Zpol
Member
- Joined
- Apr 14, 2013
- Messages
- 929
- Age
- 45
Ok, so to be clear... we all understand that:
- Phosphatidylcholine, in some people, can result in the formation of trimethylamine N-oxide (aka TMAO).
-> TMAO is an endotoxin that forms when certain gut microbes (I'm not sure which strains) consume foods containing the choline group of the PC lipid.
-> Presence of TMAO is highly correlated to heart disease.
-> One way to know if gut microbiota are forming this TMAO, is if one develops a fishy smell when consuming PC.
-> Rats have reported a fishy smell when using MitoLipin, a PC supplement
-> The fishy smell in MitoLipin is from the Vitamin E which is used as a solvent
-> MitoLipin does not produce TMAO in those who have the aforementioned gut microbes
= MitoLipin does not contribute to the formation of TMAO, and is therefore safe for rats who have heart disease.
Am I correct in this logic?
- Phosphatidylcholine, in some people, can result in the formation of trimethylamine N-oxide (aka TMAO).
-> TMAO is an endotoxin that forms when certain gut microbes (I'm not sure which strains) consume foods containing the choline group of the PC lipid.
-> Presence of TMAO is highly correlated to heart disease.
-> One way to know if gut microbiota are forming this TMAO, is if one develops a fishy smell when consuming PC.
-> Rats have reported a fishy smell when using MitoLipin, a PC supplement
-> The fishy smell in MitoLipin is from the Vitamin E which is used as a solvent
-> MitoLipin does not produce TMAO in those who have the aforementioned gut microbes
= MitoLipin does not contribute to the formation of TMAO, and is therefore safe for rats who have heart disease.
Am I correct in this logic?