Advice for hypertrophic cardiomyopathy

[gabys225]

I was diagnosed with hypertrophic cardiomyopathy (Left ventricle hypertrophy). I overdid liver for many years prior to this, and I believe that the excess vitamin A caused/causes a release of free iron from the tissues, which then proceeds to damage everything. This was verified with labs (both vA and high iron) so I began donating and had rapid improvement in fatigue/blood pressure, but the hypertrophy remains, and his since grown slightly.

The echo shows that the deposition of tissue is fibrous and not muscular, which I took to be good news, thinking this could be addressed with fibrinolytic enzymes.

I have combed through most of the forum posts and want to know if anyone knows of anything else that I can do, that I may be missing or overlooking.

-B vitamins, with extra thiamine
-Molecular hydrogen for hydroxyl radical quenching
-Ribose for the heart tissue
-EDTA (helped further bring my iron levels down safely, I am still continuing this)
-CoQ10 (although I may switch this to idebenone given the excess iron causes hypoxia and coq10 turns into an oxidant under such conditions)
-K2
-Aspirin
-Magnesium

Should I seek to lower FFA as well? What do you guys think? I know cardiomyopathies are energetic (as are all things!), my main concern is eliminating the excess fibrous tissue.


Thank you to anyone that responds.

 

[JohnHafterson]

You should do some reading on taurine and hypertrophy, some interesting research articles here for you:

taurine hypertrophy at DuckDuckGo

DuckDuckGo. Privacy, Simplified.

duckduckgo.com

 

[Peatness]

https://www.space-age.com/LeftVentricularHypertrophy.pdf

Treatment of hypertrophic cardiomyopathy with coenzyme Q10 - PubMed

Hypertrophic cardiomyopathy (HCM) is manifested by severe thickening of the left ventricle with significant diastolic dysfunction. Previous observations on the improvement in diastolic function and left ventricular wall thickness through the therapeutic administration of coenzyme Q10 (CoQ10) in...

pubmed.ncbi.nlm.nih.gov

Coenzyme Q10 attenuates diastolic dysfunction, cardiomyocyte hypertrophy and cardiac fibrosis in the db/db mouse model of type 2 diabetes - PubMed

These data demonstrate that coenzyme Q(10) attenuates oxidative stress and left ventricular diastolic dysfunction and remodelling in the diabetic heart. Addition of coenzyme Q(10) to the current therapy used in diabetic patients with diastolic dysfunction warrants further investigation.

pubmed.ncbi.nlm.nih.gov

Copper deficiency and heart disease: molecular basis, recent advances and current concepts - PubMed

Copper is an essential trace element and has profound influence on cardiac myopathy and heart metabolism. Dietary Cu restriction in rats results in cardiomyopathy, and affects the integrity of the basal lamina of cardiac myocytes and capillaries. Decreased levels of delta subunits of ATP...

pubmed.ncbi.nlm.nih.gov

Cardiovascular Disease from Copper Deficiency—A History

ABSTRACT. Although the nutritional essentiality of copper was established in 1928, a preoccupation with hematology delayed the discovery of cardiovascular disea

academic.oup.com

 

[gabys225]

Thank you Pina, thank you John for the thoughtful replies. I'll be reading up on all of this.

 

[peateats1]

Would estrogen play a role? I'm currently dealing with high diastolic and low pulse pressure, so this is really interesting. I had huge endotoxin, estrogen, and iron issues over the last two years.