Travis
Member
- Joined
- Jul 14, 2016
- Messages
- 3,189
Kartoffel, it is plainly obvious to me that you: (1) follow me around, (2) send me personal messages, and (3) harass me. You are insulting me with denials and you're attempts of framing me as delusional....paranoid...beeing stalked or insulted...hysterical...
So how does hyperglycemia cause tiredness in one instance, yet can cause agitation and excitability in another?
I think the receptor-displacement of ligands can actually explain paradoxical results, depending of course on whether or not the μ-receptor had been initially occupied by an opioid? Caffeoyl quinide could also block intestinal μ-receptors, known to be present and active in that location, thereby allowing increased passage of dietary-derived exorphins. The nullifying of high-affinity binding sites would logically increase the mobility of other ligands.
And I don't get angry at all alternate ideas, only when they are contrived just to spite me—as was obviously done in this case.
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