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The Relation Between The Low T3 Syndrome In The Clinical Course Of Myocardial Infarction And Heart F

Discussion in 'Scientific Studies' started by Drareg, Sep 7, 2016.

  1. Drareg

    Drareg Member

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    Abstract
    It has been proven that either excess or deficiency of thyroid hormones has harmful influence on the cardiovascular system function. On the other hand, severe systemic conditions like myocardial infarction or severe heart failure may affect thyroid hormones secretion and their peripheral conversion, leading to low T3 syndrome. Amongst many mechanisms causing T4 to T3 conversion disturbances, important role plays decreased activity of D1 deiodinase and increased activity of D3 deiodinase. The animal research confirmed that thyroid hormones influence cardiomiocytes phenotype and morphology. They inhibit inflammation, apoptosis and cardiac remodelling after myocardial infarction. It was also proven that free triiodothyronine similarly to brain natriuretic peptide predict long-term prognosis in chronic and acute heart failure patients. Potential influence of low T3 syndrome on the course of myocardial infarction and heart failure may have significant impact on the future research on individualization of myocardial infarction and heart failure treatment depending on patient's thyroid status.
     
  2. ecstatichamster

    ecstatichamster Member

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    you are on fire today, thanks for the studies!
     
  3. OP
    Drareg

    Drareg Member

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    Your welcome, should be good to have any deiodinase orientated thyroid studies in one place.
    Will be interesting if we find a substance that can influence them,flavones for example.
     
  4. keith

    keith Member

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    I posted this study earlier High Free T4 May Increase Sudden Cardiac Death, but didn't get any responses. Think deficiencies in conversion of T4 to T3 would be responsible for possible heart disease being associated with high T4 levels?
     
  5. OP
    Drareg

    Drareg Member

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    Makes sense or T4 could be going straight to Rt3 depending on stress levels and current epigentics of patient.
     
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