bruschi11
Member
- Joined
- Dec 20, 2016
- Messages
- 514
I was planning to write out my full story here. It’s a lot. I’m 34 and I’m certain I have iron toxicity of the brain. It’s very possible it all stems from Cyanobacteria being in my stomach (which was in there for 6 months.) Killed it in late 2020. Cyano die off is bmaa which is attributed to the als- Parkinson’s dementia complex of Guam in 1900s. It’s a bad thing- destroys function of SOD1 (zn/cu) enzyme.
Do I have this? I don’t know. Zinc and copper still work in my body if methylation is working. I had a bad setback starting in August that got severe in September. It was mainly copper dumping that hurt me. Prior to the dumping- copper was best friend. Afterward it was torture until I got methylation working. But what happens then? By pushing methylation, NAC to clean up the toxic copper- I go zinc deficient. That was the very very worst thing that could’ve happened. But I’m getting it in. Just in a much worse body chemistry than before. The iron in system got deep in brain. It is a mess.
I’m in agreement with Ray that iron is the evil. It needs to be used by the body as shown by my many htmas. When iron is in range on htma- I am good. When low? I’m horrible. I believe that’s toxic unused iron building up when it’s low.
I am trying to understand iron metabolism. I think I’m putting it together decently. Zinc was the last piece of puzzle that makes a lot of sense here.
I see mainly you start with fe2 (ferrous) which converts to fe3 (iron) through copper. Fe3 actually protects against fe2. But when you have toxic unused copper? Fe3 + Cu2 (toxic) causes severe toxicity. I think this instance basically puts a stopgap on iron metabolism. And blocks the entire operation. So BOTH Fe2 and Fe3 build up.
Zinc and magnesium convert Fe3 to transferrin. Transferrin is the good guy binding Fe3 too. Transferrin is (good)cortisol essentially anti-inflammatory. We need transferrin and it’s the real hero. But transferring itself can still rise and cause issues. What binds transferring? Calcium and chromium. Two things that saved me in times past. Times when I used too much zinc prior.
I don’t have much to add here. It took me all this time to really put the above together. 2 years since it all began. Now I’m really getting shakes. Very minor. But it’s clearly becoming Parkinson’s if I don’t get ahold of this quickly.
Last week starting zinc, betaine hcl, and using binder (bentonite clay) really helped me. They were great at first. But since then a magnesium bath really crashed me. It was so bad. Next time I take mag will be orally.
I think zinc is missing piece in iron metabolism. But don’t go too high cuz it can hurt copper which is essential for fe2 to fe3. If anyone has anything to add, helpful advice I could use it.
I also think chelators/binders like cutlers approach, shoemaker, OSR. Many of the benefits are from binding iron. Heck look at cutler feeding all that zinc after. I had success with chelating and zinc early this summer. And bentonite on numerous occasions. I wonder why?!
I’m scared. Last spring it looked like I was getting better. I could go on about how I messed up and what things happened. But I will later. Im just more interested to talk about iron metabolism if anyone can chime in.
Do I have this? I don’t know. Zinc and copper still work in my body if methylation is working. I had a bad setback starting in August that got severe in September. It was mainly copper dumping that hurt me. Prior to the dumping- copper was best friend. Afterward it was torture until I got methylation working. But what happens then? By pushing methylation, NAC to clean up the toxic copper- I go zinc deficient. That was the very very worst thing that could’ve happened. But I’m getting it in. Just in a much worse body chemistry than before. The iron in system got deep in brain. It is a mess.
I’m in agreement with Ray that iron is the evil. It needs to be used by the body as shown by my many htmas. When iron is in range on htma- I am good. When low? I’m horrible. I believe that’s toxic unused iron building up when it’s low.
I am trying to understand iron metabolism. I think I’m putting it together decently. Zinc was the last piece of puzzle that makes a lot of sense here.
I see mainly you start with fe2 (ferrous) which converts to fe3 (iron) through copper. Fe3 actually protects against fe2. But when you have toxic unused copper? Fe3 + Cu2 (toxic) causes severe toxicity. I think this instance basically puts a stopgap on iron metabolism. And blocks the entire operation. So BOTH Fe2 and Fe3 build up.
Zinc and magnesium convert Fe3 to transferrin. Transferrin is the good guy binding Fe3 too. Transferrin is (good)cortisol essentially anti-inflammatory. We need transferrin and it’s the real hero. But transferring itself can still rise and cause issues. What binds transferring? Calcium and chromium. Two things that saved me in times past. Times when I used too much zinc prior.
I don’t have much to add here. It took me all this time to really put the above together. 2 years since it all began. Now I’m really getting shakes. Very minor. But it’s clearly becoming Parkinson’s if I don’t get ahold of this quickly.
Last week starting zinc, betaine hcl, and using binder (bentonite clay) really helped me. They were great at first. But since then a magnesium bath really crashed me. It was so bad. Next time I take mag will be orally.
I think zinc is missing piece in iron metabolism. But don’t go too high cuz it can hurt copper which is essential for fe2 to fe3. If anyone has anything to add, helpful advice I could use it.
I also think chelators/binders like cutlers approach, shoemaker, OSR. Many of the benefits are from binding iron. Heck look at cutler feeding all that zinc after. I had success with chelating and zinc early this summer. And bentonite on numerous occasions. I wonder why?!
I’m scared. Last spring it looked like I was getting better. I could go on about how I messed up and what things happened. But I will later. Im just more interested to talk about iron metabolism if anyone can chime in.