somuch4food
Member
- Joined
- Aug 23, 2018
- Messages
- 1,281
Eat what you want to eat. Get more rest. Don't exercise.
I second that. Enjoyment of food is important for digestion.
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Eat what you want to eat. Get more rest. Don't exercise.
Thanks for chiming in, Pompadour@Vinny , you've said, that you didn't tolerate strong substances like garlic etc. So be very very prudent with essential oils! They can provoke a very acute response from the pancreas and gallbladder. Just a humble word of caution. Wish you to solve your problems!
I have done this as well, and I did find it effective, at least in the shorter term. The thing that always throws a wrench in this for me is the really lean competitive eaters. Here's 96 pound Nela Zisser demolishing 5 pounds of fries (over 7,000 calories) in 20 minutes-
She's doing challenges like this weekly, and has been doing it for a few years, and her figure just doesn't change. How can this thin girl (almost too thin) go through 2-5x the daily caloric intake (and also sheer weight of food) in a single meal of a man 70 pounds heavier than her, and it not have an impact on her body comp? No doubt there is something wrong with always restricting calories.
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I have done this as well, and I did find it effective, at least in the shorter term. The thing that always throws a wrench in this for me is the really lean competitive eaters. Here's 96 pound Nela Zisser demolishing 5 pounds of fries (over 7,000 calories) in 20 minutes-
She's doing challenges like this weekly, and has been doing it for a few years, and her figure just doesn't change. How can this thin girl (almost too thin) go through 2-5x the daily caloric intake (and also sheer weight of food) in a single meal of a man 70 pounds heavier than her, and it not have an impact on her body comp? No doubt there is something wrong with always restricting calories.
And she's not the only one. Molly Schuler is 120 pounds, and she does 2-3x the amount Nela does on a weekly basis, and she isn't ballooning up, either.
I know Kelj and Billy Craig have made similar arguments. One thing I do remember..... when monitoring calories in a planned deficit, I would have a week eating at maintenance (at least) every 4-6 weeks. I was also regularly going on vacations (often to Vegas), and during those times, all restrictions were off! Can you say buffet? There is absolutely something to those "breaks" that helps with the goal.
Lol. This is the kind of thing that'll give Peat a heart attack.
How old is she? If she's still finishing growing then metabolism is still high and can take care of PUFA by diluting it. It's once growth is over that PUFA starts to rapidly build-up at 5x the rate as before and slows metabolism.
Also I needed to watch this to purify my eyes :
I doubt they'll be any different w/r to use of sat fats, but I could be wrong. People go to the supermarket and they grab anything on the shelves that says 'vegetable' for their cooking oil. Thinking if it says 'vegetable' it must be good.She's in her mid twenties, and also lives in New Zealand. It's possible more saturated oils are generally used for cooking there.
She's in her mid twenties, and also lives in New Zealand. It's possible more saturated oils are generally used for cooking there.
If she's in her mid 20s then my guess would be that she's relying heavily on fat oxidation and not sugar oxidation and that's why she's able to be thin. I was stick thin when I was eating lots of PUFA (people would always ask me if I was even eating at all) because it makes you oxidize fat.
She looks really young, I thought she was around 16. Girls I knew in high school looked older than her, not in a bad way but in the sense of having more mature facial development. My face has started maturing again after reducing PUFA consumption (it had stopped while I was eating PUFA and burning fat), so I think burning fat might have other bad consequences even if it does keep you thin. (Or maybe she just naturally has young-looking facial features.)
The infectious element in obesity is real in me. I imagine the bacterial content in my vascular system is large enough to form a quorum, whereupon p. gingivalis is able to form strong biofilms as well as interact with other microorganisms to form a synergistic and symbiotic relationship of the vilest kind to me. This is equivalent to having a government where corruption is institutionalized (much like the US deep state, if you agree that it exists). P. gingivalis is able to sequestering peptides (incretins) that have an effect on when and how much insulin is secreted and used as part of balancing our metabolic production of energy. The effect of this interference by the bacteria is to channel blood glucose away from tissue absorption into fat production in the liver. It also deprives the body of relying on glycogen when our blood sugar runs low. We are left to eat more, and as we eat more, a large portion of the input isn't used for energy production, but is stored in the form of fats. This leads to my weight gain, and as time goes on, it would become worse as I could become obese.
They could be throwing it up after.
This is exactly why I think we should emphasize immune system related approaches when tackling bacteria dependent issues. Antimicrobials certainly have their place but all to often the mucosal immunity is overlooked.You may be interested by this one: Augmentation of Cationic Antimicrobial Peptide Production with Histone Deacetylase Inhibitors as a Novel Epigenetic Therapy for Bacterial Infections
"Indeed, pathogens have evolved the opposite strategy of down-regulating CAMP expression to better establish themselves in the host. For example, several bacterial pathogens down-regulate the cathelicidin LL-37, secretory leukocyte protease inhibitor (SLPI), and, or human beta defensins (e.g., HBD-1, HBD-2, HBD-3) in tissue culture cells [8,9], and animal infection models [10]. These results are consistent with the detection of significantly lower concentrations of CAMPs in infected individuals."
"Yin and Chung reported that the oral pathogens Poryphyromonas gingivalis and Fusobacterium nucleatum modulated HDAC1 and, or HDAC2 expression in a human immortalized human keratinocyte cells and primary gingival epithelial cells [16]. P gingivalis caused the most significant suppression of HDAC gene transcription and lower numbers of bacteria able to significantly reduce HDAC-1 mRNA compared to HDAC-2 mRNA, which required a higher multiplicity of infection."
She,s got a video 3 years ago, where she,s answering some questions (mostly dumm, imo). No, they don't throw up, that's a myth.They could be throwing it up after.
This is exactly why I think we should emphasize immune system related approaches when tackling bacteria dependent issues. Antimicrobials certainly have their place but all to often the mucosal immunity is overlooked.
There are a lot of studies showing leaky gut induced by an introduction of a pathogenic bacteria to the microbiome. Also IGA, Host defense peptides, and pretty much every other antimicrobial defense protein has been shown to reduce intestinal permeability. I would say that mucosal immunity is the most important factor in leaky gut.I wonder whether mucosal immunity is one or the most significant factor in gut dysbiosis. Maybe leaky gut can be caused by pathogens in the gut lining.
There must be other factors like bile production and flow or what you eat.
There are a lot of studies showing leaky gut induced by an introduction of a pathogenic bacteria to the microbiome. Also IGA, Host defense peptides, and pretty much every other antimicrobial defense protein has been shown to reduce intestinal permeability. I would say that mucosal immunity is the most important factor in leaky gut.
Ironically that was exactly the same for me. I turned 50 last November and I’m finally back to a comfort body composition. You just have to decide what you want and come up with a plan to get there in a way that doesn’t involve destroying your health.In a way, I should consider myself lucky. It happened at a quite late age in my life - 46-47.