kineticz
Member
Understanding ketogenesis, mitochondria and ATP production
It has to be correct that protecting the myelin sheath in the brain is central to any other attempts to boost ATP and heal the body.
The brain cannot produce it's own ATP and is supplied by cells and dondrites via the spine, this is made of PUFA.
If people read my full post they would see how I made it clear that the brain supply of ATP is central to my hypothesis in relation to the way methylation switches to angiotensin and high homocysteine which causes heart disease. With the brain consuming so much ATP it would only take a small drop in supply for all other organs to go slow, and the heart to become stiff, the kidneys to become calcified.
tara said:post 113206I agree that having the CNS cells functioning well is centrally important.kineticz said:post 113204 Without cell consistency, specifically brain cell, consistency, everything else is arbitrary.
Exactly what the facts are about optimal cell membrane composition continues to be contentious, as far as I can tell. I'm sure you've read the story about the Burrs and William Brown, and Peat's take on degeneration and PUFA.kineticz said:post 113204 You can rely on thyroid hormone to promote mitochondrial biogenesis, and use sugar to tie up fats, but that doesn't optimise the PUFA membranes that exist in the brain. It's fact.
It has to be correct that protecting the myelin sheath in the brain is central to any other attempts to boost ATP and heal the body.
The brain cannot produce it's own ATP and is supplied by cells and dondrites via the spine, this is made of PUFA.
If people read my full post they would see how I made it clear that the brain supply of ATP is central to my hypothesis in relation to the way methylation switches to angiotensin and high homocysteine which causes heart disease. With the brain consuming so much ATP it would only take a small drop in supply for all other organs to go slow, and the heart to become stiff, the kidneys to become calcified.
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