Treatment for COVID-19 using Methylene Blue

md_a

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Joined
Aug 31, 2015
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468
Using Methylene Blue in nebulized form for treating patients with Coronavirus (Covid-19)

As COVID-19(Coronavirus) ravages the world, a quick, novel, cost effective cure for this malaise needs to be found.

Being a pulmonologist (lung specialist) in India, I have been treating pneumonia, other respiratory ailments and Tuberculosis patients (with XDR & MDR TB) for more than 42 years.

I’ve achieved remarkable success in treating my patients with Methylene Blue and with documented evidence. When used in sub lingual and nebulised form M.B. helps remarkably in clearing alveolar capillary block.

Covid causes a silent hypoxia wherein patients despite having acute hypoxia (low oxygen saturation spO2) show no clinical symptoms. Patient is not dyspnoeic in spite of having very low oxygen saturation. These soon escalate to a cytokine storm and patient goes in the irreversible phase of the respiratory distress.

Covid patients show resemblance to methemoglobinemia. MB is considered an approved drug of choice for methemoglobinemia. Additionally, M.B. has a strong antifibrotic action and is very fast acting.

Given the above presentations, Methylene Blue can be used as a treatment to all Coronavirus symptomatic patients & as a prophylactic drug to all vulnerable population. Used in low dosage (as prescribed below) it shows no significant side effects (less than 2% of patients complained of irritation in the nose/nausea for a day which subsided on its own). Given its low cost, it is also a viable regime for poorer countries like India. A number of patients(with varying levels of respiratory distress) have been treated using MB in nebulised/sub-lingual for over the course of my years of practice.

Potential Treatment regime for the novel Coronavirus (Covid-19)

Primary course of administration is in Nebulized form and Sublingually together. For patients with severe Cyanosis (oxygen levels less than 85%), IV may be required based on the clinical condition of the patient.

Administration steps as below (under medical supervision only as MB is known to be toxic in incorrect dosages)

  • 0.1% Methylene Blue to be administered as inhalation through a nebulizer (Primary line of treatment)
  • Nebulization to be given through a nasal mask
  • Sublingual administration will be as a supportive line of treatment
Administration of Methylene Blue in nebulized form

Administration of Methylene Blue in nebulized form

A person wearing a nebulizer and ideal way
Post my recommendations, a hospital in Mumbai started administering Methylene Blue through Nebulizer as well as sublingually on COVID-19 confirmed patients with positive results.

Methylene blue (Methylthioninium Chloride) is a thiazine dye & a medication. It is easily available in drug stores across India. It is safe to consume in low dosage (0.1 mg/kg in IV form) under proper medical supervision. It was the first antiseptic dye to be used therapeutically and it is present on WHO Model List of Essential Medicines. In fact, its use was widespread even before the advent of sulphonamides and penicillin. Its benefit is it rapidly arrests fibrosis which proves beneficial against virulent strains.

[Update: on why MB works]

There are multiple reasons why MB is effective; the chief is Nitric Oxide mediated reduction in inflammatory cascades in addition to inhibiting canonical inflammasomes.

Radiologically (i.e. X Ray) Covid/Swine Flu are similar & resemble any acute viral/fungal loefflers or aspiration pneumonia. Clinical distinction can be made by giving intranasal oxygen — you’d notice an improvement in normal pneumonia but not for Swine Flu or Covid-19 as these viruses would lead to alveolar capillary blockage (i.e. block ability of the lungs to operate in simple words). MB clears the blockage within 24 hours and O2 levels start improving. Covid-19 is a more virulent strain compared to H1N1 and a fast acting drug is necessitated.

Due to the clinico/pathological resemblance between H1N1 & Covid-19 — final outcome is expected to be similar post administration of MB.

1*yB4L1aCRTh9nk7lmOudX9Q.jpeg

1*yB4L1aCRTh9nk7lmOudX9Q.jpeg

(The above comparative X Rays are a part of a series. X Rays shared post consent from the patient)

Note : Some readers have questioned why it took 4 year to clear the lesions — given the state of the patient when he came to me, the chances of survival more than 6 months were less than 10% as his lung was severely damaged(X ray 1). In medical parlance, the above X Ray (X Ray 2) progress is considered a miracle.

(Will post more details about this as and when I get time — for queries please email me)

The detailed treatment regimen can be found by clicking here.

Edit 1
Note :
We continue to see very good results with Methylene Blue nebulization/sublingual administration. Given the positive results we see, we firmly believe that Methylene Blue is the cost effective treatment of choice in these trying times.
We have received complaints that some organizations/people are using Dr Golwalkars’ name to discredit Methylene Blue for their own benefits. Please note that we continue to see very good results with Methylene Blue and continue to advise its administration.
Comment below or mail me @ [email protected]


View: https://dr-deepak-golwalkar.medium.com/treatment-for-covid-19-using-methylene-blue-d23fc5a31a4d
 

Blossom

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This is awesome! Thank you so much.
 

AndrewGesell

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Using Methylene Blue in nebulized form for treating patients with Coronavirus (Covid-19)

As COVID-19(Coronavirus) ravages the world, a quick, novel, cost effective cure for this malaise needs to be found.

Being a pulmonologist (lung specialist) in India, I have been treating pneumonia, other respiratory ailments and Tuberculosis patients (with XDR & MDR TB) for more than 42 years.

I’ve achieved remarkable success in treating my patients with Methylene Blue and with documented evidence. When used in sub lingual and nebulised form M.B. helps remarkably in clearing alveolar capillary block.

Covid causes a silent hypoxia wherein patients despite having acute hypoxia (low oxygen saturation spO2) show no clinical symptoms. Patient is not dyspnoeic in spite of having very low oxygen saturation. These soon escalate to a cytokine storm and patient goes in the irreversible phase of the respiratory distress.

Covid patients show resemblance to methemoglobinemia. MB is considered an approved drug of choice for methemoglobinemia. Additionally, M.B. has a strong antifibrotic action and is very fast acting.

Given the above presentations, Methylene Blue can be used as a treatment to all Coronavirus symptomatic patients & as a prophylactic drug to all vulnerable population. Used in low dosage (as prescribed below) it shows no significant side effects (less than 2% of patients complained of irritation in the nose/nausea for a day which subsided on its own). Given its low cost, it is also a viable regime for poorer countries like India. A number of patients(with varying levels of respiratory distress) have been treated using MB in nebulised/sub-lingual for over the course of my years of practice.

Potential Treatment regime for the novel Coronavirus (Covid-19)

Primary course of administration is in Nebulized form and Sublingually together. For patients with severe Cyanosis (oxygen levels less than 85%), IV may be required based on the clinical condition of the patient.

Administration steps as below (under medical supervision only as MB is known to be toxic in incorrect dosages)

  • 0.1% Methylene Blue to be administered as inhalation through a nebulizer (Primary line of treatment)
  • Nebulization to be given through a nasal mask
  • Sublingual administration will be as a supportive line of treatment
Administration of Methylene Blue in nebulized form

Administration of Methylene Blue in nebulized form

A person wearing a nebulizer and ideal way
Post my recommendations, a hospital in Mumbai started administering Methylene Blue through Nebulizer as well as sublingually on COVID-19 confirmed patients with positive results.

Methylene blue (Methylthioninium Chloride) is a thiazine dye & a medication. It is easily available in drug stores across India. It is safe to consume in low dosage (0.1 mg/kg in IV form) under proper medical supervision. It was the first antiseptic dye to be used therapeutically and it is present on WHO Model List of Essential Medicines. In fact, its use was widespread even before the advent of sulphonamides and penicillin. Its benefit is it rapidly arrests fibrosis which proves beneficial against virulent strains.

[Update: on why MB works]

There are multiple reasons why MB is effective; the chief is Nitric Oxide mediated reduction in inflammatory cascades in addition to inhibiting canonical inflammasomes.

Radiologically (i.e. X Ray) Covid/Swine Flu are similar & resemble any acute viral/fungal loefflers or aspiration pneumonia. Clinical distinction can be made by giving intranasal oxygen — you’d notice an improvement in normal pneumonia but not for Swine Flu or Covid-19 as these viruses would lead to alveolar capillary blockage (i.e. block ability of the lungs to operate in simple words). MB clears the blockage within 24 hours and O2 levels start improving. Covid-19 is a more virulent strain compared to H1N1 and a fast acting drug is necessitated.

Due to the clinico/pathological resemblance between H1N1 & Covid-19 — final outcome is expected to be similar post administration of MB.

1*yB4L1aCRTh9nk7lmOudX9Q.jpeg

1*yB4L1aCRTh9nk7lmOudX9Q.jpeg

(The above comparative X Rays are a part of a series. X Rays shared post consent from the patient)

Note : Some readers have questioned why it took 4 year to clear the lesions — given the state of the patient when he came to me, the chances of survival more than 6 months were less than 10% as his lung was severely damaged(X ray 1). In medical parlance, the above X Ray (X Ray 2) progress is considered a miracle.

(Will post more details about this as and when I get time — for queries please email me)

The detailed treatment regimen can be found by clicking here.


Comment below or mail me @ [email protected]


View: https://dr-deepak-golwalkar.medium.com/treatment-for-covid-19-using-methylene-blue-d23fc5a31a4d

That's incredible, such amazing work. Thank you so much for sharing. I need to find a nebulizer then.

Does the article imply that O2 does not work on covid pneumonia patients at all?
 
OP
md_a

md_a

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Joined
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Messages
468
That's incredible, such amazing work. Thank you so much for sharing. I need to find a nebulizer then.

Does the article imply that O2 does not work on covid pneumonia patients at all?
BW: Now I remember you once telling me that at one point there was something called "firemen's oxygen" in which they had about 6 - 8 % carbon dioxide in the oxygen.
RP: Yeah, Yandell Henderson was the physiologist who got that popular to the point that fire departments all over the country started using it because the carbon dioxide at that very high concentration is extremely effective for restoring respiration to suffocated people, and for babies too that don't breathe properly.
BW: Is anyone still using carbon dioxide. Isn't it used or was used at some points in operation rooms?
RP: Oh, every now and then there is some person who says that hospitals are killing patients by giving them pure oxygen and say they would wish that hospitals switch, start using 5-8 % carbon dioxide whenever they give oxygen because it's basically stress inducing and toxic to give people oxygen in excess. At sea level we really have an excess of oxygen.
The Treatment of Pneumonia by Inhalation of Carbon Dioxide (Yandell Henderson)

"Nature has provided the lungs with several protective devices and reactions. The most obvious is the cough reflex by which irritating foreign bodies are removed. Less obvious, but more constantly acting, are the movements of respiration which are probably accompanied by peristaltic contractions and relaxations of the air tubes. The mucosa lining these tubes bears cilia which produce a continual flow of secretion from the depths of the lungs outward."

"Occlusion of an air tube puts all of these mechanisms for the clearing of the lungs out of action. The air in the occluded portion of the lung is soon absorbed, and the alveoli are deflated and collapsed. They are then gradually filled by accumulation of secretion. The conditions resulting are in all respects favorable to the development of microorganisms and, correspondingly, unfavorable both to the general defenses of the body and the special defenses of the lungs. It is a highly significant fact, as revealed by experiment, that in order to induce pneumonia in dogs it is not enough merely to introduce the pathogenic organisms into the lungs ; it is essential also to narcotize the animals so deeply that the cough reflex is abolished and respiration is depressed. In general, depressed or shallow breathing tends to permit the development of pneumonia, and deep breathing with full ventilation of the lungs tends strongly to inhibit it."

"In pneumonia it is the blocking of the lung airways, bronchi or bronchioli, by plugs of thick and sticky secretion which is the critical morbidic factor producing atelectasis [= collapse or closure of a lung resulting in reduced or absent gas exchange] and the conditions characteristic of an undrained infection." Inhalation of carbon dioxide results in opening up of pneumonic lungs."



158193817_1141212112974329_4360876660794958114_n.jpg
 

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Jam

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The detailed treatment regimen can be found by clicking here.
Dosage for Prophylaxis (i.e. for prevention)

MB shows best results as a prophylactic & should be considered as the primary line of treatment in the general unaffected population.
  • Sublingual administration of 0.1% w/v concentration daily (till the pandemic lasts) — swallow what is not absorbed in approx. 30 seconds (2.5 ml) OD
    • OR : If possible; 1.25 ML twice a day

FWIW, this equates to 2.5mg of methylene blue, or 6-7 drops of Oxidal.
 

J.R.K

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Using Methylene Blue in nebulized form for treating patients with Coronavirus (Covid-19)

As COVID-19(Coronavirus) ravages the world, a quick, novel, cost effective cure for this malaise needs to be found.

Being a pulmonologist (lung specialist) in India, I have been treating pneumonia, other respiratory ailments and Tuberculosis patients (with XDR & MDR TB) for more than 42 years.

I’ve achieved remarkable success in treating my patients with Methylene Blue and with documented evidence. When used in sub lingual and nebulised form M.B. helps remarkably in clearing alveolar capillary block.

Covid causes a silent hypoxia wherein patients despite having acute hypoxia (low oxygen saturation spO2) show no clinical symptoms. Patient is not dyspnoeic in spite of having very low oxygen saturation. These soon escalate to a cytokine storm and patient goes in the irreversible phase of the respiratory distress.

Covid patients show resemblance to methemoglobinemia. MB is considered an approved drug of choice for methemoglobinemia. Additionally, M.B. has a strong antifibrotic action and is very fast acting.

Given the above presentations, Methylene Blue can be used as a treatment to all Coronavirus symptomatic patients & as a prophylactic drug to all vulnerable population. Used in low dosage (as prescribed below) it shows no significant side effects (less than 2% of patients complained of irritation in the nose/nausea for a day which subsided on its own). Given its low cost, it is also a viable regime for poorer countries like India. A number of patients(with varying levels of respiratory distress) have been treated using MB in nebulised/sub-lingual for over the course of my years of practice.

Potential Treatment regime for the novel Coronavirus (Covid-19)

Primary course of administration is in Nebulized form and Sublingually together. For patients with severe Cyanosis (oxygen levels less than 85%), IV may be required based on the clinical condition of the patient.

Administration steps as below (under medical supervision only as MB is known to be toxic in incorrect dosages)

  • 0.1% Methylene Blue to be administered as inhalation through a nebulizer (Primary line of treatment)
  • Nebulization to be given through a nasal mask
  • Sublingual administration will be as a supportive line of treatment
Administration of Methylene Blue in nebulized form

Administration of Methylene Blue in nebulized form

A person wearing a nebulizer and ideal way
Post my recommendations, a hospital in Mumbai started administering Methylene Blue through Nebulizer as well as sublingually on COVID-19 confirmed patients with positive results.

Methylene blue (Methylthioninium Chloride) is a thiazine dye & a medication. It is easily available in drug stores across India. It is safe to consume in low dosage (0.1 mg/kg in IV form) under proper medical supervision. It was the first antiseptic dye to be used therapeutically and it is present on WHO Model List of Essential Medicines. In fact, its use was widespread even before the advent of sulphonamides and penicillin. Its benefit is it rapidly arrests fibrosis which proves beneficial against virulent strains.

[Update: on why MB works]

There are multiple reasons why MB is effective; the chief is Nitric Oxide mediated reduction in inflammatory cascades in addition to inhibiting canonical inflammasomes.

Radiologically (i.e. X Ray) Covid/Swine Flu are similar & resemble any acute viral/fungal loefflers or aspiration pneumonia. Clinical distinction can be made by giving intranasal oxygen — you’d notice an improvement in normal pneumonia but not for Swine Flu or Covid-19 as these viruses would lead to alveolar capillary blockage (i.e. block ability of the lungs to operate in simple words). MB clears the blockage within 24 hours and O2 levels start improving. Covid-19 is a more virulent strain compared to H1N1 and a fast acting drug is necessitated.

Due to the clinico/pathological resemblance between H1N1 & Covid-19 — final outcome is expected to be similar post administration of MB.

1*yB4L1aCRTh9nk7lmOudX9Q.jpeg

1*yB4L1aCRTh9nk7lmOudX9Q.jpeg

(The above comparative X Rays are a part of a series. X Rays shared post consent from the patient)

Note : Some readers have questioned why it took 4 year to clear the lesions — given the state of the patient when he came to me, the chances of survival more than 6 months were less than 10% as his lung was severely damaged(X ray 1). In medical parlance, the above X Ray (X Ray 2) progress is considered a miracle.

(Will post more details about this as and when I get time — for queries please email me)

The detailed treatment regimen can be found by clicking here.


Comment below or mail me @ [email protected]


View: https://dr-deepak-golwalkar.medium.com/treatment-for-covid-19-using-methylene-blue-d23fc5a31a4d

I wonder if Methylene Blue would be effective in clearing the spike protein in vaccinated people?
 
OP
md_a

md_a

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Messages
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I wonder if Methylene Blue would be effective in clearing the spike protein in vaccinated people?

Methylene Blue Inhibits the SARS-CoV-2 Spike–ACE2 Protein-Protein Interaction–a Mechanism that can Contribute to its Antiviral Activity Against COVID-19​


In conclusion, screening of our organic dye-based library identified MeBlu as a low-micromolar inhibitor of the interaction between SARS-CoV-2 spike protein and its cognate receptor ACE2, a PPI that is the first critical step initiating the viral entry of this coronavirus. While MeBlu shows strong polypharmacology and might be a somewhat promiscuous PPI inhibitor, its ability to inhibit this PPI could contribute to the antiviral activity of MeBlu against SARS-CoV-2 even in the absence of light making this inexpensive and widely available drug potentially useful in the prevention and treatment of COVID-19 as an oral or inhaled medication.

 
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md_a

md_a

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Did you ever look into hesperidin for effects on ace2?
"The role of our endogenous DNA mobile elements in stress, aging, and inflammation, and their structural similarity to viruses, suggests that substances that are helpful for stress-induced inflammation could be helpful for viral infections, and vice versa. All of the effective treatments for Covid-19 have had antiinflammatory effects—angiotensin blockers, cinanserin (a serotonin blocker), antihistamines, naringenin, hesperidin, fisetin and other flavonoids (Clementi, et al., 2021; Bellavite and Donzelli, 2020; Mishra, et al., 2020; Jo, et al., 2020), progesterone (Jakovac, 2020), and calcium channel blockers (Solaimanzadeh, 2020; Jayaseelan VP and Paramasivam, 2020).
These substances reduce intracellular calcium and increase carbon dioxide in relation to oxygen and lactate, with the effect of reducing random and meaningless excitation and activation of tissues, making energy available for purposeful organized activities of the organism." Ray Peat


View: https://www.youtube.com/watch?v=a7flR7Xx4As


............

HESPERIDIN Hesperidin is a bioflavonoid in citrus fruit which has a similar effect as other Iron binding compounds. It is somewhat like lactoferrin for fruit, and in our bodies has the added benefit of protecting internal organs from elevated iron toxicity. One study of Hesperidin treatment almost completely reversed the toxic effects of injected Iron in rats, using high doses over just ten days. The dose was 80mg/kg of body weight, which means a 150 lb adult would use 5.5 grams in a dose. Since hesperidin is often sold as a complex with chloride ions this is a very large amount of chloride to ingest at once, if using high doses of hesperidin hydrochloride it's a good idea to also supplement some bicarbonate (such as baking soda) to help the body offset the high amount of chloride or else neurological/breathing problems can result (it is a good practice to understand chemistry when using supplements, or to NOT use high doses of anything). Hesperidin would have the same effect at moderate doses over a longer period of time, but of course would not be much point in using it with Iron heavy foods since it would react to the iron ingested rather than chelating it from the body.

.....

Hesperidin attenuates iron-induced oxidative damage and dopamine depletion in Drosophila melanogaster model of Parkinson's disease
Conclusion
Taken together, the results of this study have revealed that acute Hsd treatment protects against Fe-induced neurotoxicity in flies, reducing mortality, inhibiting oxidative stress indexes and maintaining endogenous antioxidants, thus contributing to the restoration of cell viability. In addition, dietary supplementation of Hsd promotes a decrease in Fe concentration in the head and it has been associated with neuroprotection characterized by the improvement in AChE activity and restoration of dopamine levels and locomotor function in Drosophila melanogaster. Those observations highlight that Hsd may be a promising candidate for the treatment of sporadic PD induced by acute exposure to Fe, since Hsd exerts powerful chelating and antioxidant activity.

.......

Hesperidin Suppresses Renin-Angiotensin System Mediated NOX2 Over-Expression and Sympathoexcitation in 2K-1C Hypertensive Rats​

Abstract
Hesperidin, a flavonoid derived from citrus fruits, possesses several beneficial effects including anti-oxidation and anti-inflammation. The aim of this study was to investigate the effects of hesperidin on the renin-angiotensin system (RAS) cascade that mediated oxidative stress and sympathoexcitation in two-kidney, one-clipped (2K-1C) hypertensive rats. 2K-1C hypertension was induced in male Sprague-Dawley rats. Hypertensive rats were treated with hesperidin at 20[Formula: see text]mg/kg or 40[Formula: see text]mg/kg or losartan at 10[Formula: see text]mg/kg beginning at three weeks after surgery and then continued for four weeks ([Formula: see text]/group). Hesperidin reduced blood pressure in a dose-dependent manner in hypertensive rats compared to untreated rats ([Formula: see text]). Increased plasma angiotensin converting enzyme (ACE) activity and angiotensin II levels, as well as, upregulated AT1 receptor protein expression in aortic tissues were attenuated in hypertensive rats treated with hesperidin. Hesperidin suppressed oxidative stress markers and NADPH oxidase over-expression, and restored plasma nitric oxide metabolites in 2K-1C rats. This was associated with improvement of the vascular response to acetylcholine in isolated mesenteric vascular beds and aortic rings from 2K-1C rats treated with hesperidin ([Formula: see text]). Enhancement of nerve-mediated vasoconstriction related to high plasma noradrenaline in the 2K-1C group was alleviated by hesperidin treatment ([Formula: see text]). Furthermore, losartan exhibited antihypertensive effects by suppressing the RAS cascade and oxidative stress and improved vascular dysfunction observed in 2K-1C rats ([Formula: see text]). Based on these results, it can be presumed that hesperidin is an antihypertensive agent. Its antihypertensive action might be associated with reducing RAS cascade-induced NOX2 over-expression and sympathoexcitation in 2K-1C hypertensive rats.

 

Motif

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Joined
Nov 24, 2017
Messages
2,757
"The role of our endogenous DNA mobile elements in stress, aging, and inflammation, and their structural similarity to viruses, suggests that substances that are helpful for stress-induced inflammation could be helpful for viral infections, and vice versa. All of the effective treatments for Covid-19 have had antiinflammatory effects—angiotensin blockers, cinanserin (a serotonin blocker), antihistamines, naringenin, hesperidin, fisetin and other flavonoids (Clementi, et al., 2021; Bellavite and Donzelli, 2020; Mishra, et al., 2020; Jo, et al., 2020), progesterone (Jakovac, 2020), and calcium channel blockers (Solaimanzadeh, 2020; Jayaseelan VP and Paramasivam, 2020).
These substances reduce intracellular calcium and increase carbon dioxide in relation to oxygen and lactate, with the effect of reducing random and meaningless excitation and activation of tissues, making energy available for purposeful organized activities of the organism." Ray Peat


View: https://www.youtube.com/watch?v=a7flR7Xx4As


............

HESPERIDIN Hesperidin is a bioflavonoid in citrus fruit which has a similar effect as other Iron binding compounds. It is somewhat like lactoferrin for fruit, and in our bodies has the added benefit of protecting internal organs from elevated iron toxicity. One study of Hesperidin treatment almost completely reversed the toxic effects of injected Iron in rats, using high doses over just ten days. The dose was 80mg/kg of body weight, which means a 150 lb adult would use 5.5 grams in a dose. Since hesperidin is often sold as a complex with chloride ions this is a very large amount of chloride to ingest at once, if using high doses of hesperidin hydrochloride it's a good idea to also supplement some bicarbonate (such as baking soda) to help the body offset the high amount of chloride or else neurological/breathing problems can result (it is a good practice to understand chemistry when using supplements, or to NOT use high doses of anything). Hesperidin would have the same effect at moderate doses over a longer period of time, but of course would not be much point in using it with Iron heavy foods since it would react to the iron ingested rather than chelating it from the body.

.....

Hesperidin attenuates iron-induced oxidative damage and dopamine depletion in Drosophila melanogaster model of Parkinson's disease
Conclusion
Taken together, the results of this study have revealed that acute Hsd treatment protects against Fe-induced neurotoxicity in flies, reducing mortality, inhibiting oxidative stress indexes and maintaining endogenous antioxidants, thus contributing to the restoration of cell viability. In addition, dietary supplementation of Hsd promotes a decrease in Fe concentration in the head and it has been associated with neuroprotection characterized by the improvement in AChE activity and restoration of dopamine levels and locomotor function in Drosophila melanogaster. Those observations highlight that Hsd may be a promising candidate for the treatment of sporadic PD induced by acute exposure to Fe, since Hsd exerts powerful chelating and antioxidant activity.

.......

Hesperidin Suppresses Renin-Angiotensin System Mediated NOX2 Over-Expression and Sympathoexcitation in 2K-1C Hypertensive Rats​

Abstract
Hesperidin, a flavonoid derived from citrus fruits, possesses several beneficial effects including anti-oxidation and anti-inflammation. The aim of this study was to investigate the effects of hesperidin on the renin-angiotensin system (RAS) cascade that mediated oxidative stress and sympathoexcitation in two-kidney, one-clipped (2K-1C) hypertensive rats. 2K-1C hypertension was induced in male Sprague-Dawley rats. Hypertensive rats were treated with hesperidin at 20[Formula: see text]mg/kg or 40[Formula: see text]mg/kg or losartan at 10[Formula: see text]mg/kg beginning at three weeks after surgery and then continued for four weeks ([Formula: see text]/group). Hesperidin reduced blood pressure in a dose-dependent manner in hypertensive rats compared to untreated rats ([Formula: see text]). Increased plasma angiotensin converting enzyme (ACE) activity and angiotensin II levels, as well as, upregulated AT1 receptor protein expression in aortic tissues were attenuated in hypertensive rats treated with hesperidin. Hesperidin suppressed oxidative stress markers and NADPH oxidase over-expression, and restored plasma nitric oxide metabolites in 2K-1C rats. This was associated with improvement of the vascular response to acetylcholine in isolated mesenteric vascular beds and aortic rings from 2K-1C rats treated with hesperidin ([Formula: see text]). Enhancement of nerve-mediated vasoconstriction related to high plasma noradrenaline in the 2K-1C group was alleviated by hesperidin treatment ([Formula: see text]). Furthermore, losartan exhibited antihypertensive effects by suppressing the RAS cascade and oxidative stress and improved vascular dysfunction observed in 2K-1C rats ([Formula: see text]). Based on these results, it can be presumed that hesperidin is an antihypertensive agent. Its antihypertensive action might be associated with reducing RAS cascade-induced NOX2 over-expression and sympathoexcitation in 2K-1C hypertensive rats.


Great, thanks !

So if somebody catches covid - what would be a treatment that would be recommended with stuff that everybody can get , for example I can’t get ivermectin that easy.
 
OP
md_a

md_a

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Joined
Aug 31, 2015
Messages
468
Great, thanks !

So if somebody catches covid - what would be a treatment that would be recommended with stuff that everybody can get , for example I can’t get ivermectin that easy.
Methylene blue (1mg -15 mg per day) and related quinones such as (emodina from Cascara Sagrada and Aloe vera, vitamin K, ubiquinone, tetracycline, minocycline, azithromycin, erythromycin, Pau D'Arco - lapacho, etc.) have direct effects against many versions of coronavirus.
Methylene blue used in combination with light / sun treatment, is more efficient, but not needed, because methylene blue itself is quite potent virucidal.
Anti-serotonin / anti-endotoxin chemicals such as ciproheptadine-Peritol and Benadryl have anti-viral effects even against viruses considered lethal.
Famotidine - The known on-target activity of famotidine considered the known primary mechanism of action is as an antagonist of the histamine H2 receptor.
Methylene blue in large quantities can have harmful serotonergic effects and ciproheptadine which is a potent antiviral should control for any adverse effects from higher doses of methylene blue.
Niacinamide is also a powerful antiviral.
Pau D’arco tea is anti-viral, antimicrobial, antibacterial, anti-fungal, anti-inflammatory, analgesic, antioxidant, anti-aging and supports metabolism.
Pure Pau D’arco peel is known for its healing properties through naphthoquinone called Beta-lapachone.
Glycine / gelatin is antiviral.
Antiparasitic ivermectin destroys COVID-19 cells ???
Chronic viral infection is usually due to high cortisol and endotoxin-promoting estrogen. Estrogens in food / estrogenic toxins in the environment activate retroviruses. Increased adrenaline suppresses the immune system and both increase vulnerability to viral infection. Unsaturated fats (omega 3 and 6), carotenes stimulate estrogen through aromatase.
The "virus" does not kill, but the immune reaction is what causes a cycle of inflammation (cytokine storm), which can lead to the death of the infected person. If there is a cytokine storm, then beta-blockers: clonidine, propranolol give the body a better chance to fight.
Excess iron is involved in the cytokine storm.
L-Theanine, aspirin, niacinamide, salt, honey.
Sun / Vitamin D / light treatment, vitamin K, vitamin E, Thyroid, Natural progesterone, Pregnenolone, easily digestible foods: sufficient protein in combination with fruit, Maple syrup (Maple Syrup), avoidance of PUFA (omega 3 and 6 - sunflower oil for example), Iron avoidance, Iron chelation; honey, salt, coffee, aspirin, niacinamide, L-Theanine and glycine / gelatin, lactoderin, vitamin C, oregano oil, baking soda, magnesium oil, zinc, vitamin B1, B2, B6, medicinal charcoal (absorbs toxins) , chamomile tea.
Gargle with salt and lemon a very effective solution, aerosols with salt, aerosols with baking soda.
Riboflavin and endotoxin charcoal.
Copper prevents the spread of respiratory viruses.
Olive leaf extract have anti-viral properties.
Monolaurin has been found to be effective against viruses.
EMODINE: Anti-viral / anti-bacterial / Anti-parasitic / anti-fungal
Lidocaine has an antibacterial effect.
Lactoferrin has anti-fungal, anti-viral and anti-bacterial properties.
Policosanol is anti PUFA (polyunsaturated fat)
Hesperidin is a bioflavonoid in citrus fruit which has a similar effect as other Iron binding compounds. It is somewhat like lactoferrin for fruit, and in our bodies has the added benefit of protecting internal organs from elevated iron toxicity.
Hesperidin decrease AT1.
The virus activates the angiotensin receptor, so the receptor blockers are protective - losartan, telmisartan, etc. Progesterone is a natural inhibitor of that receptor.
The virus uses ACE2 (angiotensin 2 conversion enzyme) as a receptor and enters cells through the AT1 angiotensin receptor. Losartan, an angiotensin blocker, is an effective protection and has many other anti-inflammatory effects. Cinanserine a serotonin blocker and ciproheptadine could help. Progesterone lowers the angiotensin receptor, but does not act immediately as losartan does.
Vitamin B1 decreases the type 1 angiotensin 2 receptor or AT1 receptor through which the virus has access to the cell.
"The role of our endogenous DNA mobile elements in stress, aging, and inflammation, and their structural similarity to viruses, suggests that substances that are helpful for stress-induced inflammation could be helpful for viral infections, and vice versa. All of the effective treatments for Covid-19 have had antiinflammatory effects—angiotensin blockers, cinanserin (a serotonin blocker), antihistamines, naringenin, hesperidin, fisetin and other flavonoids (Clementi, et al., 2021; Bellavite and Donzelli, 2020; Mishra, et al., 2020; Jo, et al., 2020), progesterone (Jakovac, 2020), and calcium channel blockers (Solaimanzadeh, 2020; Jayaseelan VP and Paramasivam, 2020).
These substances reduce intracellular calcium and increase carbon dioxide in relation to oxygen and lactate, with the effect of reducing random and meaningless excitation and activation of tissues, making energy available for purposeful organized activities of the organism." Ray Peat
Ideal body temperature 37 C, pulse 75-90
 
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Peatness

Guest
Methylene blue (1mg -15 mg per day) and related quinones such as (emodina from Cascara Sagrada and Aloe vera, vitamin K, ubiquinone, tetracycline, minocycline, azithromycin, erythromycin, Pau D'Arco - lapacho, etc.) have direct effects against many versions of coronavirus.
Methylene blue used in combination with light / sun treatment, is more efficient, but not needed, because methylene blue itself is quite potent virucidal.
Anti-serotonin / anti-endotoxin chemicals such as ciproheptadine-Peritol and Benadryl have anti-viral effects even against viruses considered lethal.
Famotidine - The known on-target activity of famotidine considered the known primary mechanism of action is as an antagonist of the histamine H2 receptor.
Methylene blue in large quantities can have harmful serotonergic effects and ciproheptadine which is a potent antiviral should control for any adverse effects from higher doses of methylene blue.
Niacinamide is also a powerful antiviral.
Pau D’arco tea is anti-viral, antimicrobial, antibacterial, anti-fungal, anti-inflammatory, analgesic, antioxidant, anti-aging and supports metabolism.
Pure Pau D’arco peel is known for its healing properties through naphthoquinone called Beta-lapachone.
Glycine / gelatin is antiviral.
Antiparasitic ivermectin destroys COVID-19 cells ???
Chronic viral infection is usually due to high cortisol and endotoxin-promoting estrogen. Estrogens in food / estrogenic toxins in the environment activate retroviruses. Increased adrenaline suppresses the immune system and both increase vulnerability to viral infection. Unsaturated fats (omega 3 and 6), carotenes stimulate estrogen through aromatase.
The "virus" does not kill, but the immune reaction is what causes a cycle of inflammation (cytokine storm), which can lead to the death of the infected person. If there is a cytokine storm, then beta-blockers: clonidine, propranolol give the body a better chance to fight.
Excess iron is involved in the cytokine storm.
L-Theanine, aspirin, niacinamide, salt, honey.
Sun / Vitamin D / light treatment, vitamin K, vitamin E, Thyroid, Natural progesterone, Pregnenolone, easily digestible foods: sufficient protein in combination with fruit, Maple syrup (Maple Syrup), avoidance of PUFA (omega 3 and 6 - sunflower oil for example), Iron avoidance, Iron chelation; honey, salt, coffee, aspirin, niacinamide, L-Theanine and glycine / gelatin, lactoderin, vitamin C, oregano oil, baking soda, magnesium oil, zinc, vitamin B1, B2, B6, medicinal charcoal (absorbs toxins) , chamomile tea.
Gargle with salt and lemon a very effective solution, aerosols with salt, aerosols with baking soda.
Riboflavin and endotoxin charcoal.
Copper prevents the spread of respiratory viruses.
Olive leaf extract have anti-viral properties.
Monolaurin has been found to be effective against viruses.
EMODINE: Anti-viral / anti-bacterial / Anti-parasitic / anti-fungal
Lidocaine has an antibacterial effect.
Lactoferrin has anti-fungal, anti-viral and anti-bacterial properties.
Policosanol is anti PUFA (polyunsaturated fat)
Hesperidin is a bioflavonoid in citrus fruit which has a similar effect as other Iron binding compounds. It is somewhat like lactoferrin for fruit, and in our bodies has the added benefit of protecting internal organs from elevated iron toxicity.
Hesperidin decrease AT1.
The virus activates the angiotensin receptor, so the receptor blockers are protective - losartan, telmisartan, etc. Progesterone is a natural inhibitor of that receptor.
The virus uses ACE2 (angiotensin 2 conversion enzyme) as a receptor and enters cells through the AT1 angiotensin receptor. Losartan, an angiotensin blocker, is an effective protection and has many other anti-inflammatory effects. Cinanserine a serotonin blocker and ciproheptadine could help. Progesterone lowers the angiotensin receptor, but does not act immediately as losartan does.
Vitamin B1 decreases the type 1 angiotensin 2 receptor or AT1 receptor through which the virus has access to the cell.
"The role of our endogenous DNA mobile elements in stress, aging, and inflammation, and their structural similarity to viruses, suggests that substances that are helpful for stress-induced inflammation could be helpful for viral infections, and vice versa. All of the effective treatments for Covid-19 have had antiinflammatory effects—angiotensin blockers, cinanserin (a serotonin blocker), antihistamines, naringenin, hesperidin, fisetin and other flavonoids (Clementi, et al., 2021; Bellavite and Donzelli, 2020; Mishra, et al., 2020; Jo, et al., 2020), progesterone (Jakovac, 2020), and calcium channel blockers (Solaimanzadeh, 2020; Jayaseelan VP and Paramasivam, 2020).
These substances reduce intracellular calcium and increase carbon dioxide in relation to oxygen and lactate, with the effect of reducing random and meaningless excitation and activation of tissues, making energy available for purposeful organized activities of the organism." Ray Peat
Ideal body temperature 37 C, pulse 75-90
Thanks for this comprehesive list, it's good to have them all in one post

I would add lysine and selenium to the list



 

Motif

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Messages
2,757
Methylene blue (1mg -15 mg per day) and related quinones such as (emodina from Cascara Sagrada and Aloe vera, vitamin K, ubiquinone, tetracycline, minocycline, azithromycin, erythromycin, Pau D'Arco - lapacho, etc.) have direct effects against many versions of coronavirus.
Methylene blue used in combination with light / sun treatment, is more efficient, but not needed, because methylene blue itself is quite potent virucidal.
Anti-serotonin / anti-endotoxin chemicals such as ciproheptadine-Peritol and Benadryl have anti-viral effects even against viruses considered lethal.
Famotidine - The known on-target activity of famotidine considered the known primary mechanism of action is as an antagonist of the histamine H2 receptor.
Methylene blue in large quantities can have harmful serotonergic effects and ciproheptadine which is a potent antiviral should control for any adverse effects from higher doses of methylene blue.
Niacinamide is also a powerful antiviral.
Pau D’arco tea is anti-viral, antimicrobial, antibacterial, anti-fungal, anti-inflammatory, analgesic, antioxidant, anti-aging and supports metabolism.
Pure Pau D’arco peel is known for its healing properties through naphthoquinone called Beta-lapachone.
Glycine / gelatin is antiviral.
Antiparasitic ivermectin destroys COVID-19 cells ???
Chronic viral infection is usually due to high cortisol and endotoxin-promoting estrogen. Estrogens in food / estrogenic toxins in the environment activate retroviruses. Increased adrenaline suppresses the immune system and both increase vulnerability to viral infection. Unsaturated fats (omega 3 and 6), carotenes stimulate estrogen through aromatase.
The "virus" does not kill, but the immune reaction is what causes a cycle of inflammation (cytokine storm), which can lead to the death of the infected person. If there is a cytokine storm, then beta-blockers: clonidine, propranolol give the body a better chance to fight.
Excess iron is involved in the cytokine storm.
L-Theanine, aspirin, niacinamide, salt, honey.
Sun / Vitamin D / light treatment, vitamin K, vitamin E, Thyroid, Natural progesterone, Pregnenolone, easily digestible foods: sufficient protein in combination with fruit, Maple syrup (Maple Syrup), avoidance of PUFA (omega 3 and 6 - sunflower oil for example), Iron avoidance, Iron chelation; honey, salt, coffee, aspirin, niacinamide, L-Theanine and glycine / gelatin, lactoderin, vitamin C, oregano oil, baking soda, magnesium oil, zinc, vitamin B1, B2, B6, medicinal charcoal (absorbs toxins) , chamomile tea.
Gargle with salt and lemon a very effective solution, aerosols with salt, aerosols with baking soda.
Riboflavin and endotoxin charcoal.
Copper prevents the spread of respiratory viruses.
Olive leaf extract have anti-viral properties.
Monolaurin has been found to be effective against viruses.
EMODINE: Anti-viral / anti-bacterial / Anti-parasitic / anti-fungal
Lidocaine has an antibacterial effect.
Lactoferrin has anti-fungal, anti-viral and anti-bacterial properties.
Policosanol is anti PUFA (polyunsaturated fat)
Hesperidin is a bioflavonoid in citrus fruit which has a similar effect as other Iron binding compounds. It is somewhat like lactoferrin for fruit, and in our bodies has the added benefit of protecting internal organs from elevated iron toxicity.
Hesperidin decrease AT1.
The virus activates the angiotensin receptor, so the receptor blockers are protective - losartan, telmisartan, etc. Progesterone is a natural inhibitor of that receptor.
The virus uses ACE2 (angiotensin 2 conversion enzyme) as a receptor and enters cells through the AT1 angiotensin receptor. Losartan, an angiotensin blocker, is an effective protection and has many other anti-inflammatory effects. Cinanserine a serotonin blocker and ciproheptadine could help. Progesterone lowers the angiotensin receptor, but does not act immediately as losartan does.
Vitamin B1 decreases the type 1 angiotensin 2 receptor or AT1 receptor through which the virus has access to the cell.
"The role of our endogenous DNA mobile elements in stress, aging, and inflammation, and their structural similarity to viruses, suggests that substances that are helpful for stress-induced inflammation could be helpful for viral infections, and vice versa. All of the effective treatments for Covid-19 have had antiinflammatory effects—angiotensin blockers, cinanserin (a serotonin blocker), antihistamines, naringenin, hesperidin, fisetin and other flavonoids (Clementi, et al., 2021; Bellavite and Donzelli, 2020; Mishra, et al., 2020; Jo, et al., 2020), progesterone (Jakovac, 2020), and calcium channel blockers (Solaimanzadeh, 2020; Jayaseelan VP and Paramasivam, 2020).
These substances reduce intracellular calcium and increase carbon dioxide in relation to oxygen and lactate, with the effect of reducing random and meaningless excitation and activation of tissues, making energy available for purposeful organized activities of the organism." Ray Peat
Ideal body temperature 37 C, pulse 75-90

wow , thanks that was very detailed, but now I also feel overwhelmed.

i wonder if somebody with severe cases really had success with some of that stuff.

It’s a lot of stuff mentioned here.
if I catch it I could take copper , zinc, selenium, vitamin c (camu camu), coconut oil for some monolaurin, tonic water, salt , potassium, magnesium and some methylene blue (but I would take not much cause I never took it and I don’t know how I react ).

would that be a good enough treatment ?

i got niacin too, but with flush effect cause only this helps with histamine symptoms, but I only take it when my histamine really goes through the roof.
 

J.R.K

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Joined
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Messages
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Methylene Blue Inhibits the SARS-CoV-2 Spike–ACE2 Protein-Protein Interaction–a Mechanism that can Contribute to its Antiviral Activity Against COVID-19​


In conclusion, screening of our organic dye-based library identified MeBlu as a low-micromolar inhibitor of the interaction between SARS-CoV-2 spike protein and its cognate receptor ACE2, a PPI that is the first critical step initiating the viral entry of this coronavirus. While MeBlu shows strong polypharmacology and might be a somewhat promiscuous PPI inhibitor, its ability to inhibit this PPI could contribute to the antiviral activity of MeBlu against SARS-CoV-2 even in the absence of light making this inexpensive and widely available drug potentially useful in the prevention and treatment of COVID-19 as an oral or inhaled medication.

Excellent information again @ md_a! Would it be presumptuous to believe that Methylene Blue would prevent any transmission origin spike protein from taking hold in an unvaccinated person? Since it is not encapsulated in the lipid nanoparticle as the mRNA is, but rather presumably a result of the internal organisms generated production of the mRNA into the spike protein?
 

J.R.K

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Joined
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Messages
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"The role of our endogenous DNA mobile elements in stress, aging, and inflammation, and their structural similarity to viruses, suggests that substances that are helpful for stress-induced inflammation could be helpful for viral infections, and vice versa. All of the effective treatments for Covid-19 have had antiinflammatory effects—angiotensin blockers, cinanserin (a serotonin blocker), antihistamines, naringenin, hesperidin, fisetin and other flavonoids (Clementi, et al., 2021; Bellavite and Donzelli, 2020; Mishra, et al., 2020; Jo, et al., 2020), progesterone (Jakovac, 2020), and calcium channel blockers (Solaimanzadeh, 2020; Jayaseelan VP and Paramasivam, 2020).
These substances reduce intracellular calcium and increase carbon dioxide in relation to oxygen and lactate, with the effect of reducing random and meaningless excitation and activation of tissues, making energy available for purposeful organized activities of the organism." Ray Peat


View: https://www.youtube.com/watch?v=a7flR7Xx4As


............

HESPERIDIN Hesperidin is a bioflavonoid in citrus fruit which has a similar effect as other Iron binding compounds. It is somewhat like lactoferrin for fruit, and in our bodies has the added benefit of protecting internal organs from elevated iron toxicity. One study of Hesperidin treatment almost completely reversed the toxic effects of injected Iron in rats, using high doses over just ten days. The dose was 80mg/kg of body weight, which means a 150 lb adult would use 5.5 grams in a dose. Since hesperidin is often sold as a complex with chloride ions this is a very large amount of chloride to ingest at once, if using high doses of hesperidin hydrochloride it's a good idea to also supplement some bicarbonate (such as baking soda) to help the body offset the high amount of chloride or else neurological/breathing problems can result (it is a good practice to understand chemistry when using supplements, or to NOT use high doses of anything). Hesperidin would have the same effect at moderate doses over a longer period of time, but of course would not be much point in using it with Iron heavy foods since it would react to the iron ingested rather than chelating it from the body.

.....

Hesperidin attenuates iron-induced oxidative damage and dopamine depletion in Drosophila melanogaster model of Parkinson's disease
Conclusion
Taken together, the results of this study have revealed that acute Hsd treatment protects against Fe-induced neurotoxicity in flies, reducing mortality, inhibiting oxidative stress indexes and maintaining endogenous antioxidants, thus contributing to the restoration of cell viability. In addition, dietary supplementation of Hsd promotes a decrease in Fe concentration in the head and it has been associated with neuroprotection characterized by the improvement in AChE activity and restoration of dopamine levels and locomotor function in Drosophila melanogaster. Those observations highlight that Hsd may be a promising candidate for the treatment of sporadic PD induced by acute exposure to Fe, since Hsd exerts powerful chelating and antioxidant activity.

.......

Hesperidin Suppresses Renin-Angiotensin System Mediated NOX2 Over-Expression and Sympathoexcitation in 2K-1C Hypertensive Rats​

Abstract
Hesperidin, a flavonoid derived from citrus fruits, possesses several beneficial effects including anti-oxidation and anti-inflammation. The aim of this study was to investigate the effects of hesperidin on the renin-angiotensin system (RAS) cascade that mediated oxidative stress and sympathoexcitation in two-kidney, one-clipped (2K-1C) hypertensive rats. 2K-1C hypertension was induced in male Sprague-Dawley rats. Hypertensive rats were treated with hesperidin at 20[Formula: see text]mg/kg or 40[Formula: see text]mg/kg or losartan at 10[Formula: see text]mg/kg beginning at three weeks after surgery and then continued for four weeks ([Formula: see text]/group). Hesperidin reduced blood pressure in a dose-dependent manner in hypertensive rats compared to untreated rats ([Formula: see text]). Increased plasma angiotensin converting enzyme (ACE) activity and angiotensin II levels, as well as, upregulated AT1 receptor protein expression in aortic tissues were attenuated in hypertensive rats treated with hesperidin. Hesperidin suppressed oxidative stress markers and NADPH oxidase over-expression, and restored plasma nitric oxide metabolites in 2K-1C rats. This was associated with improvement of the vascular response to acetylcholine in isolated mesenteric vascular beds and aortic rings from 2K-1C rats treated with hesperidin ([Formula: see text]). Enhancement of nerve-mediated vasoconstriction related to high plasma noradrenaline in the 2K-1C group was alleviated by hesperidin treatment ([Formula: see text]). Furthermore, losartan exhibited antihypertensive effects by suppressing the RAS cascade and oxidative stress and improved vascular dysfunction observed in 2K-1C rats ([Formula: see text]). Based on these results, it can be presumed that hesperidin is an antihypertensive agent. Its antihypertensive action might be associated with reducing RAS cascade-induced NOX2 over-expression and sympathoexcitation in 2K-1C hypertensive rats.


Another piece of fantastic work md_a! If one were to not want to use a supplement but rather acquire these flavonoids through food, how much citrus would a person need to consume. I remember Georgy finding that study on naringinin, glycine, and apiginin, he said one glass of orange juice was able to replicate the dosage used in the study. I find personally many benefits of focusing on foods that contain all of these substances. As well I use about .75 mg per day of Methylene Blue everyday since this whole virus debacle began as it builds up over time.
 
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md_a

md_a

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Another piece of fantastic work md_a! If one were to not want to use a supplement but rather acquire these flavonoids through food, how much citrus would a person need to consume. I remember Georgy finding that study on naringinin, glycine, and apiginin, he said one glass of orange juice was able to replicate the dosage used in the study. I find personally many benefits of focusing on foods that contain all of these substances. As well I use about .75 mg per day of Methylene Blue everyday since this whole virus debacle began as it builds up over time.
I don't usually calculate the exact amount. I inform myself approximately, then depending on the symptoms I go to one dose or another. For example, 11 years ago when I was suffering from severe hypothyroidism with a daytime temperature of 35.8 C and pulse 50, I took cynoplus and cynomel a much higher amount than normal and I reached after 2 days the temperature of 37.1 C, pulse 85- 90, then I greatly reduced the dose for maintenance. I try every day to get a little of each which includes citrus marmalade, fruit, methylene blue, chamomile tea, cocoa milk and gelatin, aspirin .... and so far I'm fine, I have not suffered from a cold or anything something else.
 

J.R.K

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Joined
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Messages
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I don't usually calculate the exact amount. I inform myself approximately, then depending on the symptoms I go to one dose or another. For example, 11 years ago when I was suffering from severe hypothyroidism with a daytime temperature of 35.8 C and pulse 50, I took cynoplus and cynomel a much higher amount than normal and I reached after 2 days the temperature of 37.1 C, pulse 85- 90, then I greatly reduced the dose for maintenance. I try every day to get a little of each which includes citrus marmalade, fruit, methylene blue, chamomile tea, cocoa milk and gelatin, aspirin .... and so far I'm fine, I have not suffered from a cold or anything something else.
We seem to be of the same thought process on this topic @md_a! I am happy that you shared this with me. It is good to know that my experience is not unique and that others have experienced similar benefits!!
 

Lejeboca

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Just stumbled on a few results of the experiments in April-May of 2020 with Methylene Blue and SARS-COV2.

The report from a Russian medical University is in Russian Ученые Сеченовского университета выявили эффективность метиленового синего при лечении коронавируса
(I don't think it was ever published because of obvious reasons. The University and the experiment setup, etc seem very credible to me though.)

Here is a quick gist of the experiments and findings:
Experiment duration April 25 to May 25 2020, two groups of people: 49 patients with confirmed covid-19 and damaged lungs between 25-75% , 39 volunteers who were doctors and nurses in the "covid ward" of hospital.

Volunteers took the MB preparations each week by drinking an individually calculated dose. Patients w covid19 in addition to standard treatment received MB in the form of inhalations and orally in combination with light therapy (red light of 665nm waves shining on the nose, throat, and chest areas). Due to the light therapy the effect of MB increased 10 times.
Methylene blue used in combination with light / sun treatment, is more efficient, but not needed, because methylene blue itself is quite potent virucidal.


The effect of these procedures was remarkable: Already on the next day many patients saw the temperature drop from 39C to 36.6C. The loss of smell was completely restored, chest pains disappeared, the ability to breathe freely was returned.

After a single inhalation procedure, the covid test was giving a negative results.
After 10th and 12th day no virus was found in any of the participants. During the experiment, none of the participants experienced side effects.

@rothko @Cooper @ecstatichamster
 

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