be classified as follows:

  1. Correction of glandular dysfunction usually includes dietary advice where deficiencies are involved. Atomidine and sometimes glandular extracts (thyroid, adrenals, etc.) are necessary.
  2. Osteopathy: Series of treatments to correct spinal subluxation that may be causing circulatory, glandular and other organ system dysfunction as well as hair loss.
  3. Others: Treatment of underlying disorders (e.g., avoidance of stress and toxic chemicals, treatment of infections, etc.) as well as complications of basic disease process (e.g., spinal subluxation usually causes widespread effects) which may contribute to or aggravate the hair loss.
Osteopathic adjustments bring about improved circulation to the scalp, nails and skin, thus leading to beneficial changes. When digestive disturbances (assimilations/eliminations) are also present as a result of spinal lesions, toxic accumulations may be substantially eliminated or prevented in the digestive tract, further improving circulation to involved areas. What may be less obvious is that the use of laxatives, colonic enemas, etc., would bring about similar results through elimination of toxins.

These few examples are based on the assumption that the physiology described by Cayce is correct. Intuitively, it seems to make sense to me.

III. Suggested Therapeutic Regimen
Baldness is not a problem treated by the average physician, for there is no recommended medical treatment presently available. The following might be considered a reasonable approach to the average patient, from a study of the Edgar Cayce readings.

1. Correction of glandular deficiency.

  • Atomidine: Various programs were prescribed. There is no given formula for arriving at dosages. One suggestion is as follows:
One drop for seven days, rest five days
Two drops for seven days, rest five days
Three drops for seven days, rest five days
May repeat this series once or twice.)

  • Thyroid extract in small doses two to three times a week for a few weeks in combination with Atomidine in severe deficiency.
  • Other glandular extracts (replacements), as necessary, e.g., adrenal.
  • Correction of mineral deficiencies and/or excesses (e.g., low calcium, high potassium). Calcios is a good source of calcium (a layer on a cracker taken every other day).
  • Dietary:
    The skin of Irish potatoes cooked in Patapar paper (to preserve active principles) supplies some essential elements for proper thyroid function. This may be eaten three or more times a week. (Roasted or baked is also good so long as it is not burnt.)

    Other helpful hints for better thyroid activity include citrus fruit juices - orange juice plus lemon or grapefruit juice plus lime - in combination with Atomidine will act on the thyroid to improve circulation to the scalp. Seafoods were recommended three times a week. Carrots are good.

    Avoid: fried, greasy foods, fried meats, starches, refined sugars, onions, garlic.
2. Osteopathy: This would be helpful if a history of spinal injury is obtained or when other symptoms and signs warrant this. Even in the absence of the above a few treatments would probably still be beneficial, since circulation to the thyroid, scalp, nails, skin, etc., Will be enhanced. The areas manipulated as well as the number of treatments should be considered on an individual basis.

3. Local measures:

  • Crude oil massage to the scalp (one teaspoon) to stay on from one-half hour to 45 minutes. Then cleanse with a 20% grain alcohol solution. Follow this with a massage using white petroleum (Vaseline) into scalp (not too greasy).
  • An alternative would be a scalp massage with pure hog lard, leaving this on overnight after covering head with an oil cap.
  • Shampoo in the morning with olive oil shampoo followed by a massage with white Vaseline cut with a little alcohol (one drop grain alcohol to one ounce water). Either may be done once a week.
  • Violet ray treatment (20-25 times) to scalp, spine, scapula, umbilical area for a total of five to ten minutes on a daily basis. Ultraviolet ray treatments may be used instead, this done every third day, limiting treatment to scalp and spine for three to five minutes for 20-25 treatments.
4. Other: Maintain proper elimination through the use of laxatives, colonic enemas, massages, etc. Diathermy, vibrator, hydrotherapy, radio-active appliance were also recommended in some instances.

A prescription is given in reading 636-1 for restoring hair color. Dosage is half a teaspoon three times a day after meals for ten days with five days rest periods.

Steps 1 and 3 seem to be a must, while 2 and 4 may be utilized at the discretion of the therapist, depending on the case being treated.

[Note: The preceding overview was written by Hezekiah U. Chinwah, M.D. and is excerpted from the Physician's Reference Notebook, Copyright © 1968 by the Edgar Cayce Foundation, Virginia Beach, VA.]


Overview of Baldness
 

Ableton

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what I want to know from all those guys here who have contributed to this forum and read more than me:

what is "stress" for you?

why are we going hypothyroid?

what would happen if we did not go hypothyroid despite the stress? Why is consuming say 6000 calories a day no guaranteed reversal of hypothyroidism? would we still "burn out" if our bodies ran hot because of...stress? or are our bodies not letting us out of this catabolic stage despite having enough energy ressources to do so because we would get cancer otherwise?

i have layed out a rudimentary theory for that. im probably not the first one to do it either, but lets take a second to address it anyways. You can claim it's "oversimplified" or whatever, but give an explanation then.



To me this just holistically, evolutionary (rise of carcinogenics, cancer, and hypothyroidism; evolution preferring survival over anything else) makes a lot of sense. Sometimes its good to take a step back and look at it simplified, instead of going into the 300th paper and claim "stress" at the end anyways.


Living kills. Eating kills us, but we have to eat to survive.


All of this (balding, hypothyroidism, balance of anabolism vs catabolism) comes down to a balance of energy (too little =death, starvation, shitty life quality; too much --> cancer in bad environment) vs. reproduction of dna/methylation (too little --> aging, baldness; too much --> cancer)

its comes down to the dichotomy of energy and cancer

thank you for adressing my points or giving me further direction, and please refrain from using the term "stress" without defining it.

I am waiting.
 
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L_C

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Reading thru this thread again got me thinking and I'm going to loosely attempt to connect the dots presented elsewhere on this forum - so bare with me.

- A healthy thyroid raises natural progesterone production and I believe Peat said T3 is important in wound healing / regenerative processes
- If progesterone levels aren't optimal this leads to more testosterone aromatizing into estrogen
- Females have menstral cycles to assist/protect them with the ebb and flow of progesterone, but it becomes a problem with PCOS
- Men's ability to produce progesterone deteriorates as they age and by age 40 they likely are estrogen dominant
- Low Thyroid causes low body temperatures and also inhibits your liver's ability to process excess estrogen
- Low body temperatures drastically reduce the body's ability to minimize fungal threats
- Fungal infections thrive in highly estrogenic environments
- Fungal infections are commonly found in the skin and scalp causing inflammation (ex. the MPB itch)
- Elevated prostaglandin levels have been observed in chronic C.Albicans infections
- Prostaglandin D2 (PGD2) is involved in the regulation of body temperature during sleep
- In low thyroid states, the adrenals are working overtime often chronically over-producing cortisol
- Cortisol affects our gut microbes and permeability leading to further colonization by opportunistic pathogens/fungi
- DHT shows up in the inflamed scalp in an attempt to reduce the inflammation
- DHT is also a known antagonistic to estrogen, cortisol, serotonin
- Finasteride has been shown as highly active in prevention of fungi (ex. C.Albicans) and major synergistic effects when taken with Fluconazole (Fin is often paired w/Nizoral shampoo)
- Finasteride is a 5 alpa-reductase inhibitor i.e. DHT blocker
- In the beginning, many see their hairloss subside on Finasteride - even if they don't grow anything back, but eventually it loses it's effectiveness (Duhh it's inhibiting DHT)
- Sebaceous glands are enlarged in balding scalps as the hair follicle shrinks during hair loss (i.e. physical obstruction)
- Sebaceous glands create oils/fats (sebum) which fungi like to feed on (ex. Malassazia) and can be colonized by acne bacteria (i.e. foliculitis /more inflammation)
- Progesterone reduces sebum activity
- While all of this is happening the scalp continues to remain inflamed ultimately leading to fibrosis

I dunno. Maybe I'm just being an idiot here - but the thyroid connection and the cascade of issues when it's low is really starting to make sense to me now. I think stuff like Fin is actually handling the fungi/inflammatory aspect of hair loss while naively assuming DHT was the culprit. However it can't stave off the systemic issues of low thyroid eventually causing PFS because of DHT inhibition (particularly in men who are already low DHT to begin with). I know for myself, labs have shown low serum DHT, so there is no shot in hell I will ever step foot near a drug like Fin or Dut.

@mrchibbs thoughts?


@JDreamer

If progesterone is reducing sebum activity, what is actually INCREASING sebum activity? Estrogen?
 

Ableton

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@JDreamer

If progesterone is reducing sebum activity, what is actually INCREASING sebum activity? Estrogen?
would like to know that as well.
I can say for sure that since I drastically reduced heated fat consumption my sebum activity is basically zero now. I can go three days without showering, when I always smelled like a bull shortly after eating anything fried.

What is happening in my body?

I suppose its pufa related since even sat fats have pufa content but IDK.

I also went very low fat so that might have something to do with it as well.

My waking temps are finally rising as well

I feel like a defect in lipid metabolism can be at play in mpb, and there have been other anecdotes like this on here as well. Fat or fatty acids are extremely catabolic to me, even saturated.

Lampofreds suggestions went in this direction as well, and not knowing about RP's opinion on hair loss a lot, I wouldn't be surprised if this ultimately comes down to PUFA. And what many of us are doing wrong, is thinking excess sat fats are okay when they have pufa in them as well. And the heating of fats are another thing that I do not know anything about, but they are the most terrible thing for me. Needlessly to say that curve of mpb correlates with use of heavily fried foods everywhere (think about the USA, and then Asia). Most seem to tolerate this stuff just fine though. I am literally falling apart if I eat deep fried food frequently lol
 
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L_C

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would like to know that as well.
I can say for sure that since I drastically reduced heated fat consumption my sebum activity is basically zero now. I can go three days without showering, when I always smelled like a bull shortly after eating anything fried.

What is happening in my body?

I suppose its pufa related since even sat fats have pufa content but IDK.

I also went very low fat so that might have something to do with it as well.

My waking temps are finally rising as well


Interesting, I don't product enough sebum either but I would say I do consume fat like bone broths. I am super confused on how to increase sebum production.
 

Inaut

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I've been starting to use malic acid as a hair rinse/conditioner and find it really gets rid of sebum quite well. I'm starting to wash my hair every other day now (as it used to look very greasy if I didn't do it every day). I take a quarter teaspoon in a quart of water in the shower with me whenever I wet my hair. I guess I prefer it to vinegar as it always left my hair stringy and stinky
 

L_C

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I've been starting to use malic acid as a hair rinse/conditioner and find it really gets rid of sebum quite well. I'm starting to wash my hair every other day now (as it used to look very greasy if I didn't do it every day). I take a quarter teaspoon in a quart of water in the shower with me whenever I wet my hair. I guess I prefer it to vinegar as it always left my hair stringy and stinky


well, good for you. My problem is that I don't produce enough sebum to begin with. I am asking for ways on how to increase sebum production.
 

outcast1979

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@JDreamer

If progesterone is reducing sebum activity, what is actually INCREASING sebum activity? Estrogen?
If progesterone reduce sebum that means that i have high pregesterone,cause after quiting propecia i had never again produce oil in my face neither my scalp,along with the rest damage.
 

JDreamer

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@JDreamer

If progesterone is reducing sebum activity, what is actually INCREASING sebum activity? Estrogen?

"Enhanced sebaceous gland activity is attributed to the potent androgen 5α-DHT5 as sebaceous gland cells possess all necessary enzymes for conversion of testosterone to 5α-DHT."

An update on the role of the sebaceous gland in the pathogenesis of acne.
  • Testosterone converts to DHT
  • DHT competes with Estrogen and is upped in response to inflammation
  • Sebaceous glands can contain 5-AR type I and II
  • Progesterone competes with Testosterone for 5-AR
At least that's how I've conceptualized it based on what I've read around here and thru PubMed.
 

JDreamer

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would like to know that as well.
I can say for sure that since I drastically reduced heated fat consumption my sebum activity is basically zero now. I can go three days without showering, when I always smelled like a bull shortly after eating anything fried.

What is happening in my body?

I suppose its pufa related since even sat fats have pufa content but IDK.

I also went very low fat so that might have something to do with it as well.

My waking temps are finally rising as well

I feel like a defect in lipid metabolism can be at play in mpb, and there have been other anecdotes like this on here as well. Fat or fatty acids are extremely catabolic to me, even saturated.

Lampofreds suggestions went in this direction as well, and not knowing about RP's opinion on hair loss a lot, I wouldn't be surprised if this ultimately comes down to PUFA. And what many of us are doing wrong, is thinking excess sat fats are okay when they have pufa in them as well. And the heating of fats are another thing that I do not know anything about, but they are the most terrible thing for me. Needlessly to say that curve of mpb correlates with use of heavily fried foods everywhere (think about the USA, and then Asia). Most seem to tolerate this stuff just fine though. I am literally falling apart if I eat deep fried food frequently lol

Going very low fat (including "good fats") seems counter-productive to cholesterol and making hormones no?

I was under the impression some sebum is necessary for greasing the hair shaft. Since it's a lipid, I would assume some of it comes from diet.
 
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