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Right. But I would take astaxanthin 4 mg twice a day, in summer period.EnoreeG said:Correlation is not necessarily causation, right?
Such_Saturation said:http://www.raypeatforum.com/forum/viewtopic.php?f=75&t=7124
The sun is rising. Hear the words of anything regarding Melanoma.
EnoreeG said:Melanoma.
LucH are you aware that RP advises against taking beta-carotene and his reasons for doing so?LucH said:Right. But I would take astaxanthin 4 mg twice a day, in summer period.EnoreeG said:Correlation is not necessarily causation, right?
Most people lack vitamin E and beta-carotene. Vit E suppresses some very agressive ROS. But the result is only 1/3 to 1/2 when there is no carotene (5 - 10 mg form natural sources: fruits and vegetables).
Don't take supplement of artificial b-carotene if you smoke.
LucH
Besides fasting, or chronic protein deficiency, the common causes of hypothyroidism are excessive stress or “aerobic” (i.e. anaerobic) exercise, and diets containing beans, lentils, nuts, unsaturated fats (including carotene), and undercooked broccoli, cauliflower, cabbage, and mustard greens. Many health conscious people become hypothyroid with a synergistic program of undercooked vegetables, legumes instead of animal proteins, oils instead of butter, carotene instead of vitamin A, and breathless exercise instead of stimulating life.
you are "misinterpreting" what I said.4peatssake said:LucH are you aware that RP advises against taking beta-carotene and his reasons for doing so?
LucH said:We convert small amounts of bêta-carotene into retinol but not everybody can do it (aging).
Wikipedia said:Disease states associated with carotenoderma include hypothyroidism, diabetes mellitus, anorexia nervosa, nephrotic syndrome, and liver disease. In hypothyroidism and diabetes mellitus, the underlying mechanism of hypercarotenemia is thought to be both impaired conversion of beta-carotene into retinol and the associated increased serum lipids. Diabetes mellitus has also been associated with carotenoderma through disease-specific diets that are rich in vegetables. In the nephrotic syndrome, the hypercarotenemia is related to the associated increased serum lipids, similar to the above entities. It is of note that kidney dysfunction in general is associated with hypercarotenemia as a result of decreased excretion of carotenoids. Liver dysfunction, regardless of origin, causes hypercarotenemia as a result of the impaired conversion of carotenoids to retinol.