Progesterone
Member
- Joined
- Feb 8, 2017
- Messages
- 1,553
All these drugs mane
Go big or go bald, beeyotch.
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All these drugs mane
Lol and here bodybuilders talk about the loss of vision and suicidal ideation from clomidI might be bigger and stronger than you
Yes, Clomid once a week works. The half life is stupid long. I think SERMs mostly work at the hair level thanks to raising SHBG a lot. I believe SHBG going low is among the worst things that could happen to your hair. Tapering off because there are better options that don't have zuclomifene.
I might be bigger and stronger than you
Yes, Clomid once a week works. The half life is stupid long. I think SERMs mostly work at the hair level thanks to raising SHBG a lot. I believe SHBG going low is among the worst things that could happen to your hair. Tapering off because there are better options that don't have zuclomifene.
Options for what, SHBG increase?
@Sourdoughbanana
Lol and here bodybuilders talk about the loss of vision and suicidal ideation from clomid
Toremifene did not enhance pulsatile or stimulated GH secretion, but decreased IGF-I by 20% in men and women. IGFBP3 was unchanged, whereas while IGFBP1 and SHBG increased in both sexes to a similar extent.
I took low dose toremifene and it made me compulsively suicidal. Never felt worse mentally in my entire existence, and the active life was like two weeksSHBG has a very high affinity for estradiol.
I want to stop SERMs eventually. I’ve been dealing with AAS so it’s all a normal post cycle therapy that I’m doing. Clomid for 8 weeks, then a pure antagonist tapered off. Anecdotally Nolvadex and Toremifene stop ALL hair shedding the same. I have Toremifene on hand.
I don’t think it matters what the bloodworks show regarding e2/DHT, what you care about is the free levels of those hormones and many others upstream like androstenedione etc. Low SHBG is awful.
Besides, there’s the IGF1/IGFBP3 topic which is discussed elsewhere. Randomly pulled one study on Toremifene since I’m taking it now:
Effects of Toremifene, a Selective Estrogen Receptor Modulator, on Spontaneous and Stimulated GH Secretion, IGF-I, and IGF-Binding Proteins in Healthy Elderly Subjects
This is all hair friendly.
SHBG has a very high affinity for estradiol.
I want to stop SERMs eventually. I’ve been dealing with AAS so it’s all a normal post cycle therapy that I’m doing. Clomid for 8 weeks, then a pure antagonist tapered off. Anecdotally Nolvadex and Toremifene stop ALL hair shedding the same. I have Toremifene on hand.
I don’t think it matters what the bloodworks show regarding e2/DHT, what you care about is the free levels of those hormones and many others upstream like androstenedione etc. Low SHBG is awful.
Besides, there’s the IGF1/IGFBP3 topic which is discussed elsewhere. Randomly pulled one study on Toremifene since I’m taking it now:
Effects of Toremifene, a Selective Estrogen Receptor Modulator, on Spontaneous and Stimulated GH Secretion, IGF-I, and IGF-Binding Proteins in Healthy Elderly Subjects
This is all hair friendly.
Do you even need a SERM post cycle if you just remove taper of testerone and have removed excess estrogen through an AI?
From my understanding it blocks estrogen in the testicles, but removing estrogen through AI accomplishes the same
I know that...Natural T production is shut down after a cycle..
Clomid is the perfect thing to begin and boost that natural T production again and kickstart it.
I know that...
You know what also suppresses T function? estrogen, which SERMS selectively block
So how much better is clomid vs. having normal estro levels when you quit AAS
You do realize the purpose of SERMS is to block estrogen from the testesUm...
Wat
You do realize the purpose of SERMS is to block estrogen from the testes
Does it? Or is only because people have high estro once they quit injecting the aromatising T?Why would recovering naturally without SERM be better after a cycle of T/steroids though?
It would just take much longer, wouldn't it?