"Coconut Oil Is Pure Poison"

[Peater Piper]

So to see them print a scary headline with literally nothing in the article to even sort of back it up on a subject that they wouldn't even purport to know about?

That's the first thing I noticed. The article was pretty neutral regarding saturated fat, but the title was pure click-bait. Not good considering many people never read past the title.

 

[Kartoffel]

That's the first thing I noticed. The article was pretty neutral regarding saturated fat, but the title was pure click-bait. Not good considering many people never read past the title.

I don't wanna seem unsympathetic, but people that base their nutritional choices on headlines like this are not gonna make rational choices anyways, and are unlikely to critically consider scientific counterarguments.

 

[Sobieski]

She's right you know. I mean let's completely ignore how quickly the fat in coconut oil is metabolised as energy, at the end of the day it's saturated and as an oil it doesn't even contain the ESSENTIAL fats. Someone please pass me the rapeseed oil.

 

[Kartoffel]

She's right you know. I mean let's completely ignore how quickly the fat in coconut oil is metabolised as energy, at the end of the day it's saturated and as an oil it doesn't even contain the ESSENTIAL fats. Someone please pass me the rapeseed oil.

Did you even listen to one word that woman said? Flaxseed oil is clearly the best oil!

 

[x-ray peat]

Reminds me of the religion of global warming. Too many people outsource their thinking to the "experts."

 

[lvysaur]

A study published in the American Journal of Clinical Nutrition indicated that people who routinely consume cheese, whole milk, and other high-fat dairy products — in essence, products high in unsaturated fatty acids

Between this and the absolute garbage that passes as anthropology journalism, this is why I almost exclusively read papers

 

[Travis]

One of my favorite ways to refute this seething stupidness—the idea that 'saturated fats cause cardiovascular disease' party line—has perennially been to consider epidemiological studies of Ian Pryor. In these he reports his observations on the population consuming the most saturated fatty acids than anyone, indisputably: The Polynesians. Not only had the Tokelauans consumed ~60% of their energy as saturated fatty acids, via coconut, they had consumed only trace levels of ω−6 fatty acids. The pigs they would eat on rare occasions, ostensibly left by European sailors, haid been highly saturated with under 5% linoleic acid. The tropical environs enjoyed by Polynesian pigs' had surely helped in this regard, acting to maintain fluidity despite their lower unsaturation index—inducing lipid kinetic energy through ambient enthalpy.

Prior, Ian. "Cholesterol, coconuts, and diet on Polynesian atolls: a natural experiment: the Pukapuka and Tokelau island studies." The American journal of clinical nutrition (1981)​

Now the fanbois of Ancel Keys would have us believe that they'd be clogged silly with beaded strings of 16∶0 cholesterol conjugates and lipoprotein(a), yet they'd been entirely free of that concern. 'Cardiovascular disease' appears more like an umbrella term invoked to describe vascular pathology in general, one encompassing at least two main forms having distinct etiologies. Linus Pauling fully describes one of them, highlighting the organism's necessity to maintain high collagen synthesis rates in high-pressure arteries. The Pauling Paradigm is well-supported and highly logical, and also synergizes well with the sodium connection as blood pressure will increase vascular stress and collagen wear. Immunological mechanisms that damage collagen, such neutrophil attack, also overlay nicely and without conflict. This paradigm also best explains the small yet consistent epidemiological connection with cigarettes, and not cigars, as smokestream nitric oxide—from cigarettes' added ammonia—will reliably oxidize vitamin C into dehydroascorbate. Ascorbic acid has been shown to increase collagen synthesis rates in vitro by a factor of seven.

Pauling, Linus. "Solution to the puzzle of human cardiovascular disease: Its primary cause is ascorbate deficiency, leading to the deposition of lipoprotein (a) and fibrinogen/fibrin in the vascular wall." Journal of Orthomolecular Medicine (1991)

Membrane cholesterol ingression is not merely a function unsaturation. The most predictive parameter for membrane sterol incorporation is its DHA content. This is independent of fish consumption as trace levels found in leaves, fruit, and grass-fed animals have enough DHA precursor. Yet there is a wild card in this called ω−6 fatty acids, a generally pathological type which competes with our synthetic elongation/desaturation enzymes yielding osbond acid as proxy. Physicochemical investigations detailing membrane sterol concentration as a function of fatty acid consistently reveal the mechanism of cholesterol ingression: The ω−3 bond of DHA excludes sterols the best, by far, and so markedly that it's not a linear function of ω−3 chain length: a salient jump in sterol exclusion is noted when going from eicosapentaenoic acid (20∶5ω−3)—and osbond acid (22∶5ω−6)—to docosahexaenoic (22∶6ω−3). Our elongation/desaturation enzymes have a significant preference for ω−3 fatty acids, allowing ω−6 incorporation only when ratios diverge from unity. Physical properties of lipids combined with consensus enzymology would predict vascular cholesterol to be largely a function of ω−6/ω−3 ratio, the denominator of which increases as a function of latitude; the enzyme Δ¹²-desaturase is expressed in helminths, yeast, and temperate plants. By equating latitude with ω−6/ω−3 ratio, in turn a direct function of membrane sterols, cardiovascular disease of non-Pauling type would then increase as a function of both latitude and historical time—the latter being a consequent of the apho-equatorial migration of humans. Omega−6/ω−3 ratios of human diets had first strayed far beyond unity during Roman times (c. 100 AD), and had acquired a tens place weeks after Wesson Oil & Snowdrift Company had obtained their first seed press (c. 1887). Today in to 21st century, deep-Southerners eating subclinical scurvy-inducing cooked food diet having strictly-suicidal ω−6/ω−3 ratios boast some the highest rates of 'cardiovascular disease,' a fact having very little to do with saturated fatty acids.

Stillwell, William. "Docosahexaenoic acid: membrane properties of a unique fatty acid." Chemistry and physics of lipids (2003)

 

[inthedark]

I saw an Instagram post on this in which one of the commenters said basically "I don't use any oils that harden solid...I don't want it to solidify in my arteries". Further comments on the post range from anger to panic.

There's so much ignorance out there that articles like this just perpetuate and capitalize on.

Harley Pasternak MSc on Instagram: “Coconut oil has found a recent surge in popularity over the last five years with countless people purchasing countless products featuring…”

People just eat this stuff up without bothering -or maybe not having the time- to look into it further. And this "nutrition specialist" just spreading misinformation when he should know enough to research further into it.

 

[lampofred]

It seems like her argument is that there are studies showing lard increases heart disease risk and becauses coconut oil has more saturated fat than lard, it is more dangerous. She is assuming lard is mostly saturated even though lard is mostly unsaturated now because animals are fed unsaturated fats. If I'm understanding it correctly, there are so many logical mistakes in what she's saying. And people will just blindly believe her because of the Harvard name and how confidently she is saying what she's saying.

 

[Kartoffel]

One of my favorite ways to refute this seething stupidness—the idea that 'saturated fats cause cardiovascular disease' party line—has perennially been to consider epidemiological studies of Ian Pryor. In these he reports his observations on the population consuming the most saturated fatty acids than anyone, indisputably: The Polynesians. Not only had the Tokelauans consumed ~60% of their energy as saturated fatty acids, via coconut, they had consumed only trace levels of ω−6 fatty acids. The pigs they would eat on rare occasions, ostensibly left by European sailors, haid been highly saturated with under 5% linoleic acid. The tropical environs enjoyed by Polynesian pigs' had surely helped in this regard, acting to maintain fluidity despite their lower unsaturation index—inducing lipid kinetic energy through ambient enthalpy.

Prior, Ian. "Cholesterol, coconuts, and diet on Polynesian atolls: a natural experiment: the Pukapuka and Tokelau island studies." The American journal of clinical nutrition (1981)​

That doesn't refute anything! Those Polynesians are protected from their terrible diet by special genetics and the intense physical exercise they get every day. They often run for hours in order to hunt their tubers, fruits, and coconuts.

 

[LeeLemonoil]

@Kartoffel
Native angry Teuton here as well. Curiously, the YouTube-lecture of her was posted very recently on a German health-topics forum where I sometimes participate. We‘ve quickly dismissed and singled out her breathtaking ignorance. Problem is, not only is she partly Havard-schooled but currently the clinical head of a department in a rather well reputed German university hospital. That’s disheartening to find that level of imcoptence in such exalted and authoritative position. Well, who knows what motivates this beautiful and healthy looking dame.

@Travis
Thanks for the valuable information

 

[raypeatclips]

@Kartoffel
Well, who knows what motivates this beautiful and healthy looking dame.

€€€

 

[Kartoffel]

That’s disheartening to find that level of imcoptence in such exalted and authoritative position.

Well, she is Harvard schooled, I think it's as simple as that. I doubt she is smart enough to know better and to knowingly make false statements in order to protect her own interests. She's simply a mindless parrot. I think German doctors are even more intolerable and mind numbingly stupid than those in other countries. They simply repeat whatever the American associations are currently saying, and follow the guidlines of the DGE. I guess it's because it's German nature to blindly repeat what your superior says, and to get angry when anyone outside your institution dares to question any of your reasons.

Ich finde, man kann ziemlich klar erkennen, dass ihr Zorn bei "Kokosöl ist Gift" nicht gespielt ist. Die Frau glaubt ihren eigenen Mist selber, was man auch ganz gut an ihrem Äußeren erkennen kann.

 

[Sobieski]

That doesn't refute anything! Those Polynesians are protected from their terrible diet by special genetics and the intense physical exercise they get every day. They often run for hours in order to hunt their tubers, fruits, and coconuts.

Last bit got me. LOL

 

[Blossom]

She should follow Travis on patreon.

 

[danishispsychic]

Coconut oil does not agree with everyone- Butter works for me- Im typo O pos. blood. I have given up on ingesting it completely -

 

[raypeatclips]

Well, she is Harvard schooled, I think it's as simple as that. I doubt she is smart enough to know better and to knowingly make false statements in order to protect her own interests. She's simply a mindless parrot.

You don't become a Harvard professor of epidemiology by going against the grain.

 

[Arrade]

Lauric acid was active on 5alphaR1 (IC(50)=17+/-3 microg/ml) and 5alphaR2 (IC(50)=19+/-9 microg/ml)

Inhibition of type 1 and type 2 5alpha-reductase activity by free fatty acids, active ingredients of Permixon. - PubMed - NCBI

Coconut oil seems often to be anti-dht. I'm not supporting the notion of cardiovascular disease from it

 

[Kartoffel]

Coconut oil does not agree with everyone- Butter works for me- Im typo O pos. blood. I have given up on ingesting it completely -

How does this disagreement between coconut oil and yourself manifest itself?

 

[Kartoffel]

Lauric acid was active on 5alphaR1 (IC(50)=17+/-3 microg/ml) and 5alphaR2 (IC(50)=19+/-9 microg/ml)

Inhibition of type 1 and type 2 5alpha-reductase activity by free fatty acids, active ingredients of Permixon. - PubMed - NCBI

Coconut oil seems often to be anti-dht. I'm not supporting the notion of cardiovascular disease from it

You say coconut oil "seems often", and yet you cite one study that doesn't contain the word coconut even once. Just because some of the shorter SFA produce an inhibition in vitro doesn't mean that coconut fat as a whole would have any such effect. Compared to most PUFA, which have been shown to be much more potent 5-AR inhibtors, cocnut oil is probably very much pro-dht.