Comparison Between Good Quality Vitamin E Supplements

Logan-

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Logan-

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@haidut, apart from your own product, which one is better in your opinion? Which one would you buy?
 

Lucenzo01

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With the upgrade Tocovit is the patrician choice.
 

Lucenzo01

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Do you get different effects from it than other vitamin e products like vit e succinate, lotion crafter or healthnatura?
What effects do you get?
I have not ordered the new version yet. I have been using the old one with great results, much better than mainstream supps. The only supplement that come close is the Health Natura one.
 

Wagner83

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I have not ordered the new version yet. I have been using the old one with great results, much better than mainstream supps. The only supplement that come close is the Health Natura one.
Thanks. What do you notice from it?
 

Frankdee20

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How much E a week is okay before Testosterone gets lowered ? I am using it to improve Triglyceride profile.
 
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Logan-

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Ray had mentioned this company as a decent source of Vitamin E in an email.

He wouldn't say which one he uses, but hinted to mixed tocopherols with the highest delta.

Does anyone know which of their products has the highest delta?

Vitamin E | ADM

Open to anyone's experience with any of their Vitamin E sources as well.
 
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Logan-

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"The unique structural features of agents like a-TOS probably make them selective for malignant cells (Neuzil et al, 2001b). While speculative at present, it is possible that the surprisingly high toxicity of a-TOS towards cancer cells and tissues is due to their persistence in such cells in the original form, while normal cells, including fibroblasts (Roberg et al, 1999), cardiac myocytes (Roberg and Ollinger, 1998), hepatocytes (Tirmenstein et al, 1999) and intestinal epithelial cells (Borel et al, 2001), are all capable of its hydrolysis to a-TOH"

"There are several reports documenting that the VE analogue potentiates cancer cells killing by the immunological apoptogen Fas, viz by mobilising the Fas receptor from the cytosol to the plasma membrane; this has been shown for both prostate and breast cancer cells (Turley et al, 1997; Yu et al, 1999). These findings suggest that a-TOS has the propensity to boost the immune system to enhance cancer surveillance. While the Fas ligand itself is highly cytotoxic, the TNF-related apoptosis inducing ligand (TRAIL) is selective for cancer cells (Bonavida et al, 1999). Therefore, the reports that a-TOS can potentiate cancer cells to killing by TRAIL may be of pharmacological importance. This has been shown for both colon cancer (Weber et al, 2002) and mesothelioma cells (Neuzil et al, unpublished) as well as for T lymphoma cells (Dalen and Neuzil, 2003)"

"Finally, a-TOS can sensitise cancer cells by upregulation of the TRAIL death receptors, as in the TRAIL resistant mesothelioma cells (Neuzil et al, unpublished"

"Probably, the strongest evidence suggesting the potential use of VE analogues in the treatment of humans with neoplastic disease comes from the laboratory of Malafa. They first showed that a-TOS promoted beast cancer dormancy in an immunocompromised mouse (Malafa and Neitzel, 2000) and, later on, also melanoma dormancy (Malafa et al, 2002b); in both cases, inhibition of angiogenesis via suppression of VEGF signaling by a-TOS was suggested as the underlying mechanism, pointing to an indirect effect of the VE analogue on cancer growth."

"Perhaps, the most exciting aspect of the potential use of compounds like a-TOS, a pro-VE, follows from its pharmacokinetics. After infusion into the circulatory system, the VE analogue associates with circulating lipoproteins (Kayden and Traber, 1993; Pussinen et al, 2000). This carrier delivers it to the microvasculature of the tumour. Since there is a constant exchange of hydrophobic molecules between lipoproteins and the peripheral tissue, a-TOS can traverse to malignant cells, where it induces apoptosis (Neuzil, 2002). Due to rapid turnover of lipoproteins (Traber et al, 1994), a-TOS is eventually cleared in the liver. Hepatocytes have a high activity of nonspecific esterases that cleave the VE ester to a-TOH. Further, during processing of lipoproteins in the liver cells, the natural stereoisomer of a-TOH associates with the highly specific a-TOH-binding protein, which inserts this most active form of VE into nascent very low-density lipoprotein that is, in turn, rescreeted into circulation via the hepatic vein (Terasawa et al, 2000). In this way, following hydrolysis of a-TOS, the circulation is enriched with VE. Therefore, the pro-VE, a-TOS and similar compounds (Birringer et al, 2003) are converted to the redox-active VE with additional beneficial activity (Neuzil, 2002), including increased protection against oxidative stress and immunosuppressive activity (LiWeber et al, 2002). Moreover, several reports showed that a-TOS Vitamin E succinate and cancer J Neuzil 1824 British Journal of Cancer (2003) 89(10), 1822–1826 & 2003 Cancer Research UK protects cells like hepatocytes from secondary deleterious effects of toxic agents, including adriamycin (Dominguez-Rodriguez et al, 2001; Fariss et al, 2001)."

I would say Vitamin E Succinate is the way to go!

 
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Logan-

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So if going by these studies for cancer then alpha tocopherol succinate is likely optimal, followed by mixed tocopherols high in gamma, & dl-alpha tocopherol acetate form could have opposite effects. not sure about dl-alpha

 
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Logan-

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Check this out. It's the one I'm using right now:

PureBulk Supplements


 
EMF Mitigation - Flush Niacin - Big 5 Minerals

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