A 3rd Opinion - What does this blood test mean?

[Rinse & rePeat]

“Phosphate toxicity offers some interesting insights into stress and aging, helping to explain the protective effects of carbon dioxide, thyroid hormone, sugar, niacinamide, and calcium. It also suggests that other natural substances used as food additives should be investigated more thoroughly. Excessive citric acid, for example, might activate dormant cancer cells (Havard, et al., 2011), and has been associated with malignancy (Blüml, et al., 2011). Nutritional research has hardly begun to investigate the optimal ratios of minerals, fats, amino acids, and other things in foods, and how they interact with the natural toxicants, antinutrients, and hormone disrupters in many organisms used for food.“ -Ray Peat

 

[Rinse & rePeat]

“Abram Hoffer's research was instrumental in getting niacin recognized as a heart protective drug, but nearly everyone who prescribes it does so to lower blood lipids. That wasn't Hoffer's understanding of its function. He thought it acted directly on blood vessels to protect their integrity. During his studies of its effects on heart disease, he saw that it also lowered cancer mortality, and so began treating cancer patients with it, with considerable success, but there was no medical cliché that could allow the profession to follow in that direction.” -Ray Peat

 

[Rinse & rePeat]

“The ratio of saturated fatty acids to polyunsaturated fatty acids is decreased in cancer. Omega-3 fats promote metastasis.” -Ray Peat

 

[Rinse & rePeat]

“When cancers are metastasizing, their phospholipids contain less stearic acid than the less malignant tumors (Bougnoux, et al., 1992), patients with advanced cancer had less stearic acid in their red blood cells (Persad, et al., 1990), and adding stearic acid to their food delayed the development of cancer in mice (Bennett, 1984). The degree of saturation of the body's fatty acids corresponds to resistance to several types of cancer that have been studied (Hawley and Gordon, 1976; Singh, et al., 1995).“ -Ray Peat

 

[Rinse & rePeat]

“When stress is very intense, as in trauma or sepsis, the reaction of liberating fatty acids can become dangerously counter-productive, producing the state of shock. In shock, the liberation of free fatty acids interferes with the use of glucose for energy and causes cells to take up water and calcium (depleting blood volume and reducing circulation) and to leak ATP, enzymes, and other cell contents (Boudreault and Grygorczyk, 2008; Wolfe, et al., 1983; Selzner, et al, 2004; van der Wijk, 2003), in something like a systemic inflammatory state (Fabiano, et al., 2008) often leading to death.” -Ray Peat

 

[Rinse & rePeat]

“Cancer researchers have become interested in this enzyme system that controls the oxidation of pyruvic acid (and thus sugar) by the mitochondria, since these enzymes are crucially defective in cancer cells (and also in diabetes). The chemical DCA, dichloroacetate, is effective against a variety of cancers, and it acts by reactivating the enzymes that oxidize pyruvic acid. Thyroid hormone, insulin, and fructose also activate these enzymes. These are the enzymes that are inactivated by excessive exposure to fatty acids, and that are involved in the progressive replacement of sugar oxidation by fat oxidation, during stress and aging, and in degenerative diseases; for example, a process that inactivates the energy-producing pyruvate dehydrogenase in Alzheimer's disease has been identified (Ishiguro, 1998). Niacinamide, by lowering free fatty acids and regulating the redox system, supporting sugar oxidation, is useful in the whole spectrum of metabolic degenerative diseases.“ -Ray Peat

 

[Rinse & rePeat]

“Cancer researchers have become interested in this enzyme system that controls the oxidation of pyruvic acid (and thus sugar) by the mitochondria, since these enzymes are crucially defective in cancer cells (and also in diabetes). The chemical DCA, dichloroacetate, is effective against a variety of cancers, and it acts by reactivating the enzymes that oxidize pyruvic acid. Thyroid hormone, insulin, and fructose also activate these enzymes. These are the enzymes that are inactivated by excessive exposure to fatty acids, and that are involved in the progressive replacement of sugar oxidation by fat oxidation, during stress and aging, and in degenerative diseases; for example, a process that inactivates the energy-producing pyruvate dehydrogenase in Alzheimer's disease has been identified (Ishiguro, 1998). Niacinamide, by lowering free fatty acids and regulating the redox system, supporting sugar oxidation, is useful in the whole spectrum of metabolic degenerative diseases.“ -Ray Peat

 

[Rinse & rePeat]

“There are many reasons to think that a slight reduction of serum phosphate would be beneficial. It has been suggested that eating fruit is protective against prostate cancer, by lowering serum phosphate (Kapur, 2000).” -Ray Peat

 

[Rinse & rePeat]

“A basic meaning of homeopathic medicine is the support of the organism's ability to heal itself; the essence of allopathy is that the physician fights "a disease" to cure the patient, e.g., by cutting out tumors or killing germs.” -Ray Peat

 

[Waynish]

Amazing that people who read this forum are still taking vaccines. There is no information deficiency... People just cannot see the signal through the noise.

 

[Rinse & rePeat]

Amazing that people who read this forum are still taking vaccines. There is no information deficiency... People just cannot see the signal through the noise.

I know right?! It seems to be a lower number of them on the forum getting them, compared to the rest of the world, according to my poll anyways, less than 14%.

The Real Vax Reason (Cast Your Vote)

I think so many scenarios fit the reason for the vaccine. I have my opinions what are yours?

raypeatforum.com

 

[Rinse & rePeat]

“Although the standard medical journals have only recently begun writing about it as a cancer treatment, emodin and related chemicals have been of interest as a non-toxic way to treat cancer, allergies, and viral and bacterial diseases for a long time.” -Ray Peat

 

[Rinse & rePeat]

“When a polycyclic bacterial antibiotic, adriamycyn (later called doxorubicin), was found to be too toxic to use as an antibiotic, the fact that it was toxic to cancer cells caused it to be developed as a cancer drug. It continued to be widely used even after it was found to cause heart failure in many of the “cured” patients, because of its “success” in killing cancer cells.

The fact that many kinds of cancer cells can be killed by emodin makes it slightly interesting as a cancer drug, but its simple generic nature has caused the drug industry to look for a more Ehrlichian magic bullet; for example, they are still looking for ways to keep doxorubicin from destroying the heart.

Emodin isn’t a magic bullet (in fact it isn’t a bullet/toxin of any sort), but when combined with all the other adaptogens, it does have a place in cancer therapy, as well as in treating many other ailments.“ —Ray Peat

 

[Rinse & rePeat]

“The outstanding physical-chemical property of bone is that it is a reservoir-buffer of carbon dioxide, able to bind huge amounts of the gas into its structure.

When carbon dioxide increases in the bloodstream it is at first absorbed rapidly by the bones, and if the blood level of CO2 is kept high day after day, the rate of absorption gradually slows down, but in experiments that have continued for several weeks the bones were still slowly absorbing more carbon dioxide; the absorption curve seems to be asymptotic. When people move to or from high altitudes, their bones appear to continue adapting to the different gas pressures for years. A reduction of atmospheric pressure (which allows the tissues to retain more carbon dioxide) helps to reduce the calcium loss caused by immobility (Litovka and Berezovs'ka, 2003; Berezovs'kyi, et al., 2000), and promotes the healing of damaged bone (Bouletreau, et al., 2002). Ultrasound treatment, which accelerates bone healing, stimulates processes similar to reduced oxygen supply (Tang, et al., 2007).

The mineral in newly formed bone is calcium carbonate, and this is gradually changed to include a large amount of calcium phosphate. Besides forming part of the mineral, carbon dioxide is also incorporated into a protein (in a process requiring vitamin K), in a process that causes this protein, osteocalcin, to bind calcium. The osteocalcin protein is firmly bonded to a collagen molecule. Collagen forms about 30% of the mass of bone; several percent of the bone consists of other organic molecules, including osteocalcin, and the rest of the mass of the bone consists of mineral.“ -Ray Peat

 

[Rinse & rePeat]

“Following the recognition that the SSRI drugs were causing osteoporosis, it was discovered that the serotonin produced in the intestine causes bone loss, and that inhibiting intestinal serotonin synthesis would stop bone loss and produce a bone building anabolic effect (Inose, et al., 2011). One group that had been concentrating on the interactions of genes commented that, recognizing the effects of intestinal serotonin, they had suddenly become aware of "whole organism physiology" (Karsenty and Gershon, 2011).

In previous newsletters I have talked about the ability of intestinal irritation and the associated increase of serotonin to cause headaches, asthma, coughing, heart and blood vessel disease, muscular dystrophy, flu-like symptoms, arthritis, inflammation of muscles and nerves, depression, and inflammatory brain diseases. With the new recognition that serotonin is a basic cause of osteoporosis, intestinal health becomes a major issue in aging research.“ -Ray Peat

 

[Rinse & rePeat]

“They noted that toxigenic C. difficile, the type of C. difficile that causes diarrhea, was absent in the samples that did not cause tumors, but was present in the samples that caused tumors in mice. When the researchers added this bacterium to the samples that originally did not cause tumors, it induced colon tumors in the mice. Further testing showed that C. difficile alone was sufficient to prompt tumor formation in the animal models.

Additional experiments led by co-author Nicholas Markham, M.D., Ph.D., assistant professor of medicine at Vanderbilt University Medical Center, and study co-leaders Franck Housseau, Ph.D., associate professor of oncology at Johns Hopkins, and Ken Lau, Ph.D., associate professor of cell and developmental biology and surgery at Vanderbilt University School of Medicine, showed that C. difficile brought about a range of changes within colon cells that made them vulnerable to cancer.

Cells exposed to this bacterium turned on genes that drive cancer and turned off genes that protect against cancer. These cells produced reactive oxygen species, unstable molecules that can damage DNA, and they also prompted immune activity associated with harmful inflammation.”

https://www.google.com/amp/s/scitechdaily.com/johns-hopkins-doctors-discover-that-a-common-infection-may-cause-cancer/amp/

 

[Rinse & rePeat]

“A mole rat has never been known to develop cancer. Their serum C-reactive protein is extremely low, indicating that they are resistant to inflammation. In humans and other animals that are susceptible to cancer, one of the genes that is likely to be silenced by stress, aging, and methylation is p53, a tumor-suppressor gene.

If the intrauterine experience, with low oxygen and high carbon dioxide, serves to “reprogram” cells to remove the accumulated effects of age and stress, and so to maximize the developmental potential of the new organism, a life that’s lived with nearly those levels of oxygen and carbon dioxide might be able to avoid the progressive silencing of genes and loss of function that cause aging and degenerative diseases.

Several diseases and syndromes are now thought to involve abnormal methylation of genes. Prader-Willi sydrome, Angelman’s syndrome, and various “autistic spectrum disorders,” as well as post-traumatic stress disorder and several kinds of cancer seem to involve excess methylation.

Moderate methionine restriction (for example, using gelatin regularly in the diet) might be practical, but if increased carbon dioxide can activate the demethylase enzymes in a controlled way, it might be a useful treatment for the degenerative diseases and for aging itself. “ -Ray Peat

 

[Rinse & rePeat]

“The study also discovered that the estrogen antagonist is effective against a multitude of other cancer types, which had long been considered non-endocrine tumors. In other words, virtually all major cancer types responsible for the majority of cancer deaths worldwide ARE in fact endocrine cancers and are driven primarily by estrogen signalling. Conversely, administering an estrogen antagonist (in this case ERX-11) may be a viable treatment for most cancers. Rather than using exotic and untested molecules such as ERX-11, interventions such as aspirin, progesterone, vitamins A/E/D/K, tetracycline antibiotics, etc are all viable anti-estrogenic interventions (especially when used in combination) and have the potential to treat 90%+ of the cancer types afflicting humans globally.”

An estrogen antagonist treats many cancers, including triple-negative breast cancer

Just a portion of the title is already paradoxical, according to mainstream medical dogma. Namely, how can an estrogen antagonist treat triple-negative breast cancer that is supposedly fully immune to any hormonal effects!? Well, maybe the whole idea of hormonal "receptors" as the only mechanism...

raypeatforum.com

 

[Rinse & rePeat]

“When cells are stressed or dying, they take up calcium, which tends to excite the cells at the same time that it inhibits their energy production, intensifying their stress. A cramp or a seizure is an example of uncontrolled cellular excitation. Prolonged excitation and stress contribute to tissue inflammation and fibrosis.

Gross calcification generally follows the fibrosis that is produced by inflammation.” -Ray Peat


“Nearly every type of tissue, including the brain, is susceptible to the inflammatory process that leads through fibrosis to calcification. The exception is the skeleton, which loses its calcium as the soft tissues absorb calcium.” -Ray Peat

 

[Rinse & rePeat]

“Increased carbon dioxide favors bone formation, and decreased bicarbonate favors the loss of calcium from bone (Canzanello, et al., 1995; Bushinsky, et al, 2001). The use of sodium bicarbonate can stimulate bone formation.” -Ray Peat