Hairlosssucks
Member
- Joined
- Mar 15, 2018
- Messages
- 181
What I’m scared of is bone demineralisation and height loss
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He’s saying hcg before I do pct ie clomid or nolva to bring back my balls.
My dht is probably higher than its ever been body hair growth after losing weight has increased.
If I raise e via clomid does it stay raised?
I could try boron to raise e2, but studies indicates it takes around 2 weeks.
Body hair growth and clear semen are in no way indicative of high DHT.
You seem like all you want to hear is from your "buddy", and none of this advice on this forum, I don't know why anyone would keep replying to you
I agree.Get bloodwork done (T, free T, E2 Sensitive, DHT, Progesterone, Prolactin), by just throwing in drugs things will rather go worse than better.
I’m using it and it’s helped a lot with my menopause symptoms.I could try boron to raise e2, but studies indicates it takes around 2 weeks.
Hi. Why is HCG supressive? Even low doses? Seems to keep T levels up after supression and testicles workingI wouldn't listen to your buddy, HCG isn't an PCT drug, it's suppressive.
The new symptoms you described sound like low DHT.
Get bloodwork done (T, free T, E2 Sensitive, DHT, Progesterone, Prolactin), by just throwing in drugs things will rather go worse than better.
As soon as you go off HCG, your T levels will be worse than the baseline before. It mimics LH and in the process shuts down natural / endogenous LH production.Hi. Why is HCG supressive? Even low doses? Seems to keep T levels up after supression and testicles working
Hi. Why is HCG supressive? Even low doses? Seems to keep T levels up after supression and testicles working
Yeh I do. I’ve dropped weight and lost fat which means even less aromatisation. My t is higher but it’s not being converted. I think I’ve also damaged my hpta axis for being at low estrogen for so long now.
Any study on effects after?As soon as you go off HCG, your T levels will be worse than the baseline before. It mimics LH and in the process shuts down natural / endogenous LH production.
Suppression of HPTA due to:
- increased aromatase activity via increased testicular aromatase expression (high e2)
- negative feedback loop of LH, body shuts down endogenous production due to excess exogenous source
As soon as you go off HCG, your T levels will be worse than the baseline before. It mimics LH and in the process shuts down natural / endogenous LH production.
Suppression of HPTA due to:
- increased aromatase activity via increased testicular aromatase expression (high e2)
- negative feedback loop of LH, body shuts down endogenous production due to excess exogenous source
BTW, I would also appreciate studies on the different T esters and it's effects on supression.As soon as you go off HCG, your T levels will be worse than the baseline before. It mimics LH and in the process shuts down natural / endogenous LH production.
Suppression of HPTA due to:
- increased aromatase activity via increased testicular aromatase expression (high e2)
- negative feedback loop of LH, body shuts down endogenous production due to excess exogenous source
1mg of testosterone reaching blood is equally suppressive whether it's from ester or not. All TRT options will suppress LH to zero. If your blood levels raise LH drops. If LH doesn't drop you have absorption issues and you aren't getting the benefits of the medication either.BTW, I would also appreciate studies on the different T esters and it's effects on supression.
BTW, I would also appreciate studies on the different T esters and it's effects on supression.
Some also claim that recovery is faster if you use HCG to reverse/prevent testicle atrophy because they are then ready to just wait signal from pituitary. I think it's likely broscience as well, because the endogenous production is regulated by hypothalamus which is shut down by HCG.
There is certainly a crash from stopping HCG mono, it isn't instant like that. Maybe it is still faster than from atrophied testicles but many just do it wrong to begin with stopping cold turkey from enanthate etc. On the other hand you don't really lose anything from keeping balls online by low dose HCG during cycle/TRT or for the last few months before quitting at least.I am the one who claimed that, and I got it from Dr Rand McClain who is far from a bro scientist.
Pituitary signalling is instantaneous once the body senses a T or E deficiency, no matter how long the shutdown has lasted, it's not a gland that atrophies.
On the other hand, testicles are glands that atrophy when unused, which is the reason why it takes time to get them back to their full functionnality.
Did before, didn't find anything specifically regarding difference in supression time regarding esters, aside from obvious thing that undecanoate having a longer life obviously will have you on T effects for longer therefore you will have to wait more time for exogenous T to drop...Instead of asking for countless studies you can take your little fingers and go to the search bar of NCBI.
So I assume you find useful to always keep HcG administration if you are having hope to stop T supplementationI am the one who claimed that, and I got it from Dr Rand McClain who is far from a bro scientist.
Pituitary signalling is instantaneous once the body senses a T or E deficiency, no matter how long the shutdown has lasted, it's not a gland that atrophies.
On the other hand, testicles are glands that atrophy when unused, which is the reason why it takes time to get them back to their full functionnality.
As testosterone is capable of causing complete suppression without an ester it is indeed obvious that extending its half life is causing longer lasting suppression. Are you curious what would happen if esters without steroids are administred?Did before, didn't find anything specifically regarding difference in supression time regarding esters, aside from obvious thing that undecanoate having a longer life obviously will have you on T effects for longer therefore you will have to wait more time for exogenous T to drop...
I am the one who claimed that, and I got it from Dr Rand McClain who is far from a bro scientist.
Pituitary signalling is instantaneous once the body senses a T or E deficiency, no matter how long the shutdown has lasted, it's not a gland that atrophies.
On the other hand, testicles are glands that atrophy when unused, which is the reason why it takes time to get them back to their full functionnality.