MaxVerstappen
Member
- Joined
- Sep 26, 2020
- Messages
- 59
I've been thinking a bit about the etiology of infections lately, but the axioms that germ theory is based upon, is derived from fraudulent science (1). And thus, one needs to start over.
In the case of tuberculosis, it is estimated that about 25% of the world population has the bacteria. A few percentage of these are actually sick (2). The disease doesn't really correlate with the bacteria. Weston Price showed that the younger generations of a modernized part of the Isle of Lewis were not showing the same resistance to tuberculosis as their ancestors (3).
Hans Selye showed that injecting toxic amounts of ACTH and cortisone induced an infection by bacteria considered non-pathogenic (the infection was prevented by injection of somatotropin (GH)) (4).
Broda Barnes showed that people with repeated infections can be treated with thyroid therapy.
Whats for sure is that bacterial infection is not necessarily contingent on the bacteria. It seems to be somewhat contingent on the endocrine system. What i further want to understand is: can a bacteria do different "jobs" if their environment is different? E.g. they have one function in presence of catabolism, and another function in presence of anabolism. Or is a proper endocrine function just responsible for inhibition of bacterial decomposition of organs?
I'd be glad if someone could send relevant litterature or input on this topic. I'd also like to understand the role of viruses and funguses, but relevant litterature is a little bit hard for me to find.
(1) THE DOCTOR'S WORLD; Revisionist History Sees Pasteur As Liar Who Stole Rival's Ideas (Published 1995)
(2) The Global Prevalence of latent tuberculosis: a systemic review and meta-analysis. The global prevalence of latent tuberculosis: a systematic review and meta-analysis
(3) Weston Price. Nutrition and Physical Degeneration. (Page 46).
(4) Hans Selye (1951). The Influence of STH, ACTH and Cortisone Upon Resistance to Infection.
In the case of tuberculosis, it is estimated that about 25% of the world population has the bacteria. A few percentage of these are actually sick (2). The disease doesn't really correlate with the bacteria. Weston Price showed that the younger generations of a modernized part of the Isle of Lewis were not showing the same resistance to tuberculosis as their ancestors (3).
Hans Selye showed that injecting toxic amounts of ACTH and cortisone induced an infection by bacteria considered non-pathogenic (the infection was prevented by injection of somatotropin (GH)) (4).
Broda Barnes showed that people with repeated infections can be treated with thyroid therapy.
Whats for sure is that bacterial infection is not necessarily contingent on the bacteria. It seems to be somewhat contingent on the endocrine system. What i further want to understand is: can a bacteria do different "jobs" if their environment is different? E.g. they have one function in presence of catabolism, and another function in presence of anabolism. Or is a proper endocrine function just responsible for inhibition of bacterial decomposition of organs?
I'd be glad if someone could send relevant litterature or input on this topic. I'd also like to understand the role of viruses and funguses, but relevant litterature is a little bit hard for me to find.
(1) THE DOCTOR'S WORLD; Revisionist History Sees Pasteur As Liar Who Stole Rival's Ideas (Published 1995)
(2) The Global Prevalence of latent tuberculosis: a systemic review and meta-analysis. The global prevalence of latent tuberculosis: a systematic review and meta-analysis
(3) Weston Price. Nutrition and Physical Degeneration. (Page 46).
(4) Hans Selye (1951). The Influence of STH, ACTH and Cortisone Upon Resistance to Infection.