Bug protein

[JewlzSanguine]

What do you think the reasoning is behind the push for bug protein? Why bug protein in particular?

 

[Summer]

Because the ruling class views the general population as cattle, and cattle eat bugs and plants.

 

[JamesGatz]

Basically the same answer as above but another reason I believe is that killing animals like cows, sheep, goats, chickens (innocent-looking animals) gives people PTSD and takes a toll on their mental health but no one cares about killing bugs since they're unattractive and look menacing

Shooting/killing a menacing/unattractive-looking organism such as a crocodile or snake is a lot easier than killing a majestic/attractive animal such as horses/cows

Also - bugs are always having sex so they reproduce very quickly because of the short hatching time as well - they also aren't fed any special crops much compared to cows/pigs/goats so they're easier to feed/tend to

In general its just a lot easier running a bug farm than animal farm

 

[David PS]

Edible insect products flying off shelves

From protein-packed snacks to low-cost fertilizer, businesses are showcasing how lifesaving and beneficial products made with insects can be.

www.fox4now.com

 

[Peatness]

Because it's a brilliant depopulation tool

African Seasonal Ataxia (ASA) is another interesting recently described clinical
entity which presumably has a thiamine-related foundation. This syndrome has
been recently described in people who live in Western Nigeria and is characterized
by ataxia, tremors, and decreased levels of consciousness. These symptoms occur
during the rainy season of July through October. ASA usually follows a large carbohydrate
meal. At its peak incidence, ASA can account for well over 70% of hospital
and clinic admissions.
Various hypotheses have been advanced to explain ASA; however, strong evidence
supports a mechanism related to thiamine deficiency. There is a clinical triad
of cerebellar ataxia, ocular disturbances, and encephalopathy usually seen in acute
thiamine deficiency. Upon examination of the dietary intake of the low socioeconomic
strata of patients, it was found that almost all had consumed significant
amounts of roasted silkworm larvae Anaphe venata. The availability of these larvae
in the marketplace corresponds to the rainy season. The larvae represent a valuable
protein source for rainforest people.
The practice of entomophagy in low socioeconomic cultures is accepted. Protein
sources are relatively scarce for these people, who subsist largely on carbohydraterich
diets. Subsequently, it has been shown that there is a thiaminase present in the
Anaphe venata larvae. During the rainy season, these larvae fall from specific trees,
and are gathered and sold in markets. Subsequent consumption of larvae containing
a thiaminase by people ordinarily eating carbohydrate-rich diets can explain the
rapid onset of symptoms resembling those of thiamine deficiency. As a corollary
to this recent description are earlier descriptions of similar outbreaks of thiamine
deficiency. There was, for example, the outbreak of the so-called Chastek paralysis,
a disorder of silver foxes on a fox ranch in Minnesota. These foxes were fed raw
fish and within a few weeks developed ataxia, changes in consciousness, seizures,
and death. Pathologically, brain lesions resembled those seen in thiamine deficiency.
Subsequent work showed the presence of a thiaminase in the viscera of the raw fish,
which had precipitated the disorder.
Inherited ataxias are a group of relatively rare neurological disorders genetically
transmitted, which have as a common denominator ataxia and the possibility of
successful thiamine treatment. These diverse, yet related ataxias include Refsum’s
disease and Friedreich’s ataxia. This group of disorders has a defect in the enzyme
pyruvate decarboxylase. Pathologically, these disorders show mitochondrial damage
in selective brain regions. Treatment regimes include thiamine and ketonic diet therapies.

Source

Preface

Thiamine Deficiency and Clinical Disorders By David W McCandless