I stand by what I said. This is a delicate and complicated subject. There are a lot of people, smart included, that are looking for magic pills on this forum and elsewhere. No disrespect to H, but with great influence comes responsibility. It is what it is.
Caffeine absolutely increases FFA in excessive doses. There is zero debate about that. As far as excessive adipose increases FFA is speculative. I posted a study last year that shows the body modulates NEFA independent of adipose mass. I'd look to find it for you, but I just moved and dont have time to get into any thread context conflicts.
Ray Peat recommends T3, he advices people to take small amounts through out the day. But he has also recommended high t3. He had a friend who's liver was destroy from heroin and alcohol and told him to take t3 50 micrograms morning and night. When he went to his doctor a year and a half later and his liver shrunk. He has also told the story of giving a women high dose desiccated thyroid and I think she was obese.
Ray Peat also recommends bromocriptine. He has talked about it in his interviews and articles, he does not think one should take it for an extended period. I think he said something like six weeks because it can cause heart valve fibrosis. I think a member here took bromocriptine for a year and I think her heart is fine. I don't think he recommends cabergoline, but I am not sure.
Caffeine does increase free fatty acids but not to the degree that it causes diabetes or lipid per oxidation. In fact it protects against diabetes and reactive oxygen species.
Long-term consumption of caffeine improves glucose homeostasis by enhancing insulinotropic action through islet insulin/insulin-like growth factor ... - PubMed - NCBI
Caffeine as an antioxidant: inhibition of lipid peroxidation induced by reactive oxygen species
Your anti-caffeine rhetoric doesn't add up.The health benefits of high or low dose caffeine are established.