Mr. God of Cars
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- May 2, 2016
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- 165
You're right. PUFA may promote lean tissue (which probably refers to muscle or something) ALONG with fat gain.
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You're right. PUFA may promote lean tissue (which probably refers to muscle or something) ALONG with fat gain.
You're right. PUFA may promote lean tissue (which probably refers to muscle or something) ALONG with fat gain.
How do you know this is junk science? (genuine question here.)Show me one study which shows that PUFA promote lean tissue except this useless piece of junk science.
So what about the study I mentioned in the comments, showing that PUFA increases 11β-HSD1 much less than saturated fats, which increases 11β-HSD1 much less than trans fats? (11β-HSD1 activates cortisol and, therefore, promotes metabolic syndrome [insulin resistance and central obesity].)Again, even this study shows that most of the SFA group gained lean tissue, as well.
Short term effects of stressors are quite often the opposite of their long term effects.So what about the study I mentioned in the comments, showing that PUFA increases 11β-HSD1 much less than saturated fats, which increases 11β-HSD1 much less than trans fats? (11β-HSD1 activates cortisol and, therefore, promotes metabolic syndrome [insulin resistance and central obesity].)
This shouldn't be this way, because Ray Peat claimed coconut oil (which is essentially saturated fat) has anti-glucocorticoid (or anti-11β-HSD1, right?) properties, which I think should be because of saturated fats.
Well I don't know about the study but I know lots of people who eat lots of pufa, margerine rapeseed oil etc. And they are muscular and lean while my fats primary come from saturated I try to avoid pufa as much as possible but I have quite a big belly .
Yh but even on a calorie deficit these people keep there muscle and lose fat easily, but on a calorie deficit I lose muscle easy and keep the fat easy.Because calories still matters, if you eat at calories maintenance even if you eat PUFAs, you won't gain fat.
I'd avoid Pufas for health reasons, but if you want to lose fat you need to eat below you calories maintenance, don't listen to people here saying that being obese is healthy
But eating more than you normally burn can increase the metabolic rate by increasing active thyroid hormone, especially if you eat lots of carbs (preferentially sucrose), selenium-, saturated fats (or no fats at all) -, and animal protein-containing foods, besides adding sodium.Because calories still matters, if you eat at calories maintenance even if you eat PUFAs, you won't gain fat.
I'd avoid Pufas for health reasons, but if you want to lose fat you need to eat below you calories maintenance, don't listen to people here saying that being obese is healthy
Source?Short term effects of stressors are quite often the opposite of their long term effects.
But eating more than you normally burn can increase the metabolic rate
Is this a deficit with enough protein (1g/lb LBM)?Yh but even on a calorie deficit these people keep there muscle and lose fat easily, but on a calorie deficit I lose muscle easy and keep the fat easy.
Couple years ago before I found out about peat, I used to not care what I ate , crisps with pufa , mayonnaise, up to 5 grams of fish oil, and I would bulk up and only gain muscle and very minimal fat, since avoiding pufa I find it hard to add muscle without gaining lots of fat.
YhIs this a deficit with enough protein (1g/lb LBM)?
So what about the study I mentioned in the comments, showing that PUFA increases 11β-HSD1 much less than saturated fats, which increases 11β-HSD1 much less than trans fats? (11β-HSD1 activates cortisol and, therefore, promotes metabolic syndrome [insulin resistance and central obesity].)
This shouldn't be this way, because Ray Peat claimed coconut oil (which is essentially saturated fat) has anti-glucocorticoid (or anti-11β-HSD1, right?) properties, which I think should be because of saturated fats.
Least lipid peroxidation with HF-S?I had originally thought something is wrong with this study but I think they may be onto something. I began going through references 16-21 and found this study:
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It studied low-fat (LF), high-fat lard (HF-L), and high-fat safflower (HF-S) oil diets during weeks of re-feeding (after calorie restriction). The rats on the lard diet gained the most fat, the safflower oil was in the middle, and the high carb-diet the least (also with the least PUFA storage). Shocking right?
What’s interesting is the affect on the mitochondria, specifically the omega-6 oil causing proton leakage across the mitochondrion of the hepatic (liver) tissue, thus explaining why the omega-6 oils does not cause as much fat gain:
View attachment 15940
Basically the authors are showing uncoupling and thermogenesis in the liver occurring with the safflower oil vs lard. Not sure what to make of this...
How do you know this is junk science? (genuine question here.)