Colin Nordstrom
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- Joined
- Jul 4, 2017
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1) The adipocytes are not necessarily produce all of, or even most of, the cytokines found there. Most of those proteins are likely produced by macrophages that have infiltrated the tissue. This suggests that an inflammatory process is occurring to draw them in and activate them, which may be caused by the presence of the fat itself, or by something else that also causes the deposition of large amounts of fat into the tissue depot.Don't really understand your statement. Visceral fat produces all the inflammatory adipokines. I don't think I'm making that up. That is proven anatomical science. How can you see a deeper cause than metabolism? PUFAs are being ingested, they are being stored (somewhere), and they are contributing to hormonal/metabolic disturbances. Please enlighten me on how this is a symptom and not the cause.
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And milk has more SFA than fat depot in the animal's body, that's interesting.One other point, when it is said that a food is a "good source of saturated fat" or PUFA or whatever, it doesn't mean it's 99% that type of fatty acid. A "balanced" profile would be one-third each type, PUFA, MUFA, and SFA, right? Coconut oil is very very high in SFA, well over 90%, but most "saturated fats" are nowhere near that percentage. Many of them have as much or more MUFA as SFA, and pork these days has as much linoleic acid as the entire SFA contribution combined. Beef fat usually has little PUFA, in the single digits percent, but it has basically an equal percentage of MUFA and SFA. Grass fed beef has slightly more SFA and slightly less unsaturated, down to the low single digits, but it isn't a huge difference unless you are looking at specific PUFA species.
This is the same when fat depots are reported. You have to find the raw numbers or percentages, and not trust when someone reports that a fat depot is "mostly saturated" or "mostly unsaturated." That could mean 40% SFA, since that would only leave 30 and 30 for MUFA and PUFA.
It's a slippery language situation.
That's my question for a while,The best case scenario if you have a lot of PUFA coming in is that you store it in adipocytes and then slowly oxidize it in the lipid droplets for local energy without lipolyzing and releasing it into the blood again.
Any tissue that can oxidize fatty acids can use local fatty acids, I was referring to lipolysis of lipid droplets and the export of fatty acids from adipocytes to blood. Non-adipocyte cells don't store fatty acids the same way, so they are relatively free to be oxidized or are in some kind of structural complex like phospholipid.That's my question for a while,
So we are able to use fat without lipilysis and liberation of fatty acids into blood stream?
Or is that only for adipocytes? Other cells, muscles, can't use the fat locally without lipolysis?!
Ray says muscles use fat when they are resting , they absorb free fatty acids from blood or they use local fats near them?