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C19Protocols – Reducing Risk of COVID-19 Infection and Severity
c19protocols.com
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@HDD maybe would interest your son.Thanks @bzmazu
(PDF) POST-ACUTE OR PROLONGED COVID-19: IVERMECTIN TREATMENT FOR PATIENTS WITH PERSISTENT SYMPTOMS OR POST-ACUTE
PDF | SUMMARY . INTRODUCTION: It is estimated that between 10 to 45% of people who become ill with COVID-19 will present with symptoms after the acute... | Find, read and cite all the research you need on ResearchGatewww.researchgate.net
W...O...W :-O
What a fantastic resource!
Great share @Makrosky!Thanks @bzmazu
(PDF) POST-ACUTE OR PROLONGED COVID-19: IVERMECTIN TREATMENT FOR PATIENTS WITH PERSISTENT SYMPTOMS OR POST-ACUTE
PDF | SUMMARY . INTRODUCTION: It is estimated that between 10 to 45% of people who become ill with COVID-19 will present with symptoms after the acute... | Find, read and cite all the research you need on ResearchGatewww.researchgate.net
W...O...W :-O
4% homemade saline solution in distilled water.I’m looking for Peat treatment that included using a nebulizer. Does anyone know where it’s posted or what he said to nebulize?
Thank you! My sister has covid (again) with a bad cough. Thought nebulizer could help.4% homemade saline solution in distilled water.
Around 8 tsp of table salt to 1 gallon of distilled water. You can make a smaller amount of course, you just have to do the math.
It doesn't require any prep outside the above, and of course, storage in sealed mason jar (for example.)
Open in browser QUERCETIN AND CURCUMIN AS POTENTIALLY POWERFUL "PRIOLOID" THERAPEUTICSQuercetin turns fibrils into protease-sensitive, structurally loose and non-cytotoxic formsPlease note in the above image Curcumin’s effect on fibril formation.QUERCETIN I have been searching for answers to the systemic amyloidosis and anchorless prion pathologies of the Spike Protein. I believe Quercetin could prove to be a very powerful therapeutic in combatting these pathological mechanisms. What I find most interesting about Quercetin is that it breaks apart prion fibril formations. One wonders if this could potentially prevent or remedy the fibril aggregates being found by embalmers. Please note, I do not believe the fibril aggregates are any sort of “nanotechnology.” I believe they are rapidly induced and developing “prioloid” aggregates. Some highlights regarding Quercetin and amyloids: • Quercetin binding to prion protein is determined, and the network of the binding site at the molecular level is visualized. • In contrast to conventional studies of amyloidogenic drugs, quercetin accelerates fibrillation of prion protein. (A GOOD thing.) • Quercetin turns fibrils into protease-sensitive, structurally loose and non-cytotoxic forms. • Quercetin shows its uniqueness from typical antiamyloidogenic drugs. • Quercetin treatment can disaggregate moPrP (mouse prion) fibrils and lead to the formation of the proteinase-sensitive amorphous aggregates. Furthermore, quercetin-bound fibrils can reduce the membrane disruption of erythrocytes. • Quercetin is distinct from the typical function of antiamyloidogenic drugs that inhibit the formation of amyloid fibrils. CURCUMIN Curcumin may be a powerful alloy and “wing man” to Quercetin. • Curcumin alone reduced prion fibril formation significantly. • Curcumin effectively rescues the cells from apoptosis and decreases the ROS level caused by subsequent co-incubation with prion amyloid fibrils. I believe urgent clinical trials are needed to assess the efficacy of Quercetin and Curcumin as prophylaxis, and treatment of both Acute and Long COVID. Quercetin binding accelerates prion fibrillation into proteinase sensitive and loosely structured amyloidsAmyloidoses are caused by the deposition of amyloid fibrils ascribed to protein misfolding. In this study, we examined the antiamyloidogenic and antio…
www.sciencedirect.com
Quercetin Disaggregates Prion Fibrils and Decreases Fibril-Induced Cytotoxicity and Oxidative Stress - PubMedTransmissible spongiform encephalopathies (TSEs) are fatal neurodegenerative diseases caused by misfolding and aggregation of prion protein (PrP). Previous studies have demonstrated that quercetin can disaggregate some amyloid fibrils, such as amyloid β peptide (Aβ) and α-synuclein. However, the...
pubmed.ncbi.nlm.nih.gov
Curcumin Reduces Amyloid Fibrillation of Prion Protein and Decreases Reactive Oxidative StressMisfolding and aggregation into amyloids of the prion protein (PrP) is responsible for the development of fatal transmissible neurodegenerative diseases. Various studies on curcumin demonstrate promise for the prevention of Alzheimer’s disease ...
www.ncbi.nlm.nih.gov
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My son has battled long Covid 17 months. Mass cells disorder from Covid damaged his gut. He can only eat very low histamine foods. Nothing packaged except cooked rice and oats. Some fruits and vegetables.
QUERCETIN AND CURCUMIN AS POTENTIALLY POWERFUL "PRIOLOID" THERAPEUTICS
Quercetin turns fibrils into protease-sensitive, structurally loose and non-cytotoxic forms
Please note in the above image Curcumin’s effect on fibril formation.
QUERCETIN
I have been searching for answers to the systemic amyloidosis and anchorless prion pathologies of the Spike Protein. I believe Quercetin could prove to be a very powerful therapeutic in combatting these pathological mechanisms.
What I find most interesting about Quercetin is that it breaks apart prion fibril formations. One wonders if this could potentially prevent or remedy the fibril aggregates being found by embalmers. Please note, I do not believe the fibril aggregates are any sort of “nanotechnology.” I believe they are rapidly induced and developing “prioloid” aggregates.
Some highlights regarding Quercetin and amyloids:
• Quercetin binding to prion protein is determined, and the network of the binding site at the molecular level is visualized.
• In contrast to conventional studies of amyloidogenic drugs, quercetin accelerates fibrillation of prion protein. (A GOOD thing.)
• Quercetin turns fibrils into protease-sensitive, structurally loose and non-cytotoxic forms.
• Quercetin shows its uniqueness from typical antiamyloidogenic drugs.
• Quercetin treatment can disaggregate moPrP (mouse prion) fibrils and lead to the formation of the proteinase-sensitive amorphous aggregates. Furthermore, quercetin-bound fibrils can reduce the membrane disruption of erythrocytes.
• Quercetin is distinct from the typical function of antiamyloidogenic drugs that inhibit the formation of amyloid fibrils.
CURCUMIN
Curcumin may be a powerful alloy and “wing man” to Quercetin.
• Curcumin alone reduced prion fibril formation significantly.
• Curcumin effectively rescues the cells from apoptosis and decreases the ROS level caused by subsequent co-incubation with prion amyloid fibrils.
I believe urgent clinical trials are needed to assess the efficacy of Quercetin and Curcumin as prophylaxis, and treatment of both Acute and Long COVID.
Quercetin binding accelerates prion fibrillation into proteinase sensitive and loosely structured amyloids
Amyloidoses are caused by the deposition of amyloid fibrils ascribed to protein misfolding. In this study, we examined the antiamyloidogenic and antio…www.sciencedirect.comQuercetin Disaggregates Prion Fibrils and Decreases Fibril-Induced Cytotoxicity and Oxidative Stress - PubMed
Transmissible spongiform encephalopathies (TSEs) are fatal neurodegenerative diseases caused by misfolding and aggregation of prion protein (PrP). Previous studies have demonstrated that quercetin can disaggregate some amyloid fibrils, such as amyloid β peptide (Aβ) and α-synuclein. However, the...pubmed.ncbi.nlm.nih.govCurcumin Reduces Amyloid Fibrillation of Prion Protein and Decreases Reactive Oxidative Stress
Misfolding and aggregation into amyloids of the prion protein (PrP) is responsible for the development of fatal transmissible neurodegenerative diseases. Various studies on curcumin demonstrate promise for the prevention of Alzheimer’s disease ...www.ncbi.nlm.nih.gov
My son has battled long Covid 17 months. Mass cells disorder from Covid damaged his gut. He can only eat very low histamine foods. Nothing packaged except cooked rice and oats. Some fruits and vegetables.
Protocols which have human studies showing efficacy (for patients ill with COVID).
1. IVM: Studies, Dosing
2. HCQ: Studies, Dosing, Dosing2
3. H1+H2 blockers: Study, Dosing
4. ??please insert any new treatments, surely there are more by now
Nemo's thoughts on combining 1,2,3 depending on exposure/longhaul etc link, link1, link2, longhaul, longhaul2.
He has reacted to IVM but used it carefully in beginning. Also uses H1&H2 blockers and supplements. He’s making progress, slowly.If you think histamine is an issue you might try treatment number (3) below which is based on H1&H2 histamine blockers.