Follow along with the video below to see how to install our site as a web app on your home screen.
Note: This feature may not be available in some browsers.
Click Here if you want to upgrade your account
If you were able to post but cannot do so now, send an email to admin at raypeatforum dot com and include your username and we will fix that right up for you.
Nope, just some testimonials on the forum here.@Hans, you've mentioned before a few people getting 'destroyed' from eggshells. Were they clients? We need detailed experiences to understand what's going on.
No, not much.Noice. Do you think calcium malate would be a big improvement compared to calcium citrate?
Nothing, I get paid by the anti-carbonate industry and it's humiliating. However, I would probably promote their cause regardless of payment: why avoid alternatives that don't tax digestion (may already be compromised) when you can? Convenience is the best justification since the person has to be high to use it for systemic body alkalinization (it's crappy in this regard), can't argue that it's for fear of contaminants given that they can be homemade, and the ligands aren't useless, they help to enhance uptake, prevent adverse killcification, improve other aspects of metabolism that eventually reflect on killcium handling, and so on.Also, how much are you getting paid by the citric acid industry to say that?
I see. Thanks for answering. I tried calcium acetate and it burned my throat, so I went back to calcium citrate. Calcium citrate malate tasted pretty sour last time I took it( 3 years ago). Calcium citrate tastes quite neutral. I don't notice any sour taste from it( tasteless= useless? Perhaps tasteless= harmless?). About calcium carbonate, I think it would cause death by indigestion upon insistence, at least for me.No, not much.
You have more flexibility with basic molecules for increasing the possible fates and doing useful with them.. when things are operating right. When not, it's better to use those that are more specific to prevent them from being channeled to where you don't want. This should apply to organic acids.
It also depends on where they're preferentially metabolized once ingested in spite of the amounts being compatible with what cycles in the body.
But there has to be a reason why they started to produce killcium citrate malate instead of keeping it simple as one or the other. These start to taste unappleI mean, unappealing when too concentrated, may be to discourage the consumption of an amount that overwhelms the metabolism, making it preferable to distribute them at high intakes.
Taste is a reliable guide (when something is off). Killciol is tasteless, therefore useless. Unreacted eggshells taste like the remainings of a cadaver, it hints the victim where things are heading on insistence of ingestion.
I'm glad you disclosed that. What about fumaric acid?Nothing, I get paid by the anti-carbonate industry and it's humiliating. However, I would probably promote their cause regardless of payment: why avoid alternatives that don't tax digestion (may already be compromised) when you can? Convenience is the best justification since the person has to be high to use it for systemic body alkalinization (it's crappy in this regard), can't argue that it's for fear of contaminants given that they can be homemade, and the ligands aren't useless, they help to enhance uptake, prevent adverse killcification, improve other aspects of metabolism that eventually reflect on killcium handling, and so on.
Kombucha ist zo 2019, the latest trends are Calcicha and Limeaid: the reviewless beverages because it's the first and last time trying them. Both vinegar and lime juice require dilution to become palatable, it can be done with carbonated wasser, and then sweetened. Afterwards it's the fatal phase: experimenting adding killcium carbonate to preference (that is, how fast you want the fatality) prior to wasser.
I'm not aware of any reason to favor it, I would choose instead succinate or malate (at least it's found in substantial amounts in the diet). By basic I meant central in metabolism.I'm glad you disclosed that. What about fumaric acid?
Abstract said:Purpose
To examine the effects of 12 mo of resistance training (RT, 2x/wk, N= 19) or jump training (JUMP, 3x/wk, N= 19) on bone mineral density (BMD) and bone turnover markers (BTM) in physically active (≥4 hr/wk) men (mean age: 44 ± 2 y; median: 44 y) with osteopenia of the hip or spine.
Methods
Participants rated pain and fatigue following each RT or JUMP session. All participants received supplemental calcium (1200 mg/d) and vitamin D (10 μg/d). BMD was measured at 0, 6, and 12 mo using DXA scans of the whole body (WB), total hip (TH) and lumbar spine (LS). BTM and 25 OHD were measured by ELISA. The effects of RT or JUMP on BMD and BTM were evaluated using 3×2 repeated measures ANOVA (time, group). This study was conducted in accordance with the Declaration of Helsinki and was approved by the University of Missouri IRB.
Results
At baseline, 36 of 38 participants were vitamin D sufficient (25OHD>50 nmol/L); at 12 mo, all participants were 25OHD sufficient. 25OHD did not differ between groups. WB and LS BMD significantly increased after 6 months of RT or JUMP and this increase was maintained at 12 mo; TH BMD increased only in RT. Osteocalcin increased significantly after 12 mo of RT or JUMP; CTx decreased significantly after 6 mo and returned to baseline concentrations at 12 mo in both RT and JUMP. Pain and fatigue ratings after RT or JUMP sessions were very low at 0, 6, and 12 mo.
Conclusion
RT or JUMP, which appeared safe and feasible, increased BMD of the whole body and lumbar spine, while RT also increased hip BMD, in moderately active, osteopenic men.
Abstract said:Refeeding syndrome (RS) is a complex disease that occurs when nutritional support is initiated after a period of starvation. The hallmark feature is the hypophosphataemia, however other biochemical abnormalities like hypokalaemia, hypomagnesaemia, thiamine deficiency and disorder of sodium and fluid balance are common.
The incidence of RS is unknown as no universally accepted definition exists, but it is frequently underdiagnosed.
RS is a potentially fatal, but preventable, disorder. The identification of patients at risk is crucial to improve their management.
If RS is diagnosed, there is one guideline (NICE 2006) in place to help its treatment (but it is based on low quality of evidence).
The aims of this review are: highlight the importance of this problem in malnourished patients, discuss the pathophysiology and clinical characteristics, with a final series of recommendations to reduce the risk of the syndrome and facilitate the treatment.
Abstract said:We compared the effects of calcium carbonate and calcium citrate on markers of bone resorption in older postmenopausal women in an open-labeled crossover study. Forty women were randomized to receive 1000 mg/day of either calcium citrate or calcium carbonate [wasn't is elemental?] for 12 weeks, followed by a 2-week washout without calcium supplements and 12 weeks treatment with the alternate calcium supplement. All women received vitamin D (900 IU/day). Thirty-four women (25 Caucasian, nine Hispanic) completed the study. No significant differences in the decrease in parathyroid hormone (PTH) or bone specific alkaline phosphatase or the increase in urinary calcium/creatinine were detected between the two treatments. However, calcium citrate supplementation decreased the collagen cross-link resorption markers, urinary C-telopeptide (−31%), N-telopeptide (−30%), free deoxypyridinoline (−19%) and serum N-telopeptide (−8%), compared to no significant change following calcium carbonate supplementation (+2%, +3%, +2% and +2%, respectively; P<0.05). Calcium citrate decreased markers of bone resorption significantly more than calcium carbonate in postmenopausal women, although no differences in their effects in calcium excretion or PTH were detected.
Bone density can also be increased/maintained with sufficient protein, vitamin K2, silicon/monomethylsilanetriol, minimizing stress, net alkaline diet, etc..- 79. Parathyroid Hormone, Calcitonin, Calcium and Phosphate Metabolism, Vitamin D, Bone, and Teeth -- Guyton and Hall Textbook of Medical Physiology (9781455753451) | doctorlib.info
"Bone is deposited in proportion to the compressional load that the bone must carry. For instance, the bones of athletes become considerably heavier than those of nonathletes. Also, if a person has one leg in a cast but continues to walk on the opposite leg, the bone of the leg in the cast becomes thin and as much as 30 percent decalcified within a few weeks [?], whereas the opposite bone remains thick and normally calcified. Therefore, continual physical stress stimulates osteoblastic deposition and calcification of bone."
"Bone stress also determines the shape of bones under certain circumstances. For instance, if a long bone of the leg breaks in its center and then heals at an angle, the compression stress on the inside of the angle causes increased deposition of bone. Increased absorption occurs on the outer side of the angle where the bone is not compressed. After many years of increased deposition on the inner side of the angulated bone and absorption on the outer side, the bone can become almost straight, especially in children because of the rapid remodeling of bone at younger ages."
"Even the slightest decrease in calcium ion concentration in the extracellular fluid causes the parathyroid glands to increase their rate of secretion within minutes; if the decreased calcium concentration persists, the glands will hypertrophy, sometimes fivefold or more. For instance, the parathyroid glands become greatly enlarged in rickets, in which the level of calcium is usually depressed only a small amount. They also become greatly enlarged in pregnancy, even though the decrease in calcium ion concentration in the mother’s extracellular fluid is hardly measurable, and they are greatly enlarged during lactation because calcium is used for milk formation."
"Conversely, conditions that increase the calcium ion concentration above normal cause decreased activity and reduced size of the parathyroid glands. Such conditions include (1) excess quantities of calcium in the diet, (2) increased vitamin D in the diet, and (3) bone absorption caused by factors other than PTH (e.g., bone absorption caused by disuse of the bones)."
Abstract said:Despite the previously reported health benefits of calcium intake for the attenuation of metabolic disease, few studies have investigated the relationships among calcium intake, gut microbiota, and host metabolism. In this study, we assessed the effects of calcium supplementation on host microbial community composition and metabolic homeostasis. Mice were fed a high-fat diet with different calcium concentrations (4 and 12 g/kg) of 2 calcium supplements, calcium carbonate and calcium citrate. Supplementation with the higher concentration of calcium citrate significantly prevented body weight gain and decreased plasma biomarkers for metabolic disorder compared to calcium carbonate supplementation. Both calcium supplementation led to changes in microbial composition, increased propionate production and increased anorexigenic GLP-1 gene expression. The calcium citrate groups also experienced less metabolic endotoxemia. Our findings suggested that calcium supplementation could ameliorate host metabolic disorder caused by a high-fat diet, due to gut microbiota changes as well as decreased intestinal inflammation.