Giraffe
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Dr. Wolfgang Wodarg raised this question.
Corona viruses use spike proteins to get entry to the cell. The mRNA covid-'vaccines' are designed to make your cells produce something like these spike proteins, and this shall encourage the body to produce antibodies against those spike proteins. Antibodies (according to the theory) tag infected cells for attack by other cells of the immune system, and this is supposed to make you immunized against corona viruses.
So Dr. Wodarg said that mammals have incorporated viral genes. One of those produce a protein called syncytin, and syncytin is needed for the placenta. No syncytin = no pregnancy. He said that it has structural similarities to the spike protein of corona viruses. So he fears that antibodies to the spike protein could lead to a lack of syncytin thereby causing infertility. This needs to be ruled out, before this mRNA-thingy (called vaccine) is put on the market.
I googled a bit ... Syncytin or antibodies against it have been researched in a number of diseases.
Human endogenous retroviral syncytin exerts inhibitory effect on invasive phenotype of B16F10 melanoma cells
High prevalence of the antibody against Syncytin-1 in schizophrenia
Human endogenous retroviruses and multiple sclerosis: Innocent bystanders or disease determinants?
Corona viruses use spike proteins to get entry to the cell. The mRNA covid-'vaccines' are designed to make your cells produce something like these spike proteins, and this shall encourage the body to produce antibodies against those spike proteins. Antibodies (according to the theory) tag infected cells for attack by other cells of the immune system, and this is supposed to make you immunized against corona viruses.
So Dr. Wodarg said that mammals have incorporated viral genes. One of those produce a protein called syncytin, and syncytin is needed for the placenta. No syncytin = no pregnancy. He said that it has structural similarities to the spike protein of corona viruses. So he fears that antibodies to the spike protein could lead to a lack of syncytin thereby causing infertility. This needs to be ruled out, before this mRNA-thingy (called vaccine) is put on the market.
I googled a bit ... Syncytin or antibodies against it have been researched in a number of diseases.
Human endogenous retroviral syncytin exerts inhibitory effect on invasive phenotype of B16F10 melanoma cells
The finding of syncytin expression in some tumors implicates its potential values in the prognosis of cancers (16,19,20), since it is not expressed in normal tissues except placenta, testis, brain and osteoclasts (8-10). For example, the protein has been reported to be expressed in multiple sclerosis and leukemia, as well as in breast cancers (18,19,24,25). Yet it is still controversial whether syncytin expression represents positive or negative prognosis (18,20). In this study, we examined the impact of syncytin expression on the phenotypic and invasive properties of tumor cells using a subline of syncytin-expressing B16F10 cells as a model. Our results indicate that syncytin expression have changed the phenotype and significantly confined the proliferation and invasion of B16F10 melanoma cells in in vitro assays, suggesting that the expression of syncytin in some cancers may be a positive prognostic factor.
High prevalence of the antibody against Syncytin-1 in schizophrenia
Previous reports suggested a possible relationship between autoimmunity and SZ (Heath et al, 1967; Chen et al, 2012). In addition to the recent epidemiological studies (Benros et al, 2014; Cremaschi et al, 2017), a number of studies reported the presence of autoantibodies such as antinuclear antibodies and anti-N-methyl-D-aspartic acid receptor (NMDAR) antibodies in SZ patients (van Mierlo et al, 2015; Kristiansen et al, 2007; Coyle, 2006). On the other hand, patients with paraneoplastic syndrome associated with ovarian teratome exhibiting anti-NMDAR autoantibodies often manifest SZ-like symptoms such as hallucinations, delusions and thought disorders (Vitaliani et al, 2005). Moreover, the presence of neuro-reactive antibodies have been demonstrated in various diseases involving the neuromuscular system such as systemic lupus erythematousus (SLE) (Hanly et al, 1994), Guillain-Barre syndrome (Yuki et al, 2012) and myasthenia gravis (Patrick et al, 1973). The presence of anti-Syncytin-1 antibody in SZ patients suggests the involvement of autoimmune mechanism in its pathogenesis as well. However, although expression of HERV-W has been previously demonstrated, there is no significant difference in the extent of its expression among SZ, BD and healthy subjects (Karlsson et al, 2001). Moreover, the presence of anti-Syncytin-1 antibody does not necessarily satisfy the classical and the revised postulates defining the autoimmunity (Rose et al, 1993). It is also noted that various factors known to induce HERV-W expression, such as infection with influenza and herpes viruses, are also recognized as risk factors for SZ (Li et al, 2014; Nellåker et al, 2006). These findings collectively suggest the link between SZ and autoimmunity although its role in SZ pathogenesis is not as straightforward as in other autoimmune diseases such as SLE.
Human endogenous retroviruses and multiple sclerosis: Innocent bystanders or disease determinants?
Syncytin-1 expression is enhanced by various viruses including HSV-1 infection [138]. Yet induced expression of several HERV genes is observed in neuroinflammatory diseases including MS [69], suggesting that HERVs found in MS are a by-product of the inflammatory component of MS, diluting the contention of some researchers that HERVs are a causative pathogen of MS [201].