youngsinatra
Member
Some say that optimal methylation function is essential to raise copper and ceruloplasmin.
I know someone who tripled his ceruloplasmin (15mg/dl -> 45mg/dl) by taking 10mg of B2 per day for 2 months.
I tried the same, but it did not raise mine, but at that time I did not take thyroid and symptom-wise and with my given basal body temperature and HR back then I was hypothyroid at that time.
B2 needs to get activated to become FAD/FMN, but this process is dependent on thyroid hormone.
Hypothyroid people have FAD levels similar to people with clinical riboflavin deficiency.
(Riboflavin metabolism in the hypothyroid human adult - PubMed)
FAD is a critical cofactor for the MTHFR enzyme function, which impacts the whole methylation cycle.
Dr. Gregory Russel-Jones (specialized his whole work on functional B2 deficiency) said:
I know someone who tripled his ceruloplasmin (15mg/dl -> 45mg/dl) by taking 10mg of B2 per day for 2 months.
I tried the same, but it did not raise mine, but at that time I did not take thyroid and symptom-wise and with my given basal body temperature and HR back then I was hypothyroid at that time.
B2 needs to get activated to become FAD/FMN, but this process is dependent on thyroid hormone.
Hypothyroid people have FAD levels similar to people with clinical riboflavin deficiency.
(Riboflavin metabolism in the hypothyroid human adult - PubMed)
FAD is a critical cofactor for the MTHFR enzyme function, which impacts the whole methylation cycle.
Dr. Gregory Russel-Jones (specialized his whole work on functional B2 deficiency) said: