Click Here if you want to upgrade your account
If you were able to post but cannot do so now, send an email to admin at raypeatforum dot com and include your username and we will fix that right up for you.
Thank you! Wow. You do this all the time?
First of all thank you so much for this thread as it's given a very plausible explanation for what's going on in my particular case (described in "Why Does Starch Make Me Extremely Sick?") something I've been scrambling around trying to find. I will say that in my experience, starch+fat (as in starch with any amount of added fat, at all) is a problem possibly because starch easily generates endotoxin and fat easily transports it out. Without the fat the starch ceases to be a problem, without the starch the fat remains a problem that is simply less extreme probably due to the fact that sugars, fibers etc are still fermentable. I think this is why as Denise Minger pointed out, people seem to get magical results from both very low fat and low carb diets, but a mixed diet with significant amounts of both carbs and fats allows for both significant endotoxin production and transport out of the gut.
You can make glue out of it too.
I guess it depends on what you mean by rare. Estimate seem to range from c. > 1/100 to 1/250-300. So probably quite a few people going through my local supermarket every week.
They even compared the ratios of TNF-a/IL-10, and again the coconut oil was the lowest, including in comparison with the "low-fat".Again, the dose is completely realistic for real-world consumption standards. Maybe not for Peat standards, but how many times have you seen people drizzle on 2 tablespoons of oil and then some nuts on a "healthy salad", or eat 2 slices of pecan pie (that's 45g of fat in 250g of food
I gradually moved to a low-fat diet, and I don't plan to go back. However even when I consumed considerable amounts of butter, it wasn't anything close to that.
Your blog is excellent! You should add the link to your signature..
Agree. A great blog and I am working my way through The Scrivel Archive, imaginative drawings and I like your wit.Heh, I don't generally care for spreading my ideas (the right people will find them), and do not ever talk about my knowledge of Human Design in public either
.....
. I obviously have my chart right on my site (which was generated by software I wrote on my own using what I believe to be a more updated Ephemeris )., nor discuss any of the "advanced topics" or implications (because it's just too much detail to explain). But if it's a discussion of basic types, I'm fine with that I'm sure it's pretty evident to you why I act the way I act based on my 3.5/5.6 Cross of the Sleeping Phoenix triple-integration channel Manifestating-Generator design. penguin:, though I stopped drawing those because there really wasn't much more to say. Philosophy was set early in life, and now it's time to pursue skills, knowledge, and money 
Your replies are always interesting but why do some people get very sick anytime they eat starch? Haidut said that it can increase cortisol. I can't eat it without feeling very heavy and drugged with a very runny nose, lowered temps and swollen lymph nodes, and if I eat it several days in a row I get a flu and liver spasms... I don't have Corhn.
Interesting share. Thank you, bookmarked!Oracles,
adding more clues to our concerns with endotoxins.
When you slow down the transit time, foods remains in the intestines for longer and so carbohydrates, that are well known to be preferentially consumed by microbes, are the first to go; when that happens, microbes turn to protein and fat; an example of this is kefir fermentation. This occurs regardless of diet when the motility is diminished. As a consequence, relatively benign microbes are suppressed and the growth of problematic ones is encouraged.
Factors Controlling the Intestinal Bacteria: The Effect of Acute Obstruction and Stasis on Bacterial Types on JSTOR
“It is the general view that the addition of lactose or dextrin to the diet brings about the change in the intestinal types because these carbohydrates, unlike others, are slowly absorbed and hence reach the region of maximum bacterial activity.”
And here they interpret that as a benign thing because as they imply, a diet that lacks enough carbohydrates (or the foods remains stagnant for too long) encourages the growth of certain microbes, and so the shift occurs:
“A diet rich in protein of animal origin brings about an intestinal flora dominated by proteolytic putrefactive organisms, while a carbohydrate diet, more particularly one containing definite amounts of either lactose or dextrin, produces a complete change in the intestinal bacteria so that the predominant organisms are fermentative or aciduric.”
“This interpretation is in accord with the well-known observation that the feces of the breast-fed infant are acid in reaction whereas those of an adult on a high animal protein diet are alkaline, and coincides with the facts in the natural souring of milk and the ripening of silage, where the evidence is convincing that the first stage is carried on by members of the colon-aerogenes group, and as the acidity increases these are out-numbered by streptococci and these in turn by lactobacilli.”
“..Whatever may be the exact factor in determining the predominance of types, it is apparent that diet is not the sole factor, as the bacteria are subjected to all the variations in functions of the intestinal tract, namely, motility, secretions, and degree of absorption.”
“We have previously reported experiments in lower animals in which the evidence seemed to prove that the toxemia of acute intestinal obstruction is directly dependent on the nature of the intestinal flora.”
“It will be seen that the animals whose intestinal flora was aciduric survive the obstruction a little longer than those whose flora was proteolytic. There was, however, a much more marked difference in the postoperative behavior of these two groups. The meat eaters were listless from the first and refused their food. The lactose eaters, on the other hand, were alert and always eager for food.”
“The postoperative behavior of the meat eaters is also very similar to that of the monkey studied by Herter and Kendall7 which was sleepy and stupid when being fed a diet encouraging the growth of proteolytic organisms, but became alert when carbohydrates were added to the diet. More recently Underbill and Simpson 8 have observed similar effects in a dog fed the two types of diet. The striking thing about these experiments, however, is that irrespective of the character of the diet, i. e., whether lactose is given or not, a complete stasis of the intestinal content results in a proteolytic intestinal flora.”
They then mention a cruel experiment in which they simply closed the dogs’ intestines and compared a diet that consisted of protein+simple carbs versus a diverse diet with complex carbs:
“All of them died in the course of 2 weeks, those fed on the lactose surviving 2 or 3 days longer on the average than the meat eaters. At necropsy in all cases there was found above the site of obstruction a foul smelling, dark gray fluid, and examination revealed a great predominance of gram-negative proteolytic organisms.”
The same occured when a part of the intestine was cut and removed to later on be left hanging there obstructed, they reconnected both ends and kept the intestinal continuity #PETA; but with that obstructed part, the fecal matter starts to decay, and the same shift occurs, those microbes develop and soon cause a systemic problem when they reach circulation, “a great preponderance of gram-negative organisms” are found.
“In every case at death the bacteria in the closed loops were almost entirely gram-negative in type.”
They commented that “Animals may survive indefinitely with such closed intestinal segment” if it was sterilized before reinsertion.
“Apparently the succus entericus is not sufficiently bactericidal to suppress the growth of these organisms and is not comparable to the fluid in the free abdominal cavity in that respect. It is probable that the gram-positive bacteria disappear from these loops for the same reasons that they disappear from the normal intestine in starvation.”
“It is probable, however, that the mechanism of bacterial control is identical in both cases and is dependent on the presence of utilizable carbohydrate in all parts of the small intestine and colon. In conditions of stasis or obstruction even such carbohydrates as dextrin and lactose are probably completely absorbed in the upper part of the intestine and so cannot affect bacterial growth lower down.”
“The experiments with the closed intestinal loops demonstrate that the intestinal juice is not markedly bactericidal, at least so far as the proteolytic group of organisms is concerned. The disappearance of the gram-positive aciduric bacteria is probably due to the fact that carbohydrates are absent from such loops and that the intestinal juice is quite markedly alkaline in reaction.”
It seems to me that the most important conclusion from this is that the focus should be on the constant flow. However, to bring back the microbial balance to a safer profile you might need complex carbs.
--
Also interesting:
Abnormal intestinal motor patterns explain enteric colonization with gram-negative bacilli in late radiation enteropathy
Correlates dysmotility, impaired fasted peristalsis, not enough stomach acid with higher numbers of gram-negative bacteria.
Yeah, and although endotoxins have a deserved bad reputation, their signaling potential is often overlooked; what often passes unnoticed is the fact that to serve as signals they cannot be equal, even for a short term inflammatory response. Their structure is similar but they are different, and so they're going to affect the body differently.
