Thanks for the reply.
From my experience, I can tolerate rice. It doesn't trigger my gut problems as much as pastry does. I have already eliminated bread from my diet, I'm currently trying to handle my problems by abstaining from regular eating as much as possible. Raisins works, but roasted chickpeas giving me nausea. Since I started to cyproheptadine, I feel like I'm getting nausea more often, I can't eat chickpeas on an empty stomach anymore.
2 mg daily cypro is so far so good. At least it upped my gut's jittering threshold.
I don't know why but I get a point like pain in my bowel. On my left side. Whenever I eat something (especially pastry), I can feel and hear the sound and sensation of water flowing through my bowel.
This morning I tried one raw carrot, 1 teaspoon apple cider vinegar and 1 teaspoon olive oil. It gave me headache but somehow I felt more clear throughout the day. Also at the evening, I ate 1 clove of raw carlic with along my dinner. I'm doing these for sterilising my gut because it looks like I have 'a lot' gas problem (both upper and down side). So far, I can feel it had an effect.
I'm autistic/asperger. Autism is thought to be (?) caused from serotonin. Also:
Ectopic expression of Snord115 in choroid plexus interferes with editing but not splicing of 5-Ht2c receptor pre-mRNA in mice
Expression of serotonin 5-HT2C receptors in GABAergic cells of the anterior raphe nuclei. - PubMed - NCBI
5-ht2c has different types such as vgv, ini, vsv etc. Every one of them has different sensitivity to serotonin and basal activation levels. It's possible that fluoxetine had broke the balance of my receptors, thus allowing further neurological changes. 5ht2c has a big role because, I had a really crippling chronic anxiety disorder back in the time. All my anxiety is gone now. That's the very same receptor which causes anxiety side effect in the beggining of antidepressant medications.
Sorry for my really bad language. I had gotten very deteroirated recently. Now I don't have even an emotional attachment to my really best friend and my girlfriend. I wish I only have libido problems. Now I cannot think anything other than suicide, I'm really bothered of the blankness of life and my ***hole family.
From my experience, I can tolerate rice. It doesn't trigger my gut problems as much as pastry does. I have already eliminated bread from my diet, I'm currently trying to handle my problems by abstaining from regular eating as much as possible. Raisins works, but roasted chickpeas giving me nausea. Since I started to cyproheptadine, I feel like I'm getting nausea more often, I can't eat chickpeas on an empty stomach anymore.
2 mg daily cypro is so far so good. At least it upped my gut's jittering threshold.
I don't know why but I get a point like pain in my bowel. On my left side. Whenever I eat something (especially pastry), I can feel and hear the sound and sensation of water flowing through my bowel.
This morning I tried one raw carrot, 1 teaspoon apple cider vinegar and 1 teaspoon olive oil. It gave me headache but somehow I felt more clear throughout the day. Also at the evening, I ate 1 clove of raw carlic with along my dinner. I'm doing these for sterilising my gut because it looks like I have 'a lot' gas problem (both upper and down side). So far, I can feel it had an effect.
The 5-HT2c problem there's nothing I can say about. Even their explanations right now are quite convoluted and I cannot tell what would affect its RNA editing from that research, but it is probably part of a feedback or hormonal system.
I'm autistic/asperger. Autism is thought to be (?) caused from serotonin. Also:
Ectopic expression of Snord115 in choroid plexus interferes with editing but not splicing of 5-Ht2c receptor pre-mRNA in mice
Serotonin 5-HT2C receptor is a G-protein coupled excitatory receptor that regulates several biochemical pathways and has been implicated in obesity, mental state, sleep cycles, autism, neuropsychiatric disorders and neurodegenerative diseases. The activity of 5-HT2CR is regulated via alternative splicing and A to I editing of exon Vb of its pre-mRNA. Snord115 is a small nucleolar RNA that is expressed in mouse neurons and displays an 18-nucleotide base complementary to exon Vb of 5-HT2CR pre-mRNA. For almost two decades this putative guide element of Snord115 has wandered like a ghost through the literature in attempts to elucidate the biological significance of this complementarity. In mice, Snord115 is expressed in neurons and absent in the choroid plexus where, in contrast, 5-Ht2cr mRNA is highly abundant. Here we report the analysis of 5-Ht2cr pre-mRNA posttranscriptional processing via RNA deep sequencing in a mouse model that ectopically expresses Snord115 in the choroid plexus
Expression of serotonin 5-HT2C receptors in GABAergic cells of the anterior raphe nuclei. - PubMed - NCBI
5HT2C couples to a heterotrimeric G-protein containing Gq/11 that generates phospholipase C and IP3, which can be converted to IP6. It is thus possible that feedback between 5HT2C activity and 5HT2C editing occurs
5-ht2c has different types such as vgv, ini, vsv etc. Every one of them has different sensitivity to serotonin and basal activation levels. It's possible that fluoxetine had broke the balance of my receptors, thus allowing further neurological changes. 5ht2c has a big role because, I had a really crippling chronic anxiety disorder back in the time. All my anxiety is gone now. That's the very same receptor which causes anxiety side effect in the beggining of antidepressant medications.
Sorry for my really bad language. I had gotten very deteroirated recently. Now I don't have even an emotional attachment to my really best friend and my girlfriend. I wish I only have libido problems. Now I cannot think anything other than suicide, I'm really bothered of the blankness of life and my ***hole family.