Amazoniac
Member
Street violinists,
I realized one of these days that a lot of people with intestinal problems tend to avoid the exact foods that provide a lot of potassium due to their reactions to the carbohydrates in those foods. Mainly: milk, starch or fruits. Not only that but it usually starts with poor metabolism, and with that it's common to have deranged calcium, copper and iron metabolism; which tend to accumulate tissues and organs involved in digestion for reasons that I don’t know. The mineral balancing guys suggest that copper protects the liver for example.
Lack of enough magnesium exacerbates potassium deficiencies; which in turn, the deficiency by itself favors infections.
Potassium sorbate is quite toxic and widely used due to its effectiveness, the combination of sorbic acid and potassium; both seem to contribute to control microbial growth.
http://www.ncbi.nlm.nih.gov/pmc/articles/PMC1616818/pdf/annsurg01397-0207.pdf
“Potassium deficiency has been produced experimentally by feeding a low potassium diet or by injecting desoxycorticosterone. It was shown that potassium deficiency may lead to muscle weakness and, in dogs, to eventual paralysis. In addition to this skeletal muscle change, pathologic lesions in cardiac muscle occur which are associated with characteristic electrocardiographic changes.”
“In rats the tone and movements of the whole digestive tract decreased progressively as the deficiency continued (Fig. IB, 2B, 3A). In an average of two months, this resulted in severe distension which eventually was the cause of death. The hypotonic intestine was usually filled with large amounts of digestive-secretions, even before symptoms became severe. In many animals in the severe stage of deficiency, large amounts of gas accumulated, so that the condition resembled that of paralytic ileus. These signs rapidly disappeared upon the administration of potassium. The bladder of deficient animals was also atonic and was usually found filled with urine (Fig. iB).”
“In potassium deficient adult dogs, little change in motility of the digestive tract occurred, although there was a reduction in the strength and rate of rhythmic contractions. In these dogs, water exchange was increased early in the deficiency.”
“These studies have shown that simple potassium deficiency in rats greatly reduces motility in the smooth muscle organs of the gastro-intestinal tract and bladder. Because of the accumulation of fluid and gas, there is either an increased secretion of digestive juices and/or a decreased absorption. In potassium deficient dogs, the water exchange and volume of gastric secretions is increased.”
“So it would seem that when gastric and intestinal secretions are lost from the body, a relatively large amount of potassium is likewise lost, as these fluids contain two to three times as much potassium as an equivalent amount of serum. Two effects may ensue:
1. There may be inhibition of the intestinal musculature aggravating an ileus already established from some other cause.
2. There appears to be a stimulus to increase the volume of gastric secretion. The stomach continues to secrete potassium, and because of the increased volume of secretion, the potassium deficiency develops. Hydration and electrolyte balance can only be restored if potassium is administered.”
--
For now, questions should be addressed to jyb; which, by the way, threatened me to include this: coconut wasser, milk (+lactase if needed), potatoes, oranges, guavas, papayas, dates, grapes, melon (as someone from the forum suggested), beet greens, non-strained yogurt, some white fishes, clams, tomato sauce, broccoli, lentils, etc; coffee and chocolate are good sources but people with metabolic problems tend to be repulsed by copper-rich foods, I guess it’s prudent in those cases to respect cravings and reactions.
I realized one of these days that a lot of people with intestinal problems tend to avoid the exact foods that provide a lot of potassium due to their reactions to the carbohydrates in those foods. Mainly: milk, starch or fruits. Not only that but it usually starts with poor metabolism, and with that it's common to have deranged calcium, copper and iron metabolism; which tend to accumulate tissues and organs involved in digestion for reasons that I don’t know. The mineral balancing guys suggest that copper protects the liver for example.
Lack of enough magnesium exacerbates potassium deficiencies; which in turn, the deficiency by itself favors infections.
Potassium sorbate is quite toxic and widely used due to its effectiveness, the combination of sorbic acid and potassium; both seem to contribute to control microbial growth.
http://www.ncbi.nlm.nih.gov/pmc/articles/PMC1616818/pdf/annsurg01397-0207.pdf
“Potassium deficiency has been produced experimentally by feeding a low potassium diet or by injecting desoxycorticosterone. It was shown that potassium deficiency may lead to muscle weakness and, in dogs, to eventual paralysis. In addition to this skeletal muscle change, pathologic lesions in cardiac muscle occur which are associated with characteristic electrocardiographic changes.”
“In rats the tone and movements of the whole digestive tract decreased progressively as the deficiency continued (Fig. IB, 2B, 3A). In an average of two months, this resulted in severe distension which eventually was the cause of death. The hypotonic intestine was usually filled with large amounts of digestive-secretions, even before symptoms became severe. In many animals in the severe stage of deficiency, large amounts of gas accumulated, so that the condition resembled that of paralytic ileus. These signs rapidly disappeared upon the administration of potassium. The bladder of deficient animals was also atonic and was usually found filled with urine (Fig. iB).”
“In potassium deficient adult dogs, little change in motility of the digestive tract occurred, although there was a reduction in the strength and rate of rhythmic contractions. In these dogs, water exchange was increased early in the deficiency.”
“These studies have shown that simple potassium deficiency in rats greatly reduces motility in the smooth muscle organs of the gastro-intestinal tract and bladder. Because of the accumulation of fluid and gas, there is either an increased secretion of digestive juices and/or a decreased absorption. In potassium deficient dogs, the water exchange and volume of gastric secretions is increased.”
“So it would seem that when gastric and intestinal secretions are lost from the body, a relatively large amount of potassium is likewise lost, as these fluids contain two to three times as much potassium as an equivalent amount of serum. Two effects may ensue:
1. There may be inhibition of the intestinal musculature aggravating an ileus already established from some other cause.
2. There appears to be a stimulus to increase the volume of gastric secretion. The stomach continues to secrete potassium, and because of the increased volume of secretion, the potassium deficiency develops. Hydration and electrolyte balance can only be restored if potassium is administered.”
--
For now, questions should be addressed to jyb; which, by the way, threatened me to include this: coconut wasser, milk (+lactase if needed), potatoes, oranges, guavas, papayas, dates, grapes, melon (as someone from the forum suggested), beet greens, non-strained yogurt, some white fishes, clams, tomato sauce, broccoli, lentils, etc; coffee and chocolate are good sources but people with metabolic problems tend to be repulsed by copper-rich foods, I guess it’s prudent in those cases to respect cravings and reactions.
