- Sep 24, 2016
Yes, it increases fatty acids and causes a state of pseudo insulin resistance. At nighttime. For peripheral and skeletal healing. To spare glucose for the brain. Glucose is a poor substrate for nighttime skeletal healing. GH is doing exactly what it needs to do in that circumstance. That's the same time carnitine reaches its top importance.
This is not a focus on the thymus.
I already know about the problems with metformin, but simply knowing an AMPK inducer was for some reason involved in this is potentially important information for me.
Don't worry, I prefer to keep things nice and short too, that's why I'm not going full Travis on you (he would probably hate me even more right now for promoting methylation - whatever guys I'm just following the leads :) ).
That’s interesting. I was always puzzled that many substances that are pro-AMPK in the periphery are often pro-mTor in the brain through health-promoting in both in a regenerative context. This GH->sugar nighttime thingy seems to explain it in part. Thanks