RIP Jason Vale, Arm Wrestling World Champion And Laetrile Advocate, dies from stress caused by 2019 second raid

[burtlancast]

For those who don't know already Jason's story, he contracted incurable kidney cancer as a 17 year old teen, used conventional chemo and radiation, suffered a recurrence and decided then to only use Laetrile to control his cancer.

It worked well for dozens of years, even allowing him to become arm wrestling world champion. He decided to advertise his story and began selling raw apricot kernels himself: he got too big, got raided in 2003 and spent 5 years in jail.

Once out, he attempted to resume his Vit B17 business through other people, but alas they still raided him again in 2019..

We now learned he sadly died on July 2, 2021 at 53 years; no cause of death was announced, but we can surmise the stress caused by this new raid (his mother was also arrested) flared his kidney cancer.

Jason was already in bad shape and on crutches from a hip infection when the raid happened.

People deserve to know about Jason's work because it represents a first-hand, real account of raw cyanide-containing seeds being able to successfully prevent cancers in extremely high % of cases: it's a very affordable and simple way of controlling this disease especially for those not stricken with it.

https://videos.dailymail.co.uk/video/mol/2019/10/24/4645643577624803179/1024x576_MP4_4645643577624803179.mp4

View: https://www.youtube.com/watch?v=KYk7sgx2zLI



View: https://www.youtube.com/watch?v=y9lubMEix30



View: https://www.youtube.com/watch?v=lDaFLQP24-E




Jason and his mother, Barbara, managed to post bond (100.000 $ for both) and remain free while awaiting trial.

On another video, before the comments were locked, he said that there was some sort of lock or tracking device or something put on his leg, so he couldn't leave his house most of the day.

 

[Inaut]

@burtlancast I'm glad to see you posting more again :) Thanks for sharing

 

[achillea]

“2.6 million people die annually in low-and middle-income countries from medical errors, and that most of those deaths are related to misdiagnosis and administration of pharmaceutical products…Medication errors alone cost an estimated $42 billion (US dollars) annually. Unsafe surgical care procedures cause complications in up to 25% of patients resulting in 1 million deaths during or immediately after surgery annually…Four out of every ten patients are harmed during primary and ambulatory health care. The most detrimental errors are related to diagnosis, prescription and the use of medicines.” — The World Health Organization

But if you cure anyone with a seed you are put in jail..

IS THERE SOMETHING WRONG WITH THIS PICTURE ?????????????

 

[Dr. B]

For those who don't know already Jason's story, he contracted incurable cancer as a 17 year old teen, used conventional chemo and radiation, suffered a recurrence and decided then to only use Laetrile to control his cancer.

It worked well for dozens of years, even allowing him to become arm wrestling world champion. He decided to advertise his story and began selling raw apricot kernels himself: he got too big, got raided and spent 5 years in jail.

Once out, he attempted to resume his Vit B17 business through other people, but alas they still raided him again one month ago.

thats interesting, and it also makes sense laetrile could cure it? going off wikipedias article on it, laetrile/amygdalin seem like they are toxic and damaging to all cells. i think a while back there was a study or post of the same thing occurring with pau d arco bark/beta lapachone? it was confirmed to be cancer curing but the amounts needed to do so made it toxic to all cells. still, probably safer than the pharma drugs and chemo though?

 

[reaching]

Very interesting


View: https://youtu.be/hmkkwBTqVPc

 

[Dr. B]

Very interesting


View: https://youtu.be/hmkkwBTqVPc

what is? i cant hear it

 

[Inaut]

what is? i cant hear it

apple seeds ( for b17/cyanide compound)

 

[Dr. B]

apple seeds ( for b17/cyanide compound)

its not toxic/dangerous?
what does it do

 

[Ben.]

its not toxic/dangerous?
what does it do

Laetrile/Amygdalin (PDQ®)​

Health Professional Version
PDQ Integrative, Alternative, and Complementary Therapies Editorial Board.
Published online: March 18, 2021.

• Laetrile is another name for the natural product amygdalin, which is a chemical constituent found in the pits of many fruits and in numerous plants.
• Hydrogen cyanide is thought to be the main anticancer compound formed from laetrile via in situ release.
• Laetrile was first used as a cancer treatment in Russia in 1845, and in the United States in the 1920s.
• Laetrile has shown little anticancer activity in animal studies and no anticancer activity in human clinical trials.
• The side effects associated with laetrile toxicity mirror the symptoms of cyanide poisoning, including liver damage, difficulty walking (caused by damaged nerves), fever, coma, and death.
• Laetrile is not approved for use in the United States.
• Inappropriate advertisement of laetrile as a cancer treatment has resulted in a U.S. Food and Drug Administration investigation that culminated in charges and conviction of one distributor.

The term laetrile is derived from the terms laevorotatory and mandelonitrile and is used to describe a purified form of the chemical amygdalin, a cyanogenic glucoside (a plant compound that contains sugar and produces hydrogen cyanide) found in the pits of many fruits and raw nuts and in other plants, such as lima beans, clover, and sorghum.[1-6] In body fluids and at physiological pH, hydrogen cyanide dissolves to form the cyanide anion. The term vitamin B-17 was given to laetrile by E.T. Krebs Jr, but it is not an approved designation by the Committee on Nomenclature of the American Institute of Nutrition Vitamins. In the 1970s, laetrile gained popularity as an anticancer agent. By 1978, more than 70,000 individuals in the United States were reported to have been treated with it.[2,7,8]

Although the names laetrile, Laetrile, vitamin B-17, and amygdalin are often used interchangeably, they are not the same product. The chemical composition of U.S.-patented Laetrile (mandelonitrile-beta-glucuronide), a semisynthetic derivative of amygdalin, is different from the laetrile/amygdalin produced in Mexico (mandelonitrile beta-D-gentiobioside), which is made from crushed apricot pits.[15,16] Mandelonitrile, which contains a cyanide group, is a structural component of both products.[15] It has been proposed that released (hydrogen) cyanide is the active cancer-killing ingredient in laetrile, but two other breakdown products of amygdalin—prunasin (which is similar in structure to Laetrile) and benzaldehyde—may also be cancer cell inhibitors.[17-20] The studies discussed in this summary used either Mexican laetrile/amygdalin or the patented form. In most instances, the generic term laetrile will be used in this summary; however, a distinction will be made between the products when necessary.

Laetrile can be administered orally as a pill, or it can be given by injection (intravenous or intramuscular). It is commonly given intravenously for a period of time followed by oral maintenance therapy. The incidence of cyanide poisoning is much higher when laetrile is taken orally [21-23] because intestinal bacteria and some commonly eaten plants contain enzymes (beta-glucosidases) that activate the release of cyanide after laetrile has been ingested.[17,22] Relatively little breakdown occurs to yield the (hydrogen) cyanide when laetrile is injected.[7,22] Administration schedules and the length of treatment in animal models and humans vary widely.

 

[Dr. B]

Laetrile/Amygdalin (PDQ®)​

Health Professional Version
PDQ Integrative, Alternative, and Complementary Therapies Editorial Board.
Published online: March 18, 2021.

• Laetrile is another name for the natural product amygdalin, which is a chemical constituent found in the pits of many fruits and in numerous plants.
• Hydrogen cyanide is thought to be the main anticancer compound formed from laetrile via in situ release.
• Laetrile was first used as a cancer treatment in Russia in 1845, and in the United States in the 1920s.
• Laetrile has shown little anticancer activity in animal studies and no anticancer activity in human clinical trials.
• The side effects associated with laetrile toxicity mirror the symptoms of cyanide poisoning, including liver damage, difficulty walking (caused by damaged nerves), fever, coma, and death.
• Laetrile is not approved for use in the United States.
• Inappropriate advertisement of laetrile as a cancer treatment has resulted in a U.S. Food and Drug Administration investigation that culminated in charges and conviction of one distributor.

The term laetrile is derived from the terms laevorotatory and mandelonitrile and is used to describe a purified form of the chemical amygdalin, a cyanogenic glucoside (a plant compound that contains sugar and produces hydrogen cyanide) found in the pits of many fruits and raw nuts and in other plants, such as lima beans, clover, and sorghum.[1-6] In body fluids and at physiological pH, hydrogen cyanide dissolves to form the cyanide anion. The term vitamin B-17 was given to laetrile by E.T. Krebs Jr, but it is not an approved designation by the Committee on Nomenclature of the American Institute of Nutrition Vitamins. In the 1970s, laetrile gained popularity as an anticancer agent. By 1978, more than 70,000 individuals in the United States were reported to have been treated with it.[2,7,8]

Although the names laetrile, Laetrile, vitamin B-17, and amygdalin are often used interchangeably, they are not the same product. The chemical composition of U.S.-patented Laetrile (mandelonitrile-beta-glucuronide), a semisynthetic derivative of amygdalin, is different from the laetrile/amygdalin produced in Mexico (mandelonitrile beta-D-gentiobioside), which is made from crushed apricot pits.[15,16] Mandelonitrile, which contains a cyanide group, is a structural component of both products.[15] It has been proposed that released (hydrogen) cyanide is the active cancer-killing ingredient in laetrile, but two other breakdown products of amygdalin—prunasin (which is similar in structure to Laetrile) and benzaldehyde—may also be cancer cell inhibitors.[17-20] The studies discussed in this summary used either Mexican laetrile/amygdalin or the patented form. In most instances, the generic term laetrile will be used in this summary; however, a distinction will be made between the products when necessary.

Laetrile can be administered orally as a pill, or it can be given by injection (intravenous or intramuscular). It is commonly given intravenously for a period of time followed by oral maintenance therapy. The incidence of cyanide poisoning is much higher when laetrile is taken orally [21-23] because intestinal bacteria and some commonly eaten plants contain enzymes (beta-glucosidases) that activate the release of cyanide after laetrile has been ingested.[17,22] Relatively little breakdown occurs to yield the (hydrogen) cyanide when laetrile is injected.[7,22] Administration schedules and the length of treatment in animal models and humans vary widely.

do you think the side effect claims are true or are they exaggerated similar to other things
arent there quite a few things which are confirmed to be anti cancer but also toxic? like i think the wikipedia article on Pau d arco or one of the compounds in it, said they it was confirmed to have anti cancer activity but the doses needed were so high they were also toxic to other cells. isnt that how chemo itself works, its toxic to both cancer and healthy cells? theres probably many natural things which if you dose high enough, kill cancer but also healthy cells.

 

[Ben.]

... but the doses needed were so high they were also toxic to other cells. isnt that how chemo itself works, its toxic to both cancer and healthy cells? theres probably many natural things which if you dose high enough, kill cancer but also healthy cells.

Isn't that true for most things? I mean even the beloved Vitamin b3 is potentially harmfull to the liver.
In theory, water kills if you drink to much of it.

do you think the side effect claims are true or are they exaggerated similar to other things

I've no personal experience with cyanide poisining or vitamin b-17(amygdalin). I also haven't researched the topic to much.
But i think the same way alcohol or cigarettes have positive effects on the organism - despite their obvious and detrimental issues they are definiately causing - so do apple seeds probably.
Woudn't recommend people eating them however if not indicated for a certain health issue. Would love to hear more thoughts on this tho from someone more knowledgable or better yet, experienced.

 

[Dr. B]

Isn't that true for most things? I mean even the beloved Vitamin b3 is potentially harmfull to the liver.
In theory, water kills if you drink to much of it.



I've no personal experience with cyanide poisining or vitamin b-17(amygdalin). I also haven't researched the topic to much.
But i think the same way alcohol or cigarettes have positive effects on the organism - despite their obvious and detrimental issues they are definiately causing - so do apple seeds probably.
Woudn't recommend people eating them however if not indicated for a certain health issue. Would love to hear more thoughts on this tho from someone more knowledgable or better yet, experienced.

i heard its only niacin, but maybe the others too have effects
its an interesting nutrient has Ray talked about amygdalin

 

[reaching]

Isn't that true for most things? I mean even the beloved Vitamin b3 is potentially harmfull to the liver.
In theory, water kills if you drink to much of it.



I've no personal experience with cyanide poisining or vitamin b-17(amygdalin). I also haven't researched the topic to much.
But i think the same way alcohol or cigarettes have positive effects on the organism - despite their obvious and detrimental issues they are definiately causing - so do apple seeds probably.
Woudn't recommend people eating them however if not indicated for a certain health issue. Would love to hear more thoughts on this tho from someone more knowledgable or better yet, experienced.

Wait why is it? I love it...
granted i only take 50 100 grams of niacin at a time.

 

[Ben.]

Wait why is it? I love it...
granted i only take 50 100 grams of niacin at a time.

Guess you meant mg? otherwise youd prolly be dead.
I think dosages somewhere around 30-50mg maybe even 100-200 are fine and on the RPF the consensus is that Nicotinamide is safer altough liver damage has been accomplished with it aswell (and both biohackers and scientists do not agree among eachother which form is the best).
Vitamin b3 in time released form has shown the most damage. YOu should be fine tho. Also remember that niacin acts on certain pathways that nicotinamide does not. So it also depends on the individuals needs.

---

Again i am no expert but its not entire clear apparantly and there is some literature with case reports as to why:

LiverTox: Clinical and Research Information on Drug-Induced Liver Injury.​

Hepatotoxicity​

Niacin in doses above 500 mg daily causes transient, asymptomatic elevations in serum aminotransferase levels in up to 20% of people. The elevations are rarely greater than 3 times the upper limit of the normal range and usually resolve spontaneously even with continuation of the drug. The effect is partially dose related and is more common with doses above 3 g/day. In some patients, there is an overall decrease in serum proteins synthesized by the liver and, in some instances, coagulopathy with an increase in prothrombin time and decline in serum albumin, coagulation factors and apolipoproteins. These changes resolve rapidly upon stopping therapy and may not recur with lower doses.
Niacin can also cause serious hepatotoxicity, but this is uncommon. Significant hepatotoxicity is particularly common with high doses of sustained release niacin. In many cases, the injury becomes apparent after a dose increase or after switching from the regular crystalline to a sustained release form. The pattern is primarily hepatocellular, although cases with a cholestatic pattern have been described. The patients present with jaundice, itching, nausea, vomiting and fatigue. When the injury is the result of switching from the crystalline to the sustained release form, the injury may present acutely within days or a few weeks with a prodromal period of nausea, vomiting and abdominal pain, that is followed by jaundice and pruritus. Early during the injury serum aminotransferase levels are very high and then usually fall rapidly with discontinuation or dose lowering. The clinical phenotype resembles acute hepatic necrosis, suggesting a direct toxic effect. Imaging studies of the liver may reveal areas of hypodensity ("starry sky liver") interpreted as focal fatty infiltration that resolves after stopping the drug. Liver biopsy typically shows varying degrees of centrolobular necrosis with only mild inflammation.
Likelihood score: A[HD] (well known cause of clinically apparent liver injury when given in high doses).

Mechanism of Injury​

The mechanism of hepatotoxicity is assumed to be an intrinsic toxic reaction related to high serum levels of niacin that overwhelm the high affinity, low concentration nicotinic acid receptors (that are responsible for the flushing response). The finding that niacin can be restarted at lower doses after an episode of clinically apparent injury indicates that the hepatic damage is unlikely to be idiosyncratic or due to hypersensitivity.

​

Outcome and Management​

Niacin hepatotoxicity appears to be dose dependent and more common with the sustained release form of the drug. Hepatotoxicity is less common with regular, crystalline niacin or extended release niacin. Most cases are mild and resolve rapidly upon stopping the medication, although in some instances, the injury is acute and severe and progresses to liver failure that is fatal or requires emergency liver transplantation. Complete resolution of the clinical symptoms is expected within days of stopping niacin, whereas serum enzyme elevations may require several weeks or months to resolve. Rechallenge with the same form leads to rapid recurrence and should be avoided. If the injury occurred after switching to a SR formulation, the crystalline form of niacin may be restarted at a lower dose and with caution.

---

Take it with a grain of salt tho. There have been shizophrenics and other kind of illnesses that were treated with niacin on 1-5g dosages daily. Dont take time released form.
I think using something to protect the liver such as taurin, vitamin c and using some tmg can make sense if one plans to go with very high doses.
But this is getting offtopic ...

 

[reaching]

Guess you meant mg? otherwise youd prolly be dead.
I think dosages somewhere around 30-50mg maybe even 100-200 are fine and on the RPF the consensus is that Nicotinamide is safer altough liver damage has been accomplished with it aswell (and both biohackers and scientists do not agree among eachother which form is the best).
Vitamin b3 in time released form has shown the most damage. YOu should be fine tho. Also remember that niacin acts on certain pathways that nicotinamide does not. So it also depends on the individuals needs.

---

Again i am no expert but its not entire clear apparantly and there is some literature with case reports as to why:

LiverTox: Clinical and Research Information on Drug-Induced Liver Injury.​

Hepatotoxicity​

Niacin in doses above 500 mg daily causes transient, asymptomatic elevations in serum aminotransferase levels in up to 20% of people. The elevations are rarely greater than 3 times the upper limit of the normal range and usually resolve spontaneously even with continuation of the drug. The effect is partially dose related and is more common with doses above 3 g/day. In some patients, there is an overall decrease in serum proteins synthesized by the liver and, in some instances, coagulopathy with an increase in prothrombin time and decline in serum albumin, coagulation factors and apolipoproteins. These changes resolve rapidly upon stopping therapy and may not recur with lower doses.
Niacin can also cause serious hepatotoxicity, but this is uncommon. Significant hepatotoxicity is particularly common with high doses of sustained release niacin. In many cases, the injury becomes apparent after a dose increase or after switching from the regular crystalline to a sustained release form. The pattern is primarily hepatocellular, although cases with a cholestatic pattern have been described. The patients present with jaundice, itching, nausea, vomiting and fatigue. When the injury is the result of switching from the crystalline to the sustained release form, the injury may present acutely within days or a few weeks with a prodromal period of nausea, vomiting and abdominal pain, that is followed by jaundice and pruritus. Early during the injury serum aminotransferase levels are very high and then usually fall rapidly with discontinuation or dose lowering. The clinical phenotype resembles acute hepatic necrosis, suggesting a direct toxic effect. Imaging studies of the liver may reveal areas of hypodensity ("starry sky liver") interpreted as focal fatty infiltration that resolves after stopping the drug. Liver biopsy typically shows varying degrees of centrolobular necrosis with only mild inflammation.
Likelihood score: A[HD] (well known cause of clinically apparent liver injury when given in high doses).

Mechanism of Injury​

The mechanism of hepatotoxicity is assumed to be an intrinsic toxic reaction related to high serum levels of niacin that overwhelm the high affinity, low concentration nicotinic acid receptors (that are responsible for the flushing response). The finding that niacin can be restarted at lower doses after an episode of clinically apparent injury indicates that the hepatic damage is unlikely to be idiosyncratic or due to hypersensitivity.

​

Outcome and Management​

Niacin hepatotoxicity appears to be dose dependent and more common with the sustained release form of the drug. Hepatotoxicity is less common with regular, crystalline niacin or extended release niacin. Most cases are mild and resolve rapidly upon stopping the medication, although in some instances, the injury is acute and severe and progresses to liver failure that is fatal or requires emergency liver transplantation. Complete resolution of the clinical symptoms is expected within days of stopping niacin, whereas serum enzyme elevations may require several weeks or months to resolve. Rechallenge with the same form leads to rapid recurrence and should be avoided. If the injury occurred after switching to a SR formulation, the crystalline form of niacin may be restarted at a lower dose and with caution.

---

Take it with a grain of salt tho. There have been shizophrenics and other kind of illnesses that were treated with niacin on 1-5g dosages daily. Dont take time released form.
I think using something to protect the liver such as taurin, vitamin c and using some tmg can make sense if one plans to go with very high doses.
But this is getting offtopic ...

lolol yes. thank you for the study!

 

[burtlancast]

Sadly, Jason passed away this 2 august while awaiting trial.

I edited the headpost and the thread title to reflect that.

 

[Perry Staltic]

apple seeds ( for b17/cyanide compound)

I used to like to eat apple seeds, but after a while started feeling not quite right. I stopped eating them and the feeling went away. What was up with that?

 

[Birdie]

Sadly, Jason passed away this 2 august while awaiting trial.

I edited the headpost and the thread title to reflect that.

Really sorry to hear of this. And looking at the photos at the beginning of the videos was heart wrenching. A young man so full of hope and later looking beaten down.
At first I didn't understand the timing in your edit. That he had a lock on his leg is awful.

 

[burtlancast]

Here's a 20 min in depth interview with Jason by Ty Bollinger: it starts at 1h07 min in the vidéo;

Episode 11 - How to Survive & Thrive

Episode 11 - How to Survive & Thrive

conspyre.tv

And here's a short interview with Ian Jacklin


View: https://www.bitchute.com/video/wlwtBzqXtp8Y/