Such_Saturation
Member
- Joined
- Nov 26, 2013
- Messages
- 7,370
I'm sure there is... doubt he took it.No reliable way to confirm slow MOA by testing?
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I'm sure there is... doubt he took it.No reliable way to confirm slow MOA by testing?
Vitamin B2 (riboflavin) increasea MAO-A pretty reliably since the enzyme used vitamin B2 as cofactor. Using the activated version R5P seems to work very well even at 10mg doses. The increase in MAO-A and subsequent drop in serotonin explains (IMO) why vitamin B2 treats migraines. FDA is considering approving B2 for migraines, but only in combination with "established" (read profitable) treatments like the *triptan drugs.
@such saturation: So which one is the best test for this?
The methylation/undermethylation people might have some indirect tests for that. Otherwise you would probably look for neurotransmitter metabolites in the blood. @whodathunkit @haidut
Enjoy the read https://tinyurl.com/ybkad4jzwoa, fast reply there!
Mh but neurotransmitter in blood? You have not been to convinced of this yourself, haven't you? Afaik it doesn't necessarily corelate with levels in the brain. Or do you think if one has a severe MAOa deficiency, he would definetly have raised NT levels everywhere? Wouldn't exclude NT's being high in the gut for other reasons though.
Another test I did, was stool testing or whatever you call this in english. Having too much calprotectin (but not as high like in celiac) points to a mild gut inflammation. On top of that I had 10x more Enterocoocus and 8x more E. Coli bacteria. This probably altering NT levels on its own. It definetly is increasing the histamine load.
Edit: And the bad bacteria would also increase beta-glucorunidase, making it even more difficult to excrete estrogens.
Enjoy the read https://tinyurl.com/ybkad4jz