Hope I'm not being a nit here, but do animals have cell walls? Perhaps they meant gut linings instead of cell walls. Or the cell membranes of the gut lining?
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Bacterial Lipids (endotoxin), Not Cholesterol, May Be A Cause Of CVD
OP
It is not my comment, it is a quote from the authors. I think they mean passing through cells and eventually into capillaries/bloodstream.
My current concern is with endotoxins that are already in my vascular system, not the ones that are in the gut and attempting to get in through gut linings.
I find that when I use proteolytic enzymes, particularly serrapeptidase, plaques are lysed and release endotoxin. I detect an immune response and this causes inflammation, and this is reflected in increased blood pressure. Since I'm taking the enzymes to remove a cause of hypertension in the form of plaque, the problem is that the solution (serrapeptidase enzyme) also becomes a problem itself - in that it becomes another cause of hypertension.
If only there is the vascular equivalent of charcoal, which would adsorb these endotoxins. It would be a good complement to serrapeptidase. The enzyme would do its job of lysing plaque, and the adsorbent would scavenge the endotoxins released by the lysing process, thus keeping the side-effect of endotoxins released to a minimum.
Currently I'm just using progesterone to deactivate the endotoxins, but it's not enough to deal with all the endotoxins in my vascular system. I could use vitamins A, D, b2, b3, emodin, and cyproheptdance to blunt the TLR4 receptor to keep inflammation at bay. And I could take taurine (and/or TUDCA) as well as increase glycine and sugar intake to help the liver detoxify and excrete the endotoxins through bile and the fecal route.
Is there anything I'm missing?
I find that when I use proteolytic enzymes, particularly serrapeptidase, plaques are lysed and release endotoxin. I detect an immune response and this causes inflammation, and this is reflected in increased blood pressure. Since I'm taking the enzymes to remove a cause of hypertension in the form of plaque, the problem is that the solution (serrapeptidase enzyme) also becomes a problem itself - in that it becomes another cause of hypertension.
If only there is the vascular equivalent of charcoal, which would adsorb these endotoxins. It would be a good complement to serrapeptidase. The enzyme would do its job of lysing plaque, and the adsorbent would scavenge the endotoxins released by the lysing process, thus keeping the side-effect of endotoxins released to a minimum.
Currently I'm just using progesterone to deactivate the endotoxins, but it's not enough to deal with all the endotoxins in my vascular system. I could use vitamins A, D, b2, b3, emodin, and cyproheptdance to blunt the TLR4 receptor to keep inflammation at bay. And I could take taurine (and/or TUDCA) as well as increase glycine and sugar intake to help the liver detoxify and excrete the endotoxins through bile and the fecal route.
Is there anything I'm missing?
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