Mega Dosing Iodine = Bad, Destroys Thyroid Tissue Permanently

[Blossom]

So we're all going to just pretend that Giraffe's reappearance is not surprising?

I guess so, I'm happy about it though.

 

[burtlancast]

The whole panacea mentality about iodine is another red flag.

You don't buy either Szent-Gyorgyi's regimen and empirical quote about potassium iodide?

And you don't think iodine holds the key to the bromine, fluoride, heavy metals plague burdening the whole organism and it's endocrine system (thyroid included), and affecting people worldwide?

Why do you reckon they insist on putting fluoride in the water while everybody with an ounce of common sense knows it's poison?

How much $$$$$$$ in treating symptoms are we talking about here?
I can't even put a price on that and the profits the pharma industry makes by selling gimmicks to it.

Just one example: how many sleeping pills have you ingested in your lifetime?

You keep bringing up the scientific points Ray makes about iodine, but it's clear he has a skewed view of the topic and even shows contemptible ignorance on many vital areas.

Which is extremely troubling considering he insists on being an authority on thyroid.
A thyroid specialist who refuses to do his homework on iodine?

What's going on here?

 

[TreasureVibe]

Maybe the effect that I had while on high doses of iodine were not thyroid storm but rather mercury becoming free'd from the tissues, and this is what I was feeling. I've had 4 amalgam fillings for around 14 years now. There's a good chance that mercury has built up in my system and that the iodine free'd it.

Who knows? I agree that the title of this topic is kind of scaring, but that source, a pharmacist, did say it was thyroid tissue being broken by the iodine. Here's some background information about her from her website https://thyroidpharmacist.com/about/ she also has done some interviews on YouTube and does discuss Dr. Abraham and Dr. Brownstein's works.

And Dr. Peat also is against high dose iodine supplementation. But, an admin if it is allowed, could change the title to something less scaring like "Source states mega-dosing iodine can destroy thyroid tissue, unclear if true". @Blossom

 

[Amazoniac]

What about the thyroid cancer/Hashimoto of Albert Szent-Giorgy, who ingested 1 gr/day of potassium iodide all his life, lol?

burtlan, where is that from? He might have taken that much when he noticed that something wasn't right, not on a daily basis.

Anyway, it's not just about iodin:
- ARL : Understanding Thyroid Activity (already approved by Diokin)
- Zinc, copper, manganese, and selenium metabolism in thyroid disease
- Estimation of Serum Copper, Manganese, Selenium, and Zinc in Hypothyroidism Patients
- The Impact of Iron and Selenium Deficiencies on Iodine and Thyroid Metabolism: Biochemistry and Raylevance to Public Health

 

[Amazoniac]

From what I tried to find, the sources state (couldn't find a reliable one yet) that it was 1000 mg, which should be a typo and they meant mcg instead. 1 mg is a high intake but reasonable. 1 gram a day would have melted his thyroid.
Potassium iodide - Wikipedia

Nutrients to Support Thyroid Function | Life Extension

 

[ilikecats]

@burtlancast yep ray “refuses to do his homework on iodine”. What a joke. What makes you think that? Just because he misattributed the iodine skin test to Guy Abraham? Even though lots of his followers mention it/ recommend it? Is it because ray didn’t spend enough time reading the work of geniuses Guy Abraham and David Brownstein? I guarantee you rays read study after study on Iodine but he failed to read the latest gibberish by some hack creationist like Guy Abraham so that means he “hasn’t done his homework on iodine.” I don’t take any “sleeping pills” either and as far as how much money I’ve spent treating “symptoms” I couldn’t say. I just spent a month taking zero supplements and I was fine. And the only things I take with any regularity are the fat soluble, b vitamins and minerals. Does iodine megadosing make you magically not require vitamins and other minerals? I don’t take thyroid hormone (David Brownstein does).
Here is some more stuff by Ray on iodine with REFRENCES. At least half of Guy Abraham’s refrences in his articles are to other articles he’s wrote himself :

“Iodine toxicity References
Re the urinary iodine test, it’s one of the big cults going around lately, telling people they should be saturated with iodine. Iodine reacts easily with toxic PUFA (omega-3 and -6 oils) to make antithyroid molecules.

Science 1985 Oct 18;230(4723):325-7
Induction of autoimmune thyroiditis in chickens by dietary iodine.
Bagchi N, Brown TR, Urdanivia E, Sundick RS.
Clinical studies have suggested that excess dietary iodine promotes autoimmune thyroiditis; however, the lack of a suitable animal model has hampered investigation of the phenomenon. In this study, different amounts of potassium iodide were added to the diets of chicken strains known to be genetically susceptible to autoimmune thyroiditis. Administration of iodine during the first 10 weeks of life increased the incidence of the disease, as determined by histology and the measurement of autoantibodies to triiodothyronine, thyroxine, and thyroglobulin. Further support for the relation between iodine and
autoimmune thyroiditis was provided by an experiment in which iodine-deficient regimens decreased the incidence of thyroid autoantibodies in a highly susceptible strain. These results suggest that excessive consumption of iodine in the United States may be responsible for the increased incidence of autoimmune thyroiditis.

Endocrinology. 2008 Jan;149(1):424-33. Epub 2007 Sep 20.
Oxidative stress in the thyroid gland: from harmlessness to hazard depending on the iodine content.
Poncin S, Gérard AC, Boucquey M, Senou M, Calderon PB, Knoops B, Lengelé B, Many
MC, Colin IM.
Unité de Morphologie Expérimentale (MOEX), Université catholique de Louvain,
UCL-5251, B-1200 Brussels, Belgium.
In basal conditions, thyroid epithelial cells produce moderate amounts of reactive oxygen species (ROS) that are physiologically required for thyroid hormone synthesis. They are not necessarily toxic because they are continuously detoxified either in the process of hormone synthesis or by endogenous antioxidant systems. Using a rat model of goiter formation and iodine-induced involution, we found that compared with control thyroids, the oxidative stress, assessed by the detection of 4-hydroxynonenal, was strongly enhanced both in hyperplastic and involuting glands. The level of antioxidant defenses (glutathione peroxidases and peroxiredoxins) was also up-regulated in both groups, although somewhat less in the latter. Of note, increased oxidative stress came along with an inflammatory reaction, but only in involuting glands, suggesting that although antioxidant systems can adequately buffer a heavy load of ROS in goiter, it is not necessarily the case in involuting glands. The effects of 15-deoxy-Delta(12,14)-prostaglandin J2 (15dPGJ2), an endogenous ligand of peroxisome proliferated-activated receptor gamma (PPARgamma) with antiinflammatory properties, were then investigated in involuting glands. This drug strongly reduced both 4-hydroxynonenal staining and the inflammatory reaction, indicating that it can block iodine-induced cytotoxicity. When experiments were carried out with the PPARgamma antagonist, bisphenol A diglycidyl ether, 15dPGJ2-induced effects remained unchanged, suggesting that these effects were not mediated by PPARgamma. In conclusion, thyroid epithelial cells are well adapted to endogenously produced ROS in basal and goitrous conditions. In iodine-induced goiter involution, the increased oxidative stress is accompanied by inflammation that can be blocked by 15dPGJ2 through PPARgamma-independent protective effects.

Hokkaido Igaku Zasshi 1994 May;69(3):614-26
[Screening for thyroid dysfunction in adults residing in Hokkaido Japan: in
relation to urinary iodide concentration and thyroid autoantibodies]
[Article in Japanese]
Konno N, Iizuka N, Kawasaki K, Taguchi H, Miura K, Taguchi S, Murakami S, Hagiwara K, Noda Y, Ukawa S.
Department of Internal Medicine, Hokkaido Central Hospital for Social Health Insurance, Sapporo, Japan.
The prevalence of thyroid dysfunction and its relation to thyroid autoantibodies (TAA) and urinary iodide concentration (UI) was studied in apparently healthy adults in Sapporo (n = 4110) (Sapporo group), and in five coastal areas of Hokkaido (n = 1061) (coastal group) which produce iodine-rich seaweed (kelp). The frequency of above normal urinary iodine (high UI) in the morning urinary samples of coastal group was 10.8%, significantly higher than that of Sapporo group (6.4%) (p < 0.001). Frequency of positive TAA in both groups were similar. In Sapporo group TAA was positive in 6.4% of males and 13.8% of females with an age-related increase. The overall prevalence of hyperthyroidism (TSH < 0.15 mU/L) in coastal group (0.6%) was similar to that in Sapporo group (1.1%), while that of hypothyroidism (TSH > 5.0 mU/L) in coastal group (3.8%) was significantly higher than that in Sapporo group (1.3%) (P < 0.001). The frequency of high UI correlated significantly with that of hypothyroidism with negative TAA (r = 0.829, P < 0.05), but not with positive TAA, or with that of hyperthyroidism. Hypothyroidism was more prevalent in TAA negative subjects with high UI than with normal UI. Moreover, serum TSH and thyroglobulin levels were higher and free T4 level was lower in former than in latter group. These results indicate that 1) the prevalence of TAA negative hypothyroidism in iodine sufficient areas may be associated with the amount of iodine ingested, 2) this hypothyroidism is more prevalent and marked in subjects consuming further excess amounts of iodine, and 3) excessive intake of iodine should be considered an etiology of hypothyroidism in addition to chronic thyroiditis in these areas.

Clin Endocrinol (Oxf) 1989 Oct;31(4):453-65
Thyroid autoimmunity in endemic goitre caused by excessive iodine intake.
Boyages SC, Bloot AM, Maberly GF, Eastman CJ, Li M, Qian QD, Liu DR, van der Gaag RD, Drexhage HA.
Department of Medicine, Westmead Hospital, Sydney, Australia.
The pathophysiology of endemic goitre caused by excessive iodine intake is not well defined. By interacting with the immune system, iodine excess may trigger the development of autoimmune thyroid disease such as lymphocytic Hashimoto's thyroiditis (LT). In an attempt to examine this further, we compared the presence of thyroid autoantibodies in 29 goitrous children, from an iodine excess area, and in 26 healthy children, from an iodine sufficient area, of north central China. Serum was tested for antimicrosomal (MAb), anti-thyroglobulin (TgAb), second colloid antigen antibodies (CA2-Ab) and TSH
binding inhibitory immunoglobulins (TBII). Affinity chromatographically purified IgG was tested for thyroid growth-stimulating activity (TGI) by two different methods: a sensitive cytochemical bioassay (CBA) using guinea-pig thyroid explants and a mitotic arrest assay (MAA) employing a continuous rat thyroid cell line (FRTL-5). We found no increased prevalence of LT in patients with endemic iodine goitre. The levels of MAb, TgAb and CA2-Ab did not differ significantly between the two groups of children. Further, TBII were not present in either group. Thyroid growth-stimulating immunoglobulins (TGI) were the major autoantibodies found in children with goitres caused by iodine excess. In the CBA, 12 of 20 (60%) goitrous children and 0 of 12 (0% P less than 0.05) healthy children were positive for TGI. Similar results were found in the MAA, and a good correlation between results of the CBA and MAA was found (P = 0.003). Maximal TGI activity in dose-response CBA showed a good relation with clinical goitre size (r = 0.63; P less than 0.05) indicating a possible pathophysiological role for these antibodies. We conclude that endemic iodine goitre is not associated with Hashimoto's lymphocytic thyroiditis. Nevertheless,
autoimmune growth factors such as TGI may play a primary role in the pathogenesis of thyroid growth in this condition.

Endocrinol Metab Clin North Am 1987 Jun;16(2):327-42
Environmental factors affecting autoimmune thyroid disease.
Safran M, Paul TL, Roti E, Braverman LE.
Department of Medicine, University of Massachusetts Medical Center, Worcester.
A number of environmental factors affect the incidence and progression of autoimmune thyroid disease. Exposure to excess iodine, certain drugs, infectious agents and pollutants, and stress have all been implicated.

Acta Endocrinol (Copenh) 1978 Aug;88(4):703-12
A case of Hashimoto's thyroiditis with thyroid immunological abnormality manifested after habitual ingestion of seaweed.
Okamura K, Inoue K, Omae T.
An interesting case of iodide induced goitre with immunological abnormalities is described. The patient who was sensitive to synthetic penicillin had previously been treated for exudative pleuritis, congestive heart failure and acute renal failure. Following recovery, he began to ingest large amounts of seaweed after which he developed goitrous hypothyroidism. It was of interest that the serum level of gamma-globulin increased, and subsequently the antithyroid microsomal antibody became strongly positive, suggesting that thyroidal autoimmune processes had been precipitated. Biopsy of the thyroid gland revealed chronic thyroiditis, with evidence suggesting extreme stimulation by TSH. Hight thyroidal uptake of 131I, positive perchlorate discharge test and biochemical analysis of the thyroidal soluble protein showed severe impairment of hormone synthesis following continuous accumulation of excess iodide. While there is evidence suggesting that increased iodide may be an important factor in the initiation of Hashimoto's thyroiditis, this may result from the marked increased sensitivity of Hashimoto's gland to the effects of iodine. Thus an occult lesion could be unmasked in this manner. The mechanism by which iodide mediates this effect is not clear.

Presse Med 2002 Oct 26;31(35):1670-5
[Hypothyroidism related to excess iodine]
[Article in French]
Wemeau JL.
Clinique endocrinologique Marc Linquette CHRU de Lille USNA, 59037 Lille. [email protected]
WOLFF-CHAIKOFF'S EFFECT: The thyroid gland has a capacity to reduce thyroid hormone production in the presence of excess iodine by reducing the organification of the iodine. This Wolff-Chaikoff effect is observed after 48 hours and protects the organism from excessive synthesis of the thyroid hormones. This effect is usually temporary and within a few days thyroid hormone synthesis returns to normal through the so-called 'escape' phenomenon. However in a few normal individuals and in some susceptible patients, the escape does not occur. THE CONTEXT OF OCCURRENCE: Iodine-induced hypothyroidism is observed in fetuses, newborn, adults and in the elderly. It is observed in individuals without underlying overt thyroid disorder, and especially in patients with autoimmune thyroiditis or those previously treated for thyroid diseases (Graves' disease, subacute or pospartum thyroiditis, iatrogenic thyroid dysfunction...). FROM A CLINICAL AND PROGRESSIVE POINT OF VIEW: The hormone deficiency is of obvious clinical expression, or sometimes discreet and revealed by hormone exploration. It is usually temporary, regressing with a few days or weeks after iodine withdrawal. Nevertheless, some patients require transient hormone replacement therapy.

Thyroid 2001 May;11(5):427-36
Iodine and thyroid autoimmune disease in animal models.
Ruwhof C, Drexhage HA.
Department of Immunology, Erasmus University, Rotterdam.
Thyroid autoimmune diseases are complex, polygenic afflictions the penetrance of which is heavily dependent on various environmental influences. In their pathogenesis, an afferent stage (enhanced autoantigen presentation), a central stage (excessive expansion and maturation of autoreactive T and B cells), and an efferent stage (effects of autoreactive T cells and B cells on their targets) can be discerned. At each stage, a plethora of inborn, endogenous or exogenous factors is able to elicit the abnormalities characteristic of that stage, thus opening the gateway to thyroid autoimmunity. Iodine is an important exogenous modulating factor of the process. In general, iodine deficiency attenuates, while iodine excess accelerates autoimmune thyroiditis in autoimmune prone individuals. In nonautoimmune prone individuals, the effects of iodine are different. Here iodine deficiency precipitates a mild (physiological) form of thyroid autoimmune reactivity. Iodine excess stimulates thymus development. Iodine probably exerts these effects via interference in the various stages of the autoimmune process. In the afferent and efferent stage, iodine-induced alterations in thyrocyte metabolism and even necrosis most likely play a role.
By contrast, in the central phase, iodine has direct effects on thymus development, the development and function of various immune cells (T cells, B cells macrophages and dendritic cells) and the antigenicity of thyroglobulin.

Clin Immunol Immunopathol 1996 Dec;81(3):287-92
Iodine-induced autoimmune thyroiditis in NOD-H-2h4 mice.
Rasooly L, Burek CL, Rose NR.
Department of Molecular Microbiology and Immunology, School of Hygiene and Public Health, Johns Hopkins University, Baltimore, Maryland 21205, USA.
Excess iodine ingestion has been implicated in induction and exacerbation of autoimmune thyroiditis in human populations and animal models. We studied the time course and sex-related differences in iodine-induced autoimmune thyroiditis in NOD-H-2h4 mice. This strain, derived from a cross of NOD with B10.A(4R), spontaneously develops autoimmune thyroiditis but not diabetes. NOD-H-2h4 mice were given either plain water or water with 0.05% iodine for 8 weeks. Approximately 54% of female and 70% of male iodine-treated mice developed thyroid lesions, whereas only 1 of 20 control animals had thyroiditis at this
time. Levels of serum thyroxin (T4) were similar in the treatment and control groups. Thyroglobulin-specific antibodies were present in the iodine-treated group after 8 weeks of treatment but antibodies to thyroid peroxidase were not apparent in the serum of any of the animals. Levels of thyroglobulin antibodies
increased throughout the 8-week iodine ingestion period; however, no correlation was seen between the levels of total thyroglobulin antibodies and the degree of thyroid infiltration at the time of autopsy. The thyroglobulin antibodies consisted primarily of IgG2a, IgG2b, and IgM antibodies with no detectable IgA,
IgG1, or IgG3 thyroglobulin-specific antibodies. The presence of IgG2b thyroglobulin-specific antibodies correlated well with the presence of thyroid lesions.

Verh Dtsch Ges Pathol 1996;80:297-301
[Spontaneous Hashimoto-like thyroiditis in cats]
[Article in German]
Schumm-Draeger PM, Langer F, Caspar G, Rippegather K, Herrmann G, Fortmeyer HP, Usadel KH, Hubner K.
Medizinische Klinik I, Johann Wolfgang Goethe-Universitat, Frankfurt/M.
A breeding line of domestic cats spontaneously developing symptoms of hypothyroidism between the 40th and 60th day of life (fur changes, loss of appetite, growth retardation), elevated levels of antibodies against microsomal structures and thyroglobulin, and lymphocytic thyroid infiltration has been recently established at our facility. Aim of our studies was to examine the effect of high iodine ingestion or prophylactic thyroid hormone therapy on functional and morphological characteristics of this Hashimoto-like thyroiditis in cat. From birth to day 80 of life cats were treated with iodine (n = 9; 0.1 mg/l) or thyroxin (n = 13; 2.0 micrograms/ kg/d) respectively. Untreated animals served as controls (n = 12). Cat-serum was tested for thyroid function (TT3, TT4). After 8 weeks the thyroid tissue was submitted to routine histological processing (H&E) and the inflammatory activity was scored. Additionally immunohistological staining was performed for MIB-1, IgG, IgM and MHCII expression. Both untreated hypothyroid (UHC) as well as iodine-treated (IC) cats revealed a significantly higher degree of thyroid inflammation and higher levels of IgM as the thyroxin-substituted animals (TC). Epithelial proliferation decreased significantly in the IC and TC groups as compared to the untreated controls. No significant differences regarding IgG production and HLAII expression were detectable. Early thyroid hormone therapy significantly
decreases both incidence and activity of autoimmune thyroiditis in cats as measured by inflammatory infiltration, IgM production and epithelial proliferation. Animals with excess iodide intake, however, show an aggravation of the autoimmune inflammatory activity.

Autoimmunity 1995;20(3):201-6
Excess iodine induces the expression of thyroid solid cell nests in lymphocytic thyroiditis-prone BB/W rats.
Zhu YP, Bilous M, Boyages SC.
Department of Clinical Endocrinology, Westmead Hospital, Sydney, Australia. Previous epidemiological studies have suggested that lymphocytic thyroiditis and/or an increased iodine intake may be risk factors for the development of thyroid cancer. We previously reported that excess iodine accelerated the development of thyroid lymphocytic infiltration (LI) in the autoimmune BB/W rat model. We also found that excess iodine increased thyroid cell proliferation in a disordered manner. The present study was designed to further explore these observations and to address the question as to whether excess iodine under certain conditions predisposes the thyroid gland to neoplasia. To test this hypothesis, the lymphocytic thyroiditis-prone BB/W rat was exposed to excess iodine in drinking water. Ten BB/W rats at 4 weeks of age were given iodine water (NaI 0.05%) for 10 weeks, whilst another 10 BB/W rats were given tap water and served as controls. Eighteen normal Wistar rats were also divided into excess iodine and control groups, served as a comparison to the BB/W rats. We found that an excess iodine intake accelerated the development of LI in the BB/W rat. Severe LI was usually accompanied by prominent thyroid cell proliferation, evident as numerous microfollicles and cell masses, not forming normal thyroid follicles. Numerous lymphocytes and plasma cells often encroached on these areas of increased cellular proliferation. The surprising feature, and a possible indicator of activated thyroid cell proliferation, was the high incidence of thyroid solid cell nest-like lesions (SCN) in the iodine treated BB/W rats.(ABSTRACT TRUNCATED AT 250 WORDS)”

 

[Captain_Coconut]

“Iodine reacts easily with toxic PUFA (omega-3 and -6 oils) to make antithyroid molecules.”

Where is this statement backed up? I have read quite the opposite from other sources...

 

[burtlancast]

Let's hear Ray about his Wolff-Chaikoff effect during the early 1900's studies for goiter prevention with mg doses of iodine for thousands of children.

 

[burtlancast]

@burtlancast yep ray “refuses to do his homework on iodine”. What a joke. What makes you think that?

Because for all his anti authoritarian views, Peat doesn't seem to respond too well to criticism.

A member here made a thread about how Peat used to be against sugar, with passages from his earlier books. Has he explained these passages and why he subsequently changed his mind? He didn't.

Has Peat made his homework on the milk A2/A1 casein controversy? No: it contradicts in parts his writings, so he just ignores the whole scientific evidence all together.

Has Peat recognized the widespread magnesium deficiency in the population?
No, he hasn't: he would rather talk about the magnesium/calcium balance and let folks be affected by the scores of degenerative diseases caused by a chronic lack of magnesium.

Autoimmunity 1995;20(3):201-6
Excess iodine induces the expression of thyroid solid cell nests in lymphocytic thyroiditis-prone BB/W rats.

The present study was designed to further explore these observations and to address the question as to whether excess iodine under certain conditions predisposes the thyroid gland to neoplasia.

And where are the statistics of increased thyroid cancer in populations ingesting very large doses of iodine (Japanese) ?

Nowhere to be found, i'm afraid.

 

[burtlancast]

burtlan, where is that from? He might have taken that much when he noticed that something wasn't right, not on a daily basis.

Well, you might have a point. I cannot find any source attesting he did take daily these grams amounts.

 

[Mauritio]

I'm doing it by myself, of course. I'm using Lugol. And i'm feeling really well, with improved mind clarity and sleep.

Keep in mind adults are supposed to ingest 120 mg of potassium iodide/day in case of nuclear emergency, and this dose is supposed to be ingested for as long as the situation calls it.

So, you're not going to end up in the emergency unit for taking 50mg.

I suggest reading thoroughly Guy Abraham's papers, they're a gold mine when it comes to wake up to the iodine-scare scam operation that's been going on for the last 60 years.

You'll learn some very interesting facts, like ovaries making their own thyroid hormones, and iodine being anti-estrogenic and pro-progesterone.

Apropos anti-estrogenic : do you know about the iodine painting protocol? You basically just paint iodine on your testicles. There is people who sell this information in a program , but that sounded like a scam to me. I tried it anyway, since i like to try stuff and it blew my mind . definitely increased libido ,erection quality and so on. Never felt like that since i was 15. It did give me some estrogenic (?) effects after a few days and thats why i stopped.I increased the dosage a lot though. I am not even a big proponent of iodine ,in fact i am quite indecisive about it. But there is 100% sth to it . Just google "iodine painting protocoll" just make sure to take Magnesium, K2 and selenium with it. I think there was some discussion about that in a thread on this forum not quite sure though. If someonehis libido and eq wants to boost thats the go to ...

 

[burtlancast]

Lol

Better alternative than Viagra, i guess...

 

[rebuke]

Lol

Better alternative than Viagra, i guess...

I had a bad experience with the iodine protocol. Lugols solution, B2/B3, magnesium, sea salt, vitamin C, and very little selenium. At first it had seemingly profound healing effects. I went wrong somewhere along the way, was a high stress time, started relying more on higher doses of iodine and less companion nutrients. 2 months of high stress day-to-day and inconsistent supplementation, I was back to where I started. Although I cannot deny what it did for me in terms of strength, impeccable posture, resiliency, IQ, perception, vision, etc etc. Was it the iodine, or the B2 or the vitamin C? Or my thyroid tissue being eaten? I don't know. I'm tempted to try again, I just don't know if its worth the risk.

 

[Such_Saturation]

Has anyone taken a radiation detector to their throat before/after this protocol thing?

 

[Amazoniac]

I find it impressive how having a robust metabolism gives you confidence for experimentation knowing the the body will restore to normal very fast and without sequela. Travis just mentioned somewhere taking 500 mg of potassium iodid at once. Zeus is not different: I casually took 35 grams of thiamine yesterday when feeling a bit off.

Out of curiosity:

Five grains [about 300 mg!] of potassium iodine for a few days may be required to overcome colloid hydration or iodine deficiency that delay recovery.

 

[accelerator]

And where are the statistics of increased thyroid cancer in populations ingesting very large doses of iodine (Japanese) ?

Nowhere to be found, i'm afraid.

They seem to be the 3rd hightest in the world for stomach cancer. Not sure I'd trade thyroid cancer for stomach cancer, even if that was the reality.

Stomach cancer statistics | World Cancer Research Fund International

 

[ecstatichamster]

Many things can do a lot of good if you do not increase the dose. It’s so easy to increase the dose and then get bad effects of everything.

 

[Elephanto]

You don't buy either Szent-Gyorgyi's regimen and empirical quote about potassium iodide?

And you don't think iodine holds the key to the bromine, fluoride, heavy metals plague burdening the whole organism and it's endocrine system (thyroid included), and affecting people worldwide?

Why do you reckon they insist on putting fluoride in the water while everybody with an ounce of common sense knows it's poison?

How much $$$$$$$ in treating symptoms are we talking about here?
I can't even put a price on that and the profits the pharma industry makes by selling gimmicks to it.

Just one example: how many sleeping pills have you ingested in your lifetime?

You keep bringing up the scientific points Ray makes about iodine, but it's clear he has a skewed view of the topic and even shows contemptible ignorance on many vital areas.

Which is extremely troubling considering he insists on being an authority on thyroid.
A thyroid specialist who refuses to do his homework on iodine?

What's going on here?

Boron has been shown to increase Fluoride excretion as well as having decalcifying properties. Interestingly, Israel has the highest concentrations of Boron in the soil worldwide and no water fluoridation. Tamarind also removes Fluoride.

I know that Fluoride inhibits Iodine uptake by the thyroid, but do you have some information on wether simply taking Iodine is going to solve this ? Shouldn't you have to remove fluoride first and then RDA Iodine intake would be sufficient (and safer).

They seem to be the 3rd hightest in the world for stomach cancer. Not sure I'd trade thyroid cancer for stomach cancer, even if that was the reality.

Stomach cancer statistics | World Cancer Research Fund International

That's a pretty wild guess at causation by Iodine. Their very high salt intake and wasabi consumption would be more sound candidates.

 

[accelerator]

Boron has been shown to increase Fluoride excretion as well as having decalcifying properties. Interestingly, Israel has the highest concentrations of Boron in the soil worldwide and no water fluoridation. Tamarind also removes Fluoride.

I know that Fluoride inhibits Iodine uptake by the thyroid, but do you have some information on wether simply taking Iodine is going to solve this ? Shouldn't you have to remove fluoride first and then RDA Iodine intake would be sufficient (and safer).


That's a pretty wild guess at causation by Iodine. Their very high salt intake and wasabi consumption would be more sound candidates.

It is a wild guess, which is why I replied to burtlancast, because he said their low incidence of cancer was based on iodine(which seems like a wild guess to me)

Basing any one conclusion for cancer for an entire population on any one molecule is essentially a wild guess.

That's why I was showing the opposite result based on the same exact thinking he used: 1 ingredient (iodine) being good or bad based on a study showing either a increase or decrease in cancer.

That's why I don't think it's a good argument.

The speculation on 1 thing is rather surface and really doesn't prove a lot without more context. What altitude did they live at? What's there physical exercise like? What else do they eat, or don't eat? How much salt? How many organ meats? How much protein do they eat? How much saturated fat? There are hundreds of reasons why a population could have lower cancer rates of one kind of cancer besides iodine.
Plus theres conflicting evidence showing the opposite too.

Salt has been shown to improve health too.

 

[burtlancast]

Boron has been shown to increase Fluoride excretion as well as having decalcifying properties. Interestingly, Israel has the highest concentrations of Boron in the soil worldwide and no water fluoridation. Tamarind also removes Fluoride.

Yes, Boron is a very interesting mineral able to excrete fluoride from bones. But most studies use it at dosages 100 mg-1000 mg per day, which necessitate supplementation.
And Boron cannot excrete other halides than fluoride, or heavy metals like Iodine can.
The body doesn't integrate Boron to organic molecules like it does Iodine either.

So,i would certainly supplement with Boron to get stronger bones with less fluoride, but it cannot in any case replace iodine supplementation.


As far as Tamarind goes, it's just a thought: it's a delicacy meant for gourmets whose sales argument includes fluoride detox.
In the studies conducted, ingestion of this sea animal increases fluoride excretion by 30%, (compared to 1500% for iodine ingested in mg amounts).

I know that Fluoride inhibits Iodine uptake by the thyroid, but do you have some information on wether simply taking Iodine is going to solve this ? Shouldn't you have to remove fluoride first and then RDA Iodine intake would be sufficient (and safer).

Yes: taking iodine will restore thyroid hormone production. Fluoride toxicity is potentiated by iodine deficiency: the body will normalise provided it gets sufficient iodine, even in presence of excess fluoride.

It has been known for sometime now that bromide competes with chloride in the extracellular space and that the total molar concentration of bromide plus chloride remains constant. (8) This concept has been used to decrease extracellular bromide levels by saline loading. However, the presence of bromide in the thyroid gland (9) and the central nervous system (10) suggests that there is another intracellular "pool" of bromide, not responding to chloride.

In the thyroid gland, bromide competes with iodide for uptake, oxydation and organification. (9,11 and vido infra)

Therefore, increasing iodide intake should lower bromide levels in the thyroid, preventing and reversing its thyrotoxic and goitrogenic effects. The same applies to fluoride. Galletti and Joyet (12) evaluated the effect of 5-10 mg fluoride on thyroid functions in hyperthyroid patients. Although fluoride inhibited the iodide-concentrating mechanism of the thyroid, fluoride did not accumulate in the thyroid. Based on their radioactive tracer studies, they concluded "Fluorine does not impair the capacity of the gland to synthesize thyroid hormones when there is an abundance of iodide in the blood." Therefore, fluoride toxicity depends on iodide supply.

Wachters