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4. The "isolated" polio viruses are artificial particles, generated by
suction of an indifferent mass through a very fine filter into a vacuum. Its structure (no characteristic structures) differ clearly from the ones of the "viruses" in the cells. Here the information is essential that a biochemical characterization of those "isolated" viruses, although "isolation" is claimed, has never been published anywhere nor has anybody even claimed such a characterization.
This thread fascinates me.... for anyone that might still be interested:
here is Dr. Dietrich Klinghardt discussing the release of human endogenous retroviruses in response to environmental stress (EMF, chemicals, metals)
I was listening to a recent episode of bulletproof radio (not a fan of Dave Asprey although he occasionally has interesting guests) and they were discussing a biotech company that synthesizes viruses that combat and kill cancer. They’re currently doing studies with dogs in which they inject the dogs with a virus that combats bone cancer specifically. I wasn’t aware that viruses could use code to kill malignant cells. Perhaps this is their primary purpose in a stress reaction?
If viruses have an endogenous origin, functioning as signaling and information transport agents, why does the body produce antigens against them? Most people have EBV-IgG but they don't have IL.6-IgG or TNF.a-IgG
Fascinating. Thank you for sharing the link! I could get lost for days on that siteTo answer your question, have you looked into Jamie Cunliffe's counter hypotheses to immune system theory? Namely the morphostatic hypothesis? He gives a more credible explanation as to why the body has a system against outside pathogenic factors.
This does not follow. Standard theory is that viruses are produced by cells inside the body. This does not stop them being infections and transmissible.
Parents didn't invent chicken pox parties for nothing.
Maybe there's some hypothetical possibility of some people being able to be so unstressed that they can resist all viral infections, but I'm not planning on being a guinea pig for those experiments.
If viruses have an endogenous origin, functioning as signaling and information transport agents, why does the body produce antigens against them? Most people have EBV-IgG but they don't have IL.6-IgG or TNF.a-IgG
I imagine: not everyone is equally vulnerable to all the same pathogens, some have more effective immune responses to them, and the quantity of exposure may affect whether or how severely the system is infected. Maybe the immune response depends on the current state of health, previous training of the immune system, and maybe other supportive lifestyle/environmental factors. Eg. higher body temps can support the immune system in resolving infections. Higher body temps can be affected by climate, season, living conditions, as well as base metabolism. Probably nutrition makes a difference - deficiencies may be weakening. The thymus - a key organ in the development of immune responses, is apparently weakened by stress.Then why did some kids not get it even when exposed to another kid with the pox?
I imagine: not everyone is equally vulnerable to all the same pathogens, some have more effective immune responses to them, and the quantity of exposure may affect whether or how severely the system is infected. Maybe the immune response depends on the current state of health, previous training of the immune system, and maybe other supportive lifestyle/environmental factors. Eg. higher body temps can support the immune system in resolving infections. Higher body temps can be affected by climate, season, living conditions, as well as base metabolism. Probably nutrition makes a difference - deficiencies may be weakening. The thymus - a key organ in the development of immune responses, is apparently weakened by stress.
Doesn't mean there are no infectious viruses, just that some people are more susceptible.
Has anyone ever seen microscopic slides of exosomes entering the cell rather than those of them exiting? If so why would that happen?
I can only speculate, but the exosomes that bud off a cell carry cell organelles, proteins or cellular material that other cells will take up. Supposedly exosomes are involved in cell-to-cell communication and if one cell is lacking something, it could signal to other cells that substance X is low. Conversely, if there is a surplus of something toxic in a cell, if that toxin is trapped in an extracellular vesicle that then interacts with another cell, it could damage that other cell or signal that other cell to produce defensive chemicals like prostaglandins or eicosanoids. I wonder if exosomes can "metastasize" or transfer cancer to other non-cancerous cells, in addition to changing the cellular field/environment.
Microscopy, as how it's done nowadays, tends to inanimate, kill or "freeze" the cell in a moment in time and even the light from the microscope can supposedly affect these cellular functions. Supposedly Dark Field Microscopy is superior for observing some of these phenomena because you don't have the factor of light affecting cellular behavior.
In this video they mention that mast cells produce exosomes, and this would likely INCREASE or spread the inflammatory reaction from an allergen. It's possible this is why someone might get hives that spread when they consume an allergen.
One of the female scientists studying her PHD states she saw proteins from Cell B on Cell A indicating a transfer of proteins happened. Maybe if a cell no longer has use for a protein it can "lend" the protein to another cell?