THE GREAT mRNA VACCINATION EXPERIMENT (LONG-TERM EFFECTS OF MASS VACCINATION)

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Logan-

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Because the situation of complacency (not on this forum) is so desperate that I am not sure what, other than panic, can spur a change in attitude. True, panic does not help implement solutions, but in situations like this panic (IMO at least) is indispensable in forcing people to at least search for solutions. The first step is, as always, admitting we have a problem and were lied to, even if it is panic-inducing. Unfortunately, it looks like most people are all too comfortable in letting even this slide so other than panic, probably not much else will do. I am not pushing for panic as the only option per se, but I am not sure what other method is out there for shaking people out of their suicidal slumber. Let's see what happens with the FDA/Pfizer release. I think the first half of the 500K page release is supposed to happen in a month or so. If that "release" is so heavily redacted as to make it unusable, and especially if it is missing stats showing differences in health outcomes in vaxxed/unvaxxed then I don't think we need anything more definitive. I think the evidence even now is strong, but still circumstantial. FDA doing a handicap release or trying to appeal to the Supreme Court would be an even stronger indication the data is damning.
If there is no panic and no criminal prosecution of at least one major player in this fiasco within 1-2 years, the cabal wins. In just 5 years, any jump in deaths from the vaccination campaign can be explained away with various legalese and statistical manipulations, and virtually impossible to trace back to the vaccines. Especially, if data on vaxxed vs. unvaxxed health outcomes over time is no longer being collected or is classified and never released, as the CDC and most other "developed" countries' health agencies are saying they will do going forward.

IMHO cabal lost ten years ago.

Still, we cant panic here. This forum needs to keep a cool head, after all Peat said this would happen. Outside panic also does not help. Such explosions of energy are too short lived to do much, because this requires long term action to heal and to bring the perps to justice. Anger is justified, fear is not useful. Personally I don't invest too much into this. I did not take the vaccine, and NPCs have little capacity for independent thought. Millions will die and that is unfortunate. If you wish to fight in this war, you need to bring a feeling of safety and purpose to people. Those who are scared will not oppose authority , they manage their anxiety by siding with authority even if it kills them and their kids. They are so terrified of feelings. Look up what Mattias Desmet said about mass formation.

For myself, I am going to let the dead bury their dead.

 
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Vaccination and autoimmunity -'vaccinosis': a dangerous liaison?






 
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mRNA vaccines and autoimmunity:

The mRNA vaccines ultimately deliver the highly antigenic spike protein to antigen-presenting cells. As such, monoclonal antibodies against the spike protein are the expected outcome of the currently deployed mRNA vaccines. Human spike protein monoclonal antibodies were found to produce high levels of cross-reactive antibodies against endogenous human proteins (Vojdani et. al., 2021; reviewed in more detail below). Given evidence only partially reviewed here, there is sufficient reason to suspect that antibodies to the spike protein will contribute to ADE provoked by prior SARS-CoV-2 infection or vaccination, which may manifest as either acute or chronic autoimmune and inflammatory conditions. We have noted above that it is not possible to distinguish an ADE manifestation of disease from a true, non-ADE viral infection. In this light it is important to recognize that, when diseases and deaths occur shortly after vaccination with an mRNA vaccine, it can never be definitively determined, even with a full investigation, that the vaccine reaction was not a proximal cause.
International Journal of Vaccine Theory, Practice, and Research 2(1), May 10, 2021 Page | 401

Pathogenic Priming, Multisystem Inflammatory Disease, and Autoimmunity
Pathogenic priming is a concept that is similar in outcome to ADE, but different in the underlying mechanism. We discuss it here as a unique mechanism through which the mRNA vaccines could provoke associated pathologies.
In April 2020 an important paper was published regarding the potential for self-reactive antibodies to be generated following exposure to the spike protein and other antigenic epitopes spread over the length of SARS-CoV-2. Lyons-Weiler (2020) coined the phrase “pathogen priming” because he believed the more commonly used “immune enhancement” fails to capture the severity of the condition and its consequences. In his in silico analysis, Lyons-Weiler compared all antigenic SARS- CoV-2 protein epitopes flagged in the SVMTriP database (http://sysbio.unl.edu/SVMTriP/) and searched the p-BLAST database (BLAST: Basic Local Alignment Search Tool) for homology between those epitopes and endogenous human proteins. Of the 37 SARS-CoV-2 proteins analyzed, 29 had antigenic regions. All but one of these 29 had homology with human proteins (putative self- antigens) and were predicted to be autoreactogenic. The largest number of homologies were associated with the spike (S) protein and the NS3 protein, both having 6 homologous human proteins.
A functional analysis of the endogenous human proteins homologous with viral proteins found that over 1/3 of them are associated with the adaptive immune system. The author speculates that prior virus exposure or prior vaccination, either of which could initiate antibody production that targets these endogenous proteins, may be playing a role in the development of more severe disease in the elderly in particular. In this case the pre-existing antibodies act to suppress the adaptive immune system and lead to more severe disease.
Another group (Ehrenfeld et. al., 2020), in a paper predominantly about the wide range of autoimmune diseases found in association with a prior SARS-CoV-2 infection, also investigated how the spike protein could trigger such a range of diseases. They report, in Table 1 of that reference, strings of heptapeptides within the human proteome that overlap with the spike protein generated by SARS-CoV-2. They identified 26 heptapeptides found in humans and in the spike protein. It is interesting to note that 2 of the 26 overlapping heptapeptides were found to be sequential, a strikingly long string of identical peptides to be found in common between endogenous human proteins and the spike protein. Commenting on the overlapping peptides they had discovered and the potential for this to drive many types of autoimmunity simultaneously, they comment, “The clinical scenario that emerges is upsetting.” Indeed, it is.
In May of 2020 another important paper in this regard was published by Vojdani and Kharrazian (2020). The authors used both mouse and rabbit monoclonal antibodies against the 2003 SARS spike protein to test for reactivity against not only the spike protein of SARS-CoV-2, but also against several endogenous human proteins. They discovered that there was a high level of binding not only with the SARS-CoV-2 spike protein, but against a wide range of endogenous proteins. “[W]e found that the strongest reactions were with transglutaminase 3 (tTG3), transglutaminase 2 (tTG2), ENA, myelin basic protein (MBP), mitochondria, nuclear antigen (NA), α-myosin, thyroid peroxidase (TPO), collagen, claudin 5+6, and S100B.” (Vojdani and Kharrazian, 2020).
International Journal of Vaccine Theory, Practice, and Research 2(1), May 10, 2021 Page | 402

These important findings need to be emphasized. Antibodies with a high binding affinity to SARS- CoV-2 spike and other proteins also have a high binding affinity with tTG (associated with Celiac Disease), TPO (Hashimoto’s thyroiditis), myelin basic protein (multiple sclerosis), and several endogenous proteins. Unlike the autoimmune process associated with pathogen priming, these autoimmune diseases typically take years to manifest symptomatically.
The autoantibodies generated by the spike protein predicted by Lyons-Weiler (2020) and described above were confirmed with an in vitro study published more recently. In this follow-on paper, Vojdani et. al., (2021) looked again at the issue of cross-reactivity of antibodies, this time using human monoclonal antibodies (mAbs) against the SARS-CoV-2 spike protein rather than mouse and rabbit mAbs. Their results confirmed and extended their prior findings. “At a cutoff of 0.32 OD [optical density], SARS-CoV-2 membrane protein antibody reacted with 18 out of the 55 tested antigens.” These 18 endogenous antigens encompass reactivity to tissue in liver, mitochondria, the nervous and digestive system, the pancreas, and elsewhere in the body.
In a report on multisystem inflammatory syndrome in children (MIS-C), Carter et. al. (2020) studied 23 cases. Seventeen of 23 (68%) patients had serological evidence of prior SARS-CoV-2 infection. Of the three antibodies assessed in the patient population (nucleocapsid, RBD, and spike), IgG spike protein antibody optical density (which quantifies antibody concentrations against a standardized curve (Wikipedia, 2021)), was highest (see Figure 1d in Carter et al., 2020).
MIS-C is now commonly speculated to be an example of immune priming by prior exposure to SARS-CoV-2 or to other coronaviruses. Buonsenso et. al. (2020) reviewed multiple immunologic similarities between MIS-C and disease related to prior β-hemolytic Group A streptococcal infection (GAS). The authors write, “We can speculate that children's multiple exposition to SARS-CoV-2 with parents with COVID-19 can work as a priming of the immune system, as happens with GAS infection and, in genetically predisposed children, lead to [MIS-C] development. Another hypothesis is that previous infections with other coronaviruses, much more frequent in the pediatric population, may have primed the child immune system to SARS-CoV-2 virus.”
In June 2019 Galeotti and Bayry (2020) reviewed the occurrence of both autoimmune and inflammatory diseases in patients with COVID-19. They focus their analysis on MIS-C. After reviewing several previously published reports of a temporal link between COVID-19 and onset of MIS-C and describing a number of possible mechanistic connections between the two, the authors noted that no causal link had been established. In a somewhat prescient recommendation, they wrote, “A fine analysis of homology between various antigens of SARS-CoV-2 and self-antigens, by use of in silico approaches and validation in experimental models, should be considered in order to confirm this hypothesis.” It is precisely this type of in silico analysis carried out by Lyons-Weiler (2020) and by Ehrenfeld et. al. (2020) described in the opening paragraphs of this section which found the tight homology between viral antigens and self-antigens. While this may not definitively confirm the causal link hypothesized by Galeotti and Bayry, it is strong supporting evidence.
Autoimmunity is becoming much more widely recognized as a sequela of COVID-19. There are multiple reports of previously healthy individuals who developed diseases such as idiopathic thrombocytopenic purpura, Guillain-Barré syndrome and autoimmune haemolytic anaemia (Galeotti and Bayry, 2020). There are three independent case reports of systemic lupus erythemosus (SLE)
International Journal of Vaccine Theory, Practice, and Research 2(1), May 10, 2021 Page | 403

with cutaneous manifestations following symptomatic COVID-19. In one case a 39-year-old male had SLE onset two months following outpatient treatment for COVID-19 (Zamani et.al., 2021). Another striking case of rapidly progressing and fatal SLE with cutaneous manifestations is described by Slimani et.al. (2021).
Autoantibodies are very commonly found in COVID-19 patients, including antibodies found in blood (Vlachoyiannopoulos et. al., 2020) and cerebrospinal fluid (CFS) (Franke et. al., 2021). Though SARS-CoV-2 is not found in the CSF, it is theorized that the autoantibodies created in response to SARS-CoV-2 exposure may lead to at least some portion of the neurological complications documented in COVID-19 patients. One important Letter to the Editor submitted to the journal Arthritis & Rheumatology by Bertin et. al. (2020) noted the high prevalence and strong association (p=0.009) of autoantibodies against cardiolipin in COVID-19 patients with severe disease.
Zuo et. al. (2020) found anti-phospholipid autoantibodies in 52% of hospitalized COVID-19 patients and speculated that these antibodies contribute to the high incidence of coagulopathies in these patients. Schiaffino et. al. (2020) reported that serum from a high percentage of hospitalized COVID-19 patients contained autoantibodies reactive to the plasma membrane of hepatocytes and gastric cells. One patient with Guillain-Barre Syndrome was found to have antibody reactivity in cerebrospinal fluid (CFS), leading the authors to suggest that cross-reactivity with proteins in the CFS could lead to neurological complications seen in some COVID-19 patients. In a more recent review, Gao et. al. (2021) noted high levels of autoantibodies in COVID-19 patients across multiple studies. They conclude, “[O]ne of the potential side effects of giving a mass vaccine could be an mergence [sic] of autoimmune diseases especially in individuals who are genetically prone for autoimmunity.”
A recent publication compiles a great deal of evidence that autoantibodies against a broad range of receptors and tissue can be found in individuals who have had previous SARS-CoV-2 infection. “All 31 former COVID-19 patients had between 2 and 7 different GPCR-fAABs [G-protein coupled receptor functional autoantibodies] that acted as receptor agonists.” (Wallukat et. al. 2021) The diversity of GPCR-fAABs identified, encompassing both agonist and antagonist activity on target receptors, strongly correlated with a range of post-COVID-19 symptoms, including tachycardia, bradycardia, alopecia, attention deficit, PoTS, neuropathies, and others.
The same study, referencing the autoantibodies predicted by Lyons-Weiler (2020) mentioned above, notes with obvious grave concern: “The Sars-CoV-2 spike protein is a potential epitopic target for biomimicry-induced autoimmunological processes [25]. Therefore, we feel it will be extremely important to investigate whether GPCR-fAABs will also become detectable after immunisation by vaccination against the virus.”
We have reviewed the evidence here that the spike protein of SARS-CoV-2 has extensive sequence homology with multiple endogenous human proteins and could prime the immune system toward development of both auto-inflammatory and autoimmune disease. This is particularly concerning given that the protein has been redesigned with two extra proline residues to potentially impede its clearance from the circulation through membrane fusion. These diseases could present acutely and
International Journal of Vaccine Theory, Practice, and Research 2(1), May 10, 2021 Page | 404

over relatively short timespans such as with MIS-C or could potentially not manifest for months or years following exposure to the spike protein, whether via natural infection or via vaccination.
Many who test positive for COVID-19 express no symptoms. The number of asymptomatic, PCR- positive cases varies widely between studies, from a low of 1.6% to a high of 56.5% (Gao et. al., 2020). Those who are insensitive to COVID-19 probably have a very strong innate immune system. The healthy mucosal barrier's neutrophils and macrophages rapidly clear the viruses, often without the need for any antibodies to be produced by the adaptive system. However, the vaccine intentionally completely bypasses the mucosal immune system, both through its injection past the natural mucosal barriers and its artificial configuration as an RNA-containing nanoparticle. As noted in Carsetti (2020), those with a strong innate immune response almost universally experience either asymptomatic infection or only mild COVID-19 disease presentation. Nevertheless, they might face chronic autoimmune disease, as described previously, as a consequence of excessive antibody production in response to the vaccine, which was not necessary in the first place.

Worse Than the Disease? Reviewing Some Possible Unintended Consequences of the mRNA Vaccines Against COVID-19​

  • S. Seneff, G. Nigh
  • Published 16 June 2021
  • Biology
  • International Journal of Vaccine Theory, Practice, and Research

 
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Worse Than the Disease? Reviewing Some Possible Unintended Consequences of the mRNA Vaccines Against COVID-19​

  • S. Seneff, G. Nigh
  • Published 16 June 2021
  • Biology
  • International Journal of Vaccine Theory, Practice, and Research
https://pdfs.semanticscholar.org/2799/933cc09be1a78ad8c5a5dca5ede497045d58.pdf
I highly recommend this review article. The entire article is well worth reading.
 

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This is very important.

From the review article I posted above (published a year ago):
A Possible Link to Prion Diseases and Neurodegeneration
Prion diseases are a collection of neurodegenerative diseases that are induced through the misfolding of important bodily proteins, which form toxic oligomers that eventually precipitate out as fibrils causing widespread damage to neurons. Stanley Prusiner first coined the name `prion’ to describe these misfolded proteins (Prusiner, 1982). The best-known prion disease is MADCOW disease (bovine spongiform encephalopathy), which became an epidemic in European cattle beginning in the 1980s. The CDC web site on prion diseases states that “prion diseases are usually rapidly progressive and always fatal.” (Centers for Disease Control and Prevention, 2018). It is now believed that many neurodegenerative diseases, including Alzheimer’s, Parkinson’s disease, and
International Journal of Vaccine Theory, Practice, and Research 2(1), May 10, 2021 Page | 410

amyotrophic lateral sclerosis (ALS) may be prion diseases, and researchers have identified specific proteinaceous infectious particles linked to these diseases (Weickenmeier et al., 2019).
Furthermore, researchers have identified a signature motif linked to susceptibility to misfolding into toxic oligomers, called the glycine zipper motif. It is characterized by a pattern of two glycine residues spaced by three intervening amino acids, represented as GxxxG. The bovine prion linked to MADCOW has a spectacular sequence of ten GxxxGs in a row (see uniprot.org/uniprot/P10279).
More generally, the GxxxG motif is a common feature of transmembrane proteins, and the glycines play an essential role in cross-linking α-helices in the protein (Mueller et al., 2014). Prion proteins become toxic when the α-helices misfold as β-sheets, and the protein is then impaired in its ability to enter the membrane (Prusiner, 1982). Glycines within the glycine zipper transmembrane motifs in the amyloid-β precursor protein (APP) play a central role in the misfolding of amyloid- β linked to Alzheimer’s disease (Decock et al., 2016). APP contains a total of four GxxxG motifs.
When considering that the SARS-CoV-2 spike protein is a transmembrane protein, and that it contains five GxxxG motifs in its sequence (see uniprot.org/uniprot/P0DTC2), it becomes extremely plausible that it could behave as a prion. One of the GxxxG sequences is present within its membrane fusion domain. Recall that the mRNA vaccines are designed with an altered sequence that replaces two adjacent amino acids in the fusion domain with a pair of prolines. This is done intentionally in order to force the protein to remain in its open state and make it harder for it to fuse with the membrane. This seems to us like a dangerous step towards misfolding potentially leading to prion disease.
A paper published by J. Bart Classen (2021) proposed that the spike protein in the mRNA vaccines could cause prion-like diseases, in part through its ability to bind to many known proteins and induce their misfolding into potential prions. Idrees and Kumar (2021) have proposed that the spike protein’s S1 component is prone to act as a functional amyloid and form toxic aggregates. These authors wrote that S1 has the ability “to form amyloid and toxic aggregates that can act as seeds to aggregate many of the misfolded brain proteins and can ultimately lead to neurodegeneration.”
According to Tetz and Tetz (2020), the form of the spike protein in SARS-CoV-2 has prion regions that are not present in the spike proteins for other coronaviruses. While this was reported in a non- peer-reviewed article, the authors had published a previous paper in 2018 identifying prion-like regions in multiple eukaryotic viruses, so they have considerable expertise in this area (Tetz and Tetz, 2018).
A final point here relates to information about the Pfizer vaccine in particular. The European Medicines Agency (EMA) Public Assessment Report is a document submitted to gain approval to market the vaccine in Europe. It describes in detail a review of the manufacturing process as well as a wide range of associated testing data. One concerning revelation is the presence of “fragmented species” of RNA in the injection solution. These are RNA fragments resulting from early termination of the process of transcription from the DNA template. These fragments, if translated by the cell following injection, would generate incomplete spike proteins, again resulting in altered and unpredictable three-dimensional structure and a physiological impact that is at best neutral and at worst detrimental to cellular functioning. There were considerably more of these fragmented
International Journal of Vaccine Theory, Practice, and Research 2(1), May 10, 2021 Page | 411

forms of RNA found in the commercially manufactured products than in the products used in clinical trials. The latter were produced via a much more tightly controlled manufacturing process.
Pfizer claims the RNA fragments “likely... will not result in expressed proteins” due to their assumed rapid degradation within the cell. No data was presented to rule out protein expression, though, leaving the reviewers to comment, “These [fragmented RNA] forms are poorly characterised, and the limited data provided for protein expression do not fully address the uncertainties relating to the risk of translating proteins/peptides other than the intended spike protein” (EMA 2020). To our knowledge no data has been forthcoming since that time.
While we are not asserting that non-spike proteins generated from fragmented RNA would be misfolded or otherwise pathological, we believe they would at least contribute to the cellular stress that promotes prion-associated conformational changes in the spike protein that is present.
 
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“THE SPIKE PROTEIN ALONE
CAN CAUSE SEVERE DISEASE”
 

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Inaut

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I've known two people in the last 6 months to be diagnosed with aggressive glioblastomas... Both are triple vaxxed, maybe even quad at this point...

If anybody has seen The Terminal List on Prime?, you will remember that the soldiers were injected with a type of trauma blocking agent that eventually led to the troops developing brain tumors... I don't remember what it was called in the show but I'm going with CB-22(or 19) as the name of it for now. Either way, I don't find it ironic or coincidental in the slightest, just sad....Here's what i see happening in 3 simple steps

1. Create a "pandemic" to introduce a "vaccine" that causes cancer among other things(death from clotting etc.). Offer multiple vaccines to the fearful to increase chances of death.
2. Encourage frequent use bogus test swabs coated with who knows what to increase likelihood even more of specifically developing brain tumors (through the nasal cavity).
3. Diagnose these aggressive cancers and then treat(kill) patients with targeted(napalm) therapies(poisons). Tell them this is their only option for maybe one more year.

The end.
 

StrongMom

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Although there are excess deaths and so many adverse reactions, a large number of people are doing well. Almost everyone around me are vaccinated and doing well. I have been closely following them for signs of metabolic illnesses, I don’t see anything differen. My hypothesis is that young and/or metabolically healthy have eliminated the vaccine before accumulating or changing DNA. It is possible that they have metabolically aged but this will be almost impossible to differentiate from estrogen, radiation, 5g etc. like accelared aging.
 
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Although there are excess deaths and so many adverse reactions, a large number of people are doing well. Almost everyone around me are vaccinated and doing well. I have been closely following them for signs of metabolic illnesses, I don’t see anything differen. My hypothesis is that young and/or metabolically healthy have eliminated the vaccine before accumulating or changing DNA. It is possible that they have metabolically aged but this will be almost impossible to differentiate from estrogen, radiation, 5g etc. like accelared aging.
I’m baffled by this. I too have seen elderly triple jabbed who seem to be thriving.
 

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I've known two people in the last 6 months to be diagnosed with aggressive glioblastomas... Both are triple vaxxed, maybe even quad at this point...

If anybody has seen The Terminal List on Prime?, you will remember that the soldiers were injected with a type of trauma blocking agent that eventually led to the troops developing brain tumors... I don't remember what it was called in the show but I'm going with CB-22(or 19) as the name of it for now. Either way, I don't find it ironic or coincidental in the slightest, just sad....Here's what i see happening in 3 simple steps

1. Create a "pandemic" to introduce a "vaccine" that causes cancer among other things(death from clotting etc.). Offer multiple vaccines to the fearful to increase chances of death.
2. Encourage frequent use bogus test swabs coated with who knows what to increase likelihood even more of specifically developing brain tumors (through the nasal cavity).
3. Diagnose these aggressive cancers and then treat(kill) patients with targeted(napalm) therapies(poisons). Tell them this is their only option for maybe one more year.

The end.
Create a problem, offer the solution. The solution then causes other problems, which will not be associated with the first solution. Next offer solutions to the 2nd set of problems.
 
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🪖 The top question that I am asked at speaking events, and the topic that occupies most of my strategic thinking is: WHEN will there be accountability for what has happened with the jabs.

There’s a lot happening behind the scenes you don’t know about. I am working on a special post for Friday that describes exactly how we get from where we are now to criminal prosecutions. Stay tuned.

 
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After three years of closely following the plandemic I can confidently say this could not have been pulled off without an orchestrated effort by many agents. Even if you are a simple town doctor without much power would you not question the use of Mrna technology for a virus? Why would a doctor be willing to use remdesivr when it is known to cause kidney failure? Similar questions could be asked of government agencies about their willingness to implement rules that made little sense such as masks, especially for children, and social distancing. Even if they were fooled in the first year why are they still going along with it 3 years on? Then there is the denial that the injections are causing, or at least contributing, to excess deaths. How is it possible to deny the link between excess deaths and the injection when the data shows that during the mandates working age people experienced a massive die off?

Then there is the case of Campbell – I’ve looked into his background and I am willing to bet that he was positioned to lull many towards getting those injections. Cahill, Yeadon, Tenpenny, McCullough etc were already sounding the warning about the injections in 2020. Are we to believe Campbell did not know this? Even after blood clots were found following the AstraZeneca injection Campbell said they were safe. I take very little interest in what content he is putting out now. As far as I know you cannot undo the damage caused by the covid injections. Many who think they are fine now may not be in years to come. Some may never associate their poor health to that injection they took to save granny. Campbell is guilty and he knows it – I am not sure how long he can continue with that charade.

Out of respect I won’t call him what I am thinking but I will leave you with this reminder. Listen to him say Australia had no natural immunity – wt?


View: https://www.youtube.com/watch?v=qsq9C-p7rwU


Sorry I cannot watch this.


 

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Why quote me and edit what I said?
 
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Why quote me and edit what I said?
I only deleted the first sentence (“Of course we must consider all those possibilities and I have.”), as I deemed it unimportant for this topic. No other “editing” has been made.

I quoted your post as I think it poses good questions on the world we have been living in, and they are relevant to this particular thread.
 
EMF Mitigation - Flush Niacin - Big 5 Minerals

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