Arterial Plaque Via Oxidizes Cholesterol Deposits

Joined
Apr 8, 2018
Messages
1,142
Location
The Netherlands
Best peeps,

What are the major sources of oxidizes cholesterol in the diet ?
What does Peat say about oxidizes cholesterol and their deposits in arteries ?
How can we clear arterial plaque of oxidized cholesterol deposits ? If you know a way please share, thank you..

Hope to hear from you soon,

have a nice day!
 
Joined
Nov 21, 2015
Messages
10,501
Cholesterol, longevity, intelligence, and health.

A consistent program to prevent the oxidation of cholesterol would have to include all of the vitamins and minerals that are involved in antioxidant defense, avoidance of nutrients that exacerbate the destructive oxidations, and an effort to normalize the hormones and other factors, such as carbon dioxide, that have protective effects against free radical oxidation. A low level of cholesterol might increase susceptibility to the oxidants.
 

yerrag

Member
Joined
Mar 29, 2016
Messages
10,883
Location
Manila
upload_2018-10-10_17-39-52.png


From page 3, Sep 2018 Ray Peat's Newsletter
 

yerrag

Member
Joined
Mar 29, 2016
Messages
10,883
Location
Manila
upload_2018-10-10_17-46-11.png

upload_2018-10-10_17-47-21.png
 

yerrag

Member
Joined
Mar 29, 2016
Messages
10,883
Location
Manila
upload_2018-10-10_17-56-21.png
 
Z

Zsazsa

Guest
Active atherosclerotic plaques accumulate glucose
https://www.ahajournals.org/doi/full/10.1161/01.CIR.0000020548.60110.76
Inflammation is important in both the pathogenesis and outcome of atherosclerosis.1 Plaques containing numerous inflammatory cells, in particular macrophages, have a high risk of rupture, whereas those with few inflammatory cells are at lower risk.2,3 The current “gold standard” imaging technique for atherosclerosis is x-ray contrast angiography, which provides high-resolution definition of the site and severity of luminal stenoses, but no information about plaque inflammation.

There is a need to quantify plaque inflammation to predict the risk of plaque rupture and to monitor the effects of atheroma-modifying therapies. This is important because recent experimental and clinical studies strongly suggest that hepatic hydroxymethyl glutaryl coenzyme A reductase inhibitors (statins) promote plaque stability by decreasing plaque macrophage content and activity without substantially reducing plaque size and therefore angiographic appearance.4

[18F]-fluorodeoxyglucose (18FDG) is a glucose analogue that is taken up by cells in proportion to their metabolic activity.5We tested the hypothesis that plaque inflammation could be visualized and quantified non-invasively using 18FDG-PET in patients with symptomatic carotid artery disease.
 

Similar threads

Back
Top Bottom