Hey everybody,
Been reading the board on and off for a long while now and have always been impressed at how knowledgeable some are on here.
So I've just turned 40 and I am looking to finally enhance my hormones top prevent prostate cancer. My Dad has had PCa for over 20 years and its something I do not want to endeavour as well.
This is my hopefully prostate cancer prevention and life enhancer stack that I am looking to start soon.
1_ Daily testosterone to keep levels between 1000 and 1500. (Naturally I sit at about 650)
2_ Daily HCG to maintain fertility and neurosteroid cascade function. Important for progesterone and pregnenolone production of course.
3_ 12mg of Progest-E
4_ Aromatase inhibitor to keep E2 below 10 and certainly below 20. (Naturally I average low at about 15 anyway but E2 will obviously increase when Testosterone does)
I am on the fence with a 5aR inhibitor like low dose finasteride. There are many reports of post finasteride syndrome which seem attributed to disruption of pregnenolone and progesterone which should be covered with the HCG and endogenous progesterone anyway.
My three PSA’s have been
Age 38 = 2.1
Age 39 = 2.2
Age 40 = 2.4 (November just gone)
I had a 3T MRI scan of the prostate recently which shown everything normal and the prostate completely clear.
My only slight trepidation is the progesterone as I am under the impression it could feed PCa if there are microscopic cells already there?
I was told this by my friend Professor Edward Friedman who wrote a book on Prostate and Breast cancer pointing to localised oestrogen being the cause of both. He did say if there were no PCa cells present then progesterone would prevent PCa.
Even if there was microscopic cells already there and I was supplementing progesterone, would the AI and High T etc prevent the Pca ever growing?
Would love any opinions on this stack for prevention purposes.
Apologies in advance if I misrepresented something here, I know progesterone in particular is extremely favoured here so I would be interested in hearing the views on it potentially fuelling PCa.
Been reading the board on and off for a long while now and have always been impressed at how knowledgeable some are on here.
So I've just turned 40 and I am looking to finally enhance my hormones top prevent prostate cancer. My Dad has had PCa for over 20 years and its something I do not want to endeavour as well.
This is my hopefully prostate cancer prevention and life enhancer stack that I am looking to start soon.
1_ Daily testosterone to keep levels between 1000 and 1500. (Naturally I sit at about 650)
2_ Daily HCG to maintain fertility and neurosteroid cascade function. Important for progesterone and pregnenolone production of course.
3_ 12mg of Progest-E
4_ Aromatase inhibitor to keep E2 below 10 and certainly below 20. (Naturally I average low at about 15 anyway but E2 will obviously increase when Testosterone does)
I am on the fence with a 5aR inhibitor like low dose finasteride. There are many reports of post finasteride syndrome which seem attributed to disruption of pregnenolone and progesterone which should be covered with the HCG and endogenous progesterone anyway.
My three PSA’s have been
Age 38 = 2.1
Age 39 = 2.2
Age 40 = 2.4 (November just gone)
I had a 3T MRI scan of the prostate recently which shown everything normal and the prostate completely clear.
My only slight trepidation is the progesterone as I am under the impression it could feed PCa if there are microscopic cells already there?
I was told this by my friend Professor Edward Friedman who wrote a book on Prostate and Breast cancer pointing to localised oestrogen being the cause of both. He did say if there were no PCa cells present then progesterone would prevent PCa.
Even if there was microscopic cells already there and I was supplementing progesterone, would the AI and High T etc prevent the Pca ever growing?
Would love any opinions on this stack for prevention purposes.
Apologies in advance if I misrepresented something here, I know progesterone in particular is extremely favoured here so I would be interested in hearing the views on it potentially fuelling PCa.