Kelj
Member
- Joined
- Jan 4, 2019
- Messages
- 299
This study:
Error - Cookies Turned Off
Finds:
"during a 12‐year follow‐up, 42% older adults affected by prediabetes remain stable, 22% reverted to normoglycaemia, whereas 13% progressed to diabetes or 23% died."
Also, the study, interestingly, has this to say:
"glycated haemoglobin A1c (HbA1c) values increase with age amongst diabetes‐free subjects, and low glucose level may increase mortality in old age 13, 14."
As with cholesterol levels, mortality is found @ lower levels. Why are we lowering it?
I believe this quote from the Eating Disorder Institute explains the effect this study was observing:
Diabetes Mellitus Type 2, Metformin, Disease Risk and You
"we need to reaffirm that the vagaries of blood glucose and insulin levels for those actively in recovery from an eating disorder can mimic the onset of diabetes mellitus type 2 but may not actually represent the onset of type 2 at all. Given that spontaneous adjustment of aberrant blood glucose and/or insulin levels back to the norm will occur for most as recovery from an eating disorder progresses, then we would be stretching diagnostic frameworks to refer to something transient and self-correcting as equivalent to diabetes mellitus type 2 in its usual presentation in old age......
diabetes mellitus type 2 is not a disease— it’s a risk factor for developing disease. That means that not everyone with diabetes type 2 (treated or untreated) will ever develop any disease state that is more strongly correlated with the presence of type 2 than for those who don’t have the condition."
If this study had asked its subjects about anything that had been happening to cause them to undereat, especially carbs, which was corrected over the 12 year span of the study, I believe they would have found that those who corrected the energy deficiency were the ones who reverted to normal glucose levels. I have seen this happen, just as the Eating Disorder Institute explains.
Error - Cookies Turned Off
Finds:
"during a 12‐year follow‐up, 42% older adults affected by prediabetes remain stable, 22% reverted to normoglycaemia, whereas 13% progressed to diabetes or 23% died."
Also, the study, interestingly, has this to say:
"glycated haemoglobin A1c (HbA1c) values increase with age amongst diabetes‐free subjects, and low glucose level may increase mortality in old age 13, 14."
As with cholesterol levels, mortality is found @ lower levels. Why are we lowering it?
I believe this quote from the Eating Disorder Institute explains the effect this study was observing:
Diabetes Mellitus Type 2, Metformin, Disease Risk and You
"we need to reaffirm that the vagaries of blood glucose and insulin levels for those actively in recovery from an eating disorder can mimic the onset of diabetes mellitus type 2 but may not actually represent the onset of type 2 at all. Given that spontaneous adjustment of aberrant blood glucose and/or insulin levels back to the norm will occur for most as recovery from an eating disorder progresses, then we would be stretching diagnostic frameworks to refer to something transient and self-correcting as equivalent to diabetes mellitus type 2 in its usual presentation in old age......
diabetes mellitus type 2 is not a disease— it’s a risk factor for developing disease. That means that not everyone with diabetes type 2 (treated or untreated) will ever develop any disease state that is more strongly correlated with the presence of type 2 than for those who don’t have the condition."
If this study had asked its subjects about anything that had been happening to cause them to undereat, especially carbs, which was corrected over the 12 year span of the study, I believe they would have found that those who corrected the energy deficiency were the ones who reverted to normal glucose levels. I have seen this happen, just as the Eating Disorder Institute explains.